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8 documents found
1: Title: Inactivating Frameshift Mutations of HACD4 and TCP10L Tumor Suppressor Genes in Colorectal and Gastric Cancers.
Authors: Jo, Yun Sol, et.al. .
Journal: Pathology oncology research : POR (Pathol Oncol Res), 2018 .
No Abstract available.
Affiliation: Department of Pathology, College of Medicine, The Catholic University of Korea, 505 Banpo-dong, Socho-gu, Seoul, 137-701, South Korea. Department of Pathology, College of Medicine, The Catholic University of Korea, 505 Banpo-dong, Socho-gu, Seoul, 137-701, South Korea. suhulee@catholic.ac.kr. .
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2: Title: Frameshift Mutations in Repeat Sequences of ANK3, HACD4, TCP10L, TP53BP1, MFN1, LCMT2, RNMT, TRMT6, METTL8 and METTL16 Genes in Colon Cancers.
Authors: Yeon, Su Yeon, et.al. .
Journal: Pathology oncology research : POR (Pathol Oncol Res), 2017 .
Snippet: ANK3, HACD4, TCP10L, TP53BP1, MFN1, LCMT2, RNMT, TRMT6, METTL8 and METTL16 harbored 11 (13.9%), 3 (3.8%),
Affiliation: Department of Pathology, College of Medicine, The Catholic University of Korea, 505 Banpo-dong, Socho-gu, Seoul, 137-701, South Korea. Department of Pathology, College of Medicine, The Catholic University of Korea, 505 Banpo-dong, Socho-gu, Seoul, 137-701, South Korea. suhulee@catholic.ac.kr. .
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3: Title: TCP10L synergizes with MAD1 in transcriptional suppression and cell cycle arrest through mutual interaction.
Authors: Shen, Suqin, et.al. .
Journal: BMB reports (Bmb Rep), Vol. 49 (6): 325-30, 2016 .
Snippet: T-complex protein 10A homolog 2 (TCP10L) was previously demonstrated to be a potential tumor suppressor in human hepatocellular carcinoma (HCC).
Affiliation: State Key Laboratory of Genetic Engineering, Institute of Genetics, School of Life Sciences, Fudan University, Shanghai 200433, P. R. China. Cardiovascular Research Institute and Department of Physiology, University of California, San Francisco, San Francisco, CA 94158, USA. .
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4: Title: TCP10L acts as a tumor suppressor by inhibiting cell proliferation in hepatocellular carcinoma.
Authors: Zuo, Jie, et.al. .
Journal: Biochemical and biophysical research communications (Biochem Biophys Res Commun), Vol. 446 (1): 61-7, 2014 .
Snippet: Overexpression of TCP10L in HCC cells suppressed colony formation, inhibited cell cycle progression through G0/G1 phase, and attenuated cell growth in vivo.
Affiliation: State Key Laboratory of Genetic Engineering, Institute of Genetics, Fudan University, Shanghai 200433, PR China. State Key Laboratory of Genetic Engineering, Institute of Genetics, Fudan University, Shanghai 200433, PR China; Institutes of Biomedical Sciences, Fudan University, Shanghai 200032, PR China. Electronic address: longyu@fudan.edu.cn. .
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5: Title: Identification of TCP10L as primate-specific gene derived via segmental duplication and homodimerization of TCP10L through the leucine zipper motif.
Authors: Zhong, Zhaomin, et.al. .
Journal: Molecular biology reports (Mol Biol Rep), Vol. 35 (2): 171-8, 2008 .
Snippet: In Hela cells, both the exogenous wild type TCP10L and endogenous TCP10L were detected on nuclei with immunofluorescence assay.
Affiliation: State Key Laboratory of Genetic Engineering, Institute of Genetics, School of Life Science, Fudan University, Shanghai, 200433, People's Republic of China. .
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6: Title: TCP10L is expressed specifically in spermatogenic cells and binds to death associated protein kinase-3.
Authors: Yu, Hongxiu, et.al. .
Journal: International journal of andrology (Int J Androl), Vol. 28 (3): 163-70, 2005 .
Snippet: In addition, we identified death associated protein kinase 3 (DAPK-3/ZIP kinase) as a binding partner for TCP10L by yeast two-hybrid screening, followed with immunoprecipitation and subcellular localization experiments.
Affiliation: State Key Laboratory of Genetic Engineering, Institute of Genetics, School of Life Sciences, Fudan University, Shanghai 200433, China. .
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7: Title: Human liver specific transcriptional factor TCP10L binds to MAD4.
Authors: Jiang, Dao-Jun, et.al. .
Journal: Journal of biochemistry and molecular biology (J Biochem Mol Biol), Vol. 37 (4): 402-7, 2004 .
Snippet: As MAD4 is a member of the MAD family, which antagonizes the functions of MYC and promotes cell differentiation, the biological function of the interaction between TCP10L and MAD4 may be to maintain the differentiation state in liver cells.
Affiliation: State Key Laboratory of Genetic Engineering, Institute of Genetics, School of Life Science, Fudan University, Shanghai 200433, P.R. China. .
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8: Title: Identification of a novel liver-specific expressed gene, TCP10L, encoding a human leucine zipper protein with transcription inhibition activity.
Authors: Chen, Zheng, et.al. .
Journal: Journal of human genetics (J Hum Genet), Vol. 48 (11): 556-63, 2003 .
Snippet: Based on a differentially displayed cDNA fragment, which was down regulated in hepatoma tissues, we cloned a novel cDNA of 957 bp, TCP10L (T-complex protein 10 like), from the human liver cDNA library.
Affiliation: State Key Laboratory of Genetic Engineering, Institute of Genetics, School of Life Science, Fudan University, 220 Handan Road, 200433, Shanghai, P.R. China. .
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