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17 documents found
1: Title: Sorting nexin 9 (SNX9) regulates levels of the transmembrane disintegrin and metalloproteinase (ADAM)-9 at the cell surface.
Journal: The Journal of biological chemistry (J Biol Chem), 2018 .
Snippet: We confirmed previous findings of an interaction between ADAM9 and the intracellular sorting protein, sorting nexin 9 (SNX9), as well as its close homolog SNX18.
Affiliation: University of Copenhagen, Denmark. University Kiel, Germany. Aarhus University. Biotech Research and Innovation Center (BRIC), University of Copenhagen, Denmark. .
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2: Title: SNX18 regulates ATG9A trafficking from recycling endosomes by recruiting Dynamin-2.
Authors: Søreng, Kristiane, et.al. .
Journal: EMBO reports (Embo Rep), 2018 .
Snippet: Here, we show that the membrane remodelling protein SNX18, previously identified as a positive regulator of autophagy, regulates ATG9A trafficking from recycling endosomes.
Affiliation: Deparment of Molecular Medicine, Institute of Basic Medical Sciences and Centre for Cancer Cell Reprogramming, Institute of Clinical Medicine Faculty of Medicine University of Oslo, Oslo, Norway. Molecular Cell Biology of Autophagy Group, Francis Crick Institute, London, UK. Department of Medical Biochemistry and Biophysics, Umeå University, Umeå, Sweden. Deparment of Molecular Medicine, Institute of Basic Medical Sciences and Centre for Cancer Cell Reprogramming, Institute of Clinical Medicine Faculty of Medicine University of Oslo, Oslo, Norway anne.simonsen@medisin.uio.no. .
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3: Title: SopB-Mediated Recruitment of SNX18 Facilitates Salmonella Typhimurium Internalization by the Host Cell.
Authors: Liebl, David, et.al. .
Journal: Frontiers in cellular and infection microbiology (Front Cell Infect Microbiol), Vol. 7, 2017 .
Snippet: We show that after docking at the plasma membrane S. Typhimurium triggers rapid recruitment of cytosolic SNX18, a SH3-PX-BAR domain sorting nexin protein, to the bacteria-induced membrane ruffles and to the nascent Salmonella-containing vacuole.
Affiliation: Institute for Molecular Bioscience, The University of QueenslandBrisbane, QLD, Australia. Australian Infectious Diseases Research Centre, The University of QueenslandBrisbane, QLD, Australia. School of Chemistry and Molecular Biosciences, The University of QueenslandBrisbane, QLD, Australia. School of Biomedical Sciences, The University of QueenslandBrisbane, QLD, Australia. .
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4: Title: Mib1 modulates dynamin 2 recruitment via Snx18 to promote Dll1 endocytosis for efficient Notch signaling.
Authors: Okano, Makoto, et.al. .
Journal: Genes to cells : devoted to molecular & cellular mechanisms (Genes Cells), Vol. 21 (5): 425-41, 2016 .
Snippet: Here, we show that Mib1 is involved in dynamin 2 recruitment to Dll1 and that Snx18, which interacts with dynamin 2, modestly regulates Dll1 endocytosis.
Affiliation: Graduate School of Science, Nagoya University, Nagoya, Aichi, 464-8602, Japan. Graduate School of Pharmaceutical Sciences, Chiba University, Chiba, 260-8675, Japan. Department of Immunology, Tokai University School of Medicine, Isehara, Kanagawa, 259-1193, Japan. .
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5: Title: Depletion or over-expression of Sh3px1 results in dramatic changes in cell morphology.
Authors: Hicks, Lawrence, et.al. .
Journal: Biology open (Biol Open), Vol. 4 (11): 1448-61, 2015 .
Snippet: Furthermore, efficient protrusion formation by Sh3px1 required the actin nucleation factor Wasp. Tubules and protrusions were also generated upon over-expressing the mammalian orthologs Snx18 and Snx33 in S2 cells.
Affiliation: Cellular Biology and Anatomy, Georgia Regents University, Augusta, GA 30912, USA. Laboratory of Genetics, and Laboratory of Cell and Molecular Biology, University of Wisconsin-Madison, Madison, WI 53706, USA. Cellular Biology and Anatomy, Georgia Regents University, Augusta, GA 30912, USA James and Jean Culver Vision Discovery Institute, Georgia Regents University, Augusta, GA 30912, USA. Cellular Biology and Anatomy, Georgia Regents University, Augusta, GA 30912, USA ggonsalvez@gru.edu. .
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6: Title: Viral bimolecular fluorescence complementation: a novel tool to study intracellular vesicular trafficking pathways.
Authors: Dirk, Brennan S, et.al. .
Journal: PloS one, Vol. 10 (4): e0125619, 2015 .
Snippet: Using the F2A cleavage site from the foot and mouth disease virus we generated a viral BiFC expression vector capable of concurrent expression of Nef and host cellular proteins; PACS-1, MHC-I and SNX18.
Affiliation: Department of Microbiology and Immunology, The University of Western Ontario, Schulich School of Medicine and Dentistry, London, Ontario, Canada. .
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7: Title: Myosin Vb uncoupling from RAB8A and RAB11A elicits microvillus inclusion disease.
Authors: Knowles, Byron C, et.al. .
Journal: The Journal of clinical investigation (J Clin Invest), Vol. 124 (7): 2947-62, 2014 .
Snippet: Using surface biotinylation and dual immunofluorescence staining in MYO5B-KD cells expressing mutant forms of MYO5B, we observed that early microvillus inclusions were positive for the sorting marker SNX18 and derived from apical membrane internalization.
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8: Title: FIP5 phosphorylation during mitosis regulates apical trafficking and lumenogenesis.
Authors: Li, Dongying, et.al. .
Journal: EMBO reports (Embo Rep), Vol. 15 (4): 428-37, 2014 .
Snippet: The interaction of FIP5 with SNX18, which is required for the formation of apical endocytic carriers, is inhibited by GSK-3 phosphorylation at FIP5-T276.
Affiliation: Department of Cell and Developmental Biology, School of Medicine, Anschutz Medical Campus University of Colorado Denver, Aurora, CO, USA. .
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9: Title: SNX18 tubulates recycling endosomes for autophagosome biogenesis.
Authors: Knævelsrud, Helene, et.al. .
Journal: Autophagy, Vol. 9 (10): 1639-41, 2013 .
Snippet: Indeed, the PX-BAR protein SNX18 was identified as a positive regulator of autophagosome formation using an image-based siRNA screen.
Affiliation: Institute of Basic Medical Sciences; University of Oslo; Oslo, Norway. Department of Medical Biochemistry and Biophysics; Umeå University; Umeå, Sweden. .
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10: Title: Membrane remodeling by the PX-BAR protein SNX18 promotes autophagosome formation.
Authors: Knævelsrud, Helene, et.al. .
Journal: The Journal of cell biology (J Cell Biol), Vol. 202 (2): 331-49, 2013 .
Snippet: The PX-BAR protein SNX18 was identified as a positive regulator of autophagosome formation, and its Drosophila melanogaster homologue SH3PX1 was found to be required for efficient autophagosome formation in the larval fat body.
Affiliation: Institute of Basic Medical Sciences, University of Oslo, 0316 Oslo, Norway. helene.knavelsrud@ibv.uio.no .
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11: Title: SNX9, SNX18 and SNX33 are required for progression through and completion of mitosis.
Authors: Ma, Maggie P C, et.al. .
Journal: Journal of cell science (J Cell Sci), Vol. 125 (Pt 18): 4372-82, 2012 .
Snippet: Here, we report that the sorting nexin 9 (SNX9) subfamily members - SNX9, SNX18 and SNX33 - are required for progression and completion of mitosis.
Affiliation: Children's Medical Research Institute, The University of Sydney, Westmead, NSW 2145, Australia. .
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12: Title: Interaction between FIP5 and SNX18 regulates epithelial lumen formation.
Authors: Willenborg, Carly, et.al. .
Journal: The Journal of cell biology (J Cell Biol), Vol. 195 (1): 71-86, 2011 .
Snippet: We show that FIP5 mediates the transport of apical proteins from apical endosomes to the apical plasma membrane and, along with SNX18, is required for the early stages of apical lumen formation.
Affiliation: Department of Cell and Developmental Biology, University of Colorado Denver, Aurora, CO 80045, USA. .
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13: Title: Expression of sorting nexin 18 (SNX18) is dynamically regulated in developing spinal motor neurons.
Authors: Nakazawa, Shiori, et.al. .
Journal: The journal of histochemistry and cytochemistry : official journal of the Histochemistry Society (J Histochem Cytochem), Vol. 59 (2): 202-13, 2011 .
Snippet: Here the authors found that SNX18, which belongs to the Src-homology-3-PX-Bin/Amphiphysin/Rvs domain-containing SNX subfamily, was specifically expressed in differentiating motor neurons in the chick and mouse embryonic spinal cord.
Affiliation: Laboratory of Neuroscience, Faculty of Pharmaceutical Sciences, Hokkaido University, Sapporo, Japan. .
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14: Title: The SNX-PX-BAR family in macropinocytosis: the regulation of macropinosome formation by SNX-PX-BAR proteins.
Authors: Wang, Jack T H, et.al. .
Journal: PloS one, Vol. 5 (10): e13763, 2010 .
Snippet: SNX1, SNX5, SNX9, and SNX18 were also found to associate with early-stage macropinosomes within 5 minutes following organelle formation.
Affiliation: Institute for Molecular Bioscience and Australia Research Council, Centre of Excellence in Bioinformatics, The University of Queensland, St Lucia, Brisbane, Australia. .
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15: Title: The E3 ubiquitin ligase Itch regulates sorting nexin 9 through an unconventional substrate recognition domain.
Authors: Baumann, Claudia, et.al. .
Journal: The FEBS journal (Febs J), Vol. 277 (13): 2803-14, 2010 .
Snippet: SNX18, a second member of the SNX family containing an SH3 domain, was also found to bind to Itch.
Affiliation: Ludwig Institute for Cancer Research Ltd, University of Lausanne, Lausanne, Switzerland. Claudia.Baumann@licr.unil.ch .
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16: Title: SNX18 shares a redundant role with SNX9 and modulates endocytic trafficking at the plasma membrane.
Authors: Park, Joohyun, et.al. .
Journal: Journal of cell science (J Cell Sci), Vol. 123 (Pt 10): 1742-50, 2010 .
Snippet: Like SNX9, SNX18 interacts with dynamin and stimulates the basal GTPase activity of dynamin.
Affiliation: Department of Biomedical Sciences, Seoul National University, College of Medicine, Seoul 110-799, South Korea. .
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17: Title: SNX18 is an SNX9 paralog that acts as a membrane tubulator in AP-1-positive endosomal trafficking.
Authors: Håberg, Karin, et.al. .
Journal: Journal of cell science (J Cell Sci), Vol. 121 (Pt 9): 1495-505, 2008 .
Snippet: However, contrary to SNX9, which primarily acts in clathrin-mediated endocytosis at the plasma membrane, endogenous SNX18 localized to AP-1- and PACS1-positive endosomal structures, which were devoid of clathrin and resistant to Brefeldin A. Moreover, a gamma-adaptin recognition motif was defined in a low-complexity region of SNX18, and a complex of endogenous SNX18 and AP-1 could be immunoprecipitated after Brefeldin A treatment.
Affiliation: Department of Medical Biochemistry and Biophysics, Umeå University, Umeå, Sweden. .
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