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GoPubMed Proteins lists recent and important papers and reviews for proteins. Page last changed on 19 Dec 2016.

Zinc finger protein 295

Top mentioned proteins: TOP, HAD, Ciliary Neurotrophic Factor, TBP, CGR19
Papers on Znf295
Genetics of hand grip strength in mid to late life.
Mather et al., Sydney, Australia. In Age (dordr), Feb 2015
Gene-based analyses in HCS identified two significant genes (ZNF295, C2CD2), but these results were not replicated in Sydney MAS.
The complex SNP and CNV genetic architecture of the increased risk of congenital heart defects in Down syndrome.
Antonarakis et al., Genève, Switzerland. In Genome Res, 2013
One of these CNVs maps near the RIPK4 gene, and the second includes the ZBTB21 (previously ZNF295) gene, highlighting the potential role of these genes in the pathogenesis of CHD in DS.
Expression of trisomic proteins in Down syndrome model systems.
Gardiner et al., Aurora, United States. In Gene, 2013
Genes for 12 of these proteins are trisomic in the Tc1 mouse model of DS, but only SIM2 and ZNF295 showed elevated expression in Tc1 cortex when compared with controls.
Identification of a Selective Small-Molecule Inhibitor of Breast Cancer Stem Cells—Probe 2
Schreiber et al., Bethesda, United States. In Unknown Journal, 2011
Gene expression profiling revealed that ML245 regulated the expression of several pro-apoptotic/mitochondrial maintenance factors (such as caspase recruitment domain family member 10, activating transcription factor 4, mitochondrial ribosomal protein MRPL12), and DNA-modifying enzymes (such as Early Growth Response 1, zinc finger-containing proteins ZNF295 and CGRRF1, and TBP-associated factor 1D), in the CSC-like cell line and not the isogenic control cell line.
Upregulation of beta-catenin expression in down syndrome model Ts65Dn mouse brain.
El Idrissi et al., New York City, United States. In Neuroscience, 2009
Using a mouse model of DS, the Ts65Dn mouse, we have performed mRNA and protein measurements to identify genes on chromosomes not syntenic with Hsa 21 whose expression is affected by the presence of three copies of genes between loci Mrpl39 and Znf295 on mouse chromosome 16 (Mmu 16).
Novel human BTB/POZ domain-containing zinc finger protein ZNF295 is directly associated with ZFP161.
Shimizu et al., Tokyo, Japan. In Biochem Biophys Res Commun, 2005
ZNF295 may be involved in the bi-directional control of gene expression in concert with ZFP161
Dosage-dependent over-expression of genes in the trisomic region of Ts1Cje mouse model for Down syndrome.
Yamakawa et al., Wako, Japan. In Hum Mol Genet, 2004
We have therefore investigated global gene expression profiles in Ts1Cje, a mouse model for DS that displays learning deficits and has a segmental trisomy of chromosome 16 orthologous to a segment of human chromosome 21 spanning from Sod1 to Znf295.
Evolutionary breakpoints on human chromosome 21.
Gardiner et al., Bar Harbor, United States. In Genomics, 2001
The mouse chromosome 16/chromosome 17 evolutionary breakpoint is between human genes ZNF295 and UMODL1, showing there are seven genes in the chromosome 16 segment distal to Tmprss2.
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