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GoPubMed Proteins lists recent and important papers and reviews for proteins. Page last changed on 19 Dec 2016.

YY1 transcription factor a

Top mentioned proteins: CAN, V1a, SP1, Histone, CIs
Papers using YY1 antibodies
Model-based analysis of ChIP-Seq (MACS).
Bosenberg Marcus, In PLoS Genetics, 2007
... Antibodies used were anti-DCT (D-18), anti-YY1 (H-414) and anti-c-Kit (H-300) from Santa Cruz Biotechnology, anti-MITF (C5 mouse monoclonal ...
Core transcriptional regulatory circuitry in human embryonic stem cells.
Cordes Nils, In PLoS ONE, 2004
... for Western blotting and immunofluoresence included anti-Bmi1 (Upstate; Lake Placid, NY), anti-Ring1B (MBL; Woburn, MA), anti-YY1 (Santa Cruz Biotechnology; Santa Cruz, CA), anti-tubulin ...
WebLogo: A sequence logo generator.
Khanin Raya, In PLoS ONE, 2003
... Antibodies against E2F1 (sc-193), E2F4 (sc-1082x), MAX(sc-197), YY1(Sc-7341) were obtained from Santa Cruz Biotechnology, CA ...
Papers on YY1
MiR-381 inhibits epithelial ovarian cancer malignancy via YY1 suppression.
Lou et al., Harbin, China. In Tumour Biol, Feb 2016
We also characterized the phenotype regarding cell proliferation, cell migration, and cell invasion in EOC cells lines with altered expression levels of both miR-381 and its target gene, YY1.
Yin Yang 1 promotes mTORC2-mediated AKT phosphorylation.
Sui et al., Winston-Salem, United States. In J Mol Cell Biol, Feb 2016
UNASSIGNED: Yin Yang 1 (YY1) regulates both gene expression and protein modifications, and has shown a proliferative role in cancers.
Forced Activation of Notch in Macrophages Represses Tumor Growth by Upregulating miR-125a and Disabling Tumor-Associated Macrophages.
Qin et al., Key West, United States. In Cancer Res, Feb 2016
We also identified a positive feedback loop for miR-125a expression mediated by RYBP and YY1.
A novel role of Yin-Yang-1 in pulmonary tuberculosis through the regulation of the chemokine CCL4.
Huerta-Yepez et al., Mexico. In Tuberculosis (edinb), Jan 2016
Yin-Yang-1 (YY1) plays a major role in the maintenance and progression of some pulmonary diseases, including pulmonary fibrosis.
Nitric oxide-mediated sensitization of resistant tumor cells to apoptosis by chemo-immunotherapeutics.
Garban et al., Los Angeles, United States. In Redox Biol, Dec 2015
Endogenous/exogenous NO mediated its immune sensitizing effect by inhibiting NF-κΒ activity and downstream, inactivating the repressor transcription factor YY1, which inhibited both Fas and DR5 expressions.
Dual roles of nitric oxide in the regulation of tumor cell response and resistance to photodynamic therapy.
Bonavida et al., Udine, Italy. In Redox Biol, Dec 2015
The findings revealed that NO mediates its effects by interfering with a dysregulated pro-survival/anti-apoptotic NF-κB/Snail/YY1/RKIP loop which is often expressed in cancer cells.
Transcription factor trapping by RNA in gene regulatory elements.
Young et al., Cambridge, United States. In Science, Dec 2015
We show that the ubiquitously expressed TF Yin-Yang 1 (YY1) binds to both gene regulatory elements and their associated RNA species across the entire genome.
Transcription factors that interact with p53 and Mdm2.
Frazier et al., Winston-Salem, United States. In Int J Cancer, Aug 2015
In this review, we focus on transcription factors that directly interact with p53/Mdm2 through direct binding including Dmp1, E2F1, YB-1 and YY1.
The role of YY1 in oncogenesis and its potential as a drug target in cancer therapies.
Sui et al., Harbin, China. In Curr Cancer Drug Targets, 2014
Yin Yang 1 (YY1) is a multifunctional protein regulating both gene transcription and protein modifications.
Spatial Regulation of V-(D)J Recombination at Antigen Receptor Loci.
Busslinger et al., Vienna, Austria. In Adv Immunol, 2014
The B-cell-specific Pax5, ubiquitous YY1, and architectural CTCF/cohesin proteins regulate Igh locus contraction in pro-B cells by binding to multiple sites in the VH gene cluster.
Cloning and Transcriptional Activity of the Mouse Omi/HtrA2 Gene Promoter.
Liu et al., Beijing, China. In Int J Mol Sci, 2014
Bioinformatics analysis suggested that the Omi/HtrA2 gene promoter contains a CpG island at -709~+37 bp, and eight heat shock transcription factor 1 (HSF1) sites, two Sp1 transcription factor (SP1)sites, one activator protein (AP) site, seven p53 sites, and four YY1 transcription factor(YY1) sites were predicted in the core areas.
Intersection of population variation and autoimmunity genetics in human T cell activation.
Benoist et al., Cambridge, United States. In Science, 2014
We further fine-mapped and validated a single-base variant that modulates YY1 binding and the activity of an enhancer element controlling the autoimmune-associated IL2RA gene, affecting its activity in activated but not regulatory T cells.
Flexible long-range loops in the VH gene region of the Igh locus facilitate the generation of a diverse antibody repertoire.
Busslinger et al., Vienna, Austria. In Immunity, 2013
In pro-B cells, these local domains engaged in long-range interactions across the Igh locus, which depend on the regulators Pax5, YY1, and CTCF.
Yin Yang 1 deficiency in skeletal muscle protects against rapamycin-induced diabetic-like symptoms through activation of insulin/IGF signaling.
Puigserver et al., Boston, United States. In Cell Metab, 2012
Importantly, skeletal muscle-specific YY1 knockout mice were protected from rapamycin-induced diabetic-like symptoms.
Chromatin-modifying enzymes as modulators of reprogramming.
Daley et al., Boston, United States. In Nature, 2012
Whereas inhibition of the core components of the polycomb repressive complex 1 and 2, including the histone 3 lysine 27 methyltransferase EZH2, reduced reprogramming efficiency, suppression of SUV39H1, YY1 and DOT1L enhanced reprogramming.
Beta-arrestin1 regulates zebrafish hematopoiesis through binding to YY1 and relieving polycomb group repression.
Pei et al., Shanghai, China. In Cell, 2009
Study demonstrated that beta-arrestin1 is critically involved in zebrafish primitive hematopoiesis, where beta-arrestin1 binds to and sequesters the PcG recruiter YY1, thus relieving PcG-mediated repression of cdx4-hox pathway.
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