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8 documents found
1: Title: Genome-wide association study of loss of heterozygosity and metastasis-free survival in breast cancer patients.
Authors: Deryusheva, I V, et.al. .
Journal: Experimental oncology (Exp Oncol), Vol. 39 (2): 145-150, 2017 .
Snippet: The association analysis identified four genes: EDA2R, PGK1, TAF9B and CYSLTR1 that demonstrated the presence of LOH associated with metastasis-free survival (log-rank test, p < 0.03).
Affiliation: Cancer Research Institute, Tomsk National Research Medical Center of the Russian Academy of Sciences, Tomsk 634050, Russia. Institute of Transfusion Medicine and Immunology, Medical Faculty Mannheim, Heidelberg University; German Red Cross Blood Service Baden-Württemberg - Hessen; Mannheim, Germany. National Research Tomsk State University, Tomsk 634050, Russia. .
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2: Title: Comparative transcriptomic analysis identifies reprogramming and differentiation genes differentially expressed in UiPSCs and ESCs.
Authors: Shi, Liang, et.al. .
Journal: Bioscience trends (Biosci Trends), Vol. 11 (3): 355-359, 2017 .
Snippet: Furthermore, four genes associated with reprogramming and differentiation including neuronatin (NNAT), piwilike RNA-mediated gene silencing 2 (PIWIL2), early growth response 1 (EGR1) and TATA-box binding protein associated factor 9b (TAF9B) were validated by quantitative real-time PCR (qRT-PCR) assays.
Affiliation: School of Medicine and Life Sciences, University of Jinan-Shandong Academy of Medical Sciences. Key Laboratory for Rare Disease Research of Shandong Province, Key Laboratory for Biotech Drugs of the Ministry of Health, Shandong Medical Biotechnological Center, Shandong Academy of Medical Sciences. Guangzhou Biocare Institute of Cancer. .
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3: Title: Bioinformatics investigation of therapeutic mechanisms of Xuesaitong capsule treating ischemic cerebrovascular rat model with comparative transcriptome analysis.
Authors: Liao, Jiangquan, et.al. .
Journal: American journal of translational research (Unknown Journal), Vol. 8 (5): 2438-49, 2016 .
Snippet: ANTXR2, FHL3, PRCP, TYROBP, TAF9B, FGFR2, BCL11B, RB1CC1 and MBNL2 were the pivotal genes and possible action sites of XST therapeutic mechanisms.
Affiliation: Guang'anmen Hospital, China Academy of Chinese Medical SciencesNo. 5 Beixiange Street, Xicheng District, Beijing 100053, China; Graduate School, Beijing University of Chinese MedicineNo. 11 Beisanhuan East Road, Chaoyang District, Beijing 100029, China. Hubei University of Chinese Medicine No. 1 Huangjiahu West Road, Hongshan District, Wuhan 430065, China. Guang'anmen Hospital, China Academy of Chinese Medical Sciences No. 5 Beixiange Street, Xicheng District, Beijing 100053, China. .
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4: Title: Identification of Intellectual Disability Genes in Female Patients with a Skewed X-Inactivation Pattern.
Authors: Fieremans, Nathalie, et.al. .
Journal: Human mutation (Hum Mutat), Vol. 37 (8): 804-11, 2016 .
Snippet: Moreover, variants likely accounting for skewing only were detected in MED12, HDAC8, and TAF9B.
Affiliation: Human Genome Laboratory, Department of Human Genetics, KU Leuven, Belgium. Human Genome Laboratory, VIB Center for the Biology of Disease, Leuven, Belgium. Center for Human Genetics, University Hospitals Leuven, KU Leuven, Leuven, Belgium. Het GielsBos, Gierle, Belgium and Department of Neurology, University Hospital of Antwerp (UZA), Antwerp, Belgium. Genetics Unit, Hospital Universitario y Politecnico La Fe, Valencia, Spain. Human Genome Sequencing Center, Baylor College of Medicine, Houston, Texas. Department of Molecular and Human Genetics, Baylor College of Medicine, Houston, Texas. Department of Pediatrics, Baylor College of Medicine, Houston, Texas. Texas Children's Hospital, Houston, Texas. .
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5: Title: p53 Represses the Oncogenic Sno-MiR-28 Derived from a SnoRNA.
Authors: Yu, Feng, et.al. .
Journal: PloS one, Vol. 10 (6): e0129190, 2015 .
Snippet: The most abundant of these, sno-miR-28, directly targets the p53-stabilizing gene, TAF9B.
Affiliation: Centre for Personalized Cancer Medicine, University of Adelaide, Adelaide, SA, Australia; Discipline of Medicine, University of Adelaide, Adelaide, SA, Australia; Centre for Cancer Biology, SA Pathology, Adelaide, SA, Australia. Discipline of Medicine, University of Adelaide, Adelaide, SA, Australia; Centre for Cancer Biology, SA Pathology, Adelaide, SA, Australia. ACRF Cancer Genomics Facility, Centre for Cancer Biology, SA Pathology, Adelaide, Australia; School of Molecular and Biomedical Science, University of Adelaide, Adelaide, Australia. Centre for Personalized Cancer Medicine, University of Adelaide, Adelaide, SA, Australia; Discipline of Medicine, University of Adelaide, Adelaide, SA, Australia. Centre for Personalized Cancer Medicine, University of Adelaide, Adelaide, SA, Australia; Discipline of Medicine, University of Adelaide, Adelaide, SA, Australia; Swinburne University of Technology, Kuching, Sarawak, Malaysia. .
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6: Title: Core promoter factor TAF9B regulates neuronal gene expression.
Authors: Herrera, Francisco J, et.al. .
Journal: eLife, Vol. 3, 2014 .
Snippet: Here, we report that the orphan TBP-associated factor TAF9B is selectively up-regulated upon in vitro motor neuron differentiation, and is required for the transcriptional induction of specific neuronal genes, while dispensable for global gene expression in murine ES cells.
Affiliation: Department of Molecular and Cell Biology, University of California, Berkeley, Berkeley, United States Howard Hughes Medical Institute, University of California, Berkeley, Berkeley, United States CIRM Center of Excellence, Li Ka Shing Center For Biomedical and Health Sciences, University of California, Berkeley, Berkeley, United States fjherrera@berkeley.edu. Department of Molecular and Cell Biology, University of California, Berkeley, Berkeley, United States Howard Hughes Medical Institute, University of California, Berkeley, Berkeley, United States CIRM Center of Excellence, Li Ka Shing Center For Biomedical and Health Sciences, University of California, Berkeley, Berkeley, United States. Department of Molecular and Cell Biology, University of California, Berkeley, Berkeley, United States. .
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7: Title: TAF6delta orchestrates an apoptotic transcriptome profile and interacts functionally with p53.
Authors: Wilhelm, Emmanuelle, et.al. .
Journal: BMC molecular biology (Bmc Mol Biol), Vol. 11, 2010 .
Snippet: Interestingly, gene expression patterns controlled by TAF6delta share similarities with, but are not equivalent to, those reported to change following TAF9 and/or TAF9b depletion.
Affiliation: RNA Group, Département de Microbiologie et d'Infectiologie, Faculté de Médecine et Sciences de la Santé, Université de Sherbrooke, Sherbrooke, Québec J1H 5N4, Canada. .
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8: Title: TAF9b (formerly TAF9L) is a bona fide TAF that has unique and overlapping roles with TAF9.
Authors: Frontini, Mattia, et.al. .
Journal: Molecular and cellular biology (Mol Cell Biol), Vol. 25 (11): 4638-49, 2005 .
Snippet: We observed a differential induction of TAF9 and TAF9b during apoptosis that, together with their different ability to stabilize p53, points to distinct requirements for the two proteins in gene regulation.
Affiliation: Department of Transcription, Institut de Génétique et de Biologie Moléculaire et Cellulaire, UMR 7104, BP 10142, 67404 Illkirch Cedex, CU de Strasbourg, France. .
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