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10 documents found
1: Title: Hypoxia-induced Rab11-family interacting protein 4 expression promotes migration and invasion of colon cancer and correlates with poor prognosis.
Authors: Wang, Jian-Zhang, et.al. .
Journal: Molecular medicine reports (Mol Med Report), Vol. 17 (3): 3797-3806, 2018 .
Snippet: Furthermore, the results of a co‑immunoprecipitation assay and western blot analysis demonstrated that Rab11‑FIP4 interacted with Rab11 and insulin‑like growth factor 1 receptor, and increased the phosphorylation of extracellular signal‑regulated kinase 1/2 and AKT serine/threonine kinase.
Affiliation: Department of Gastroenterology, The Second Affiliated Hospital and Yuying Children's Hospital of Wenzhou Medical University, Wenzhou, Zhejiang 325027, P.R. China. Department of Gastroenterology, Ruian People's Hospital, Wenzhou, Zhejiang 325200, P.R. China. .
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2: Title: Analytical Strategy to Prioritize Alzheimer's Disease Candidate Genes in Gene Regulatory Networks Using Public Expression Data.
Journal: Journal of Alzheimer's disease : JAD (J Alzheimers Dis), Vol. 59 (4): 1237-1254, 2017 .
Snippet: Moreover, less intensive studied AD candidates (STX2, HLA-F, HLA-C, RAB11FIP4, ARAP3, AP2A2, ATP2B4, ITPR2, and ATP2A3) are also involved in neurotransmission, providing new insights into the underlying mechanism.
Affiliation: Fraunhofer Institute for Algorithms and Scientific Computing (SCAI), Schloss Birlinghoven, Sankt Augustin, Germany. Rheinische Friedrich-Wilhelms-Universität Bonn, Bonn-Aachen International Center for Information Technology, Bonn, Germany. University of Applied Sciences Koblenz, RheinAhrCampus, Remagen, Germany. Dana-Farber Cancer Institute, Boston, MA, USA. .
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3: Title: High Rab11-FIP4 expression predicts poor prognosis and exhibits tumor promotion in pancreatic cancer.
Authors: He, Yun, et.al. .
Journal: International journal of oncology (Int J Oncol), Vol. 50 (2): 396-404, 2017 .
Snippet: Rab11-FIP4 knockout in PANC-1 cells inhibited cell growth, invasion and metastasis, and arrested cell cycle progression, but did not alter apoptosis.
Affiliation: Department of Gastroenterology, The Second Affiliated Hospital and Yuying Children's Hospital of Wenzhou Medical University, Wenzhou, Zhejiang 325000, P.R. China. Department of Pathology, The Second Affiliated Hospital and Yuying Children's Hospital of Wenzhou Medical University, Wenzhou, Zhejiang 325000, P.R. China. Department of Surgery, The First Affiliated Hospital of Wenzhou Medical University, Wenzhou, Zhejiang 325000, P.R. China. Department of General Surgery, The Second Affiliated Hospital and Yuying Children's Hospital of Wenzhou Medical University, Wenzhou, Zhejiang 325000, P.R. China. .
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4: Title: Mycobacterium bovis BCG Interferes with miR-3619-5p Control of Cathepsin S in the Process of Autophagy.
Authors: Pawar, Kamlesh, et.al. .
Journal: Frontiers in cellular and infection microbiology (Front Cell Infect Microbiol), Vol. 6, 2016 .
Snippet: The lysosomal cysteine protease Cathepsin S (CTSS) and Rab11 family-interacting protein 4 (RAB11FIP4) were up-regulated and were considered to be connected to lysosomal trafficking and autophagy.
Affiliation: Department of Veterinary Medicine, Institute of Veterinary Biochemistry, Freie Universität Berlin Berlin, Germany. .
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5: Title: Systematic transcriptome analysis reveals tumor-specific isoforms for ovarian cancer diagnosis and therapy.
Authors: Barrett, Christian L, et.al. .
Journal: Proceedings of the National Academy of Sciences of the United States of America (Proc Natl Acad Sci U S A), Vol. 112 (23): E3050-7, 2015 .
Snippet: Of the 671 isoforms, the topmost 5% (n = 33) ranked based on having tumor-specific or highly restricted normal tissue expression by RT-qPCR analysis are enriched for oncogenic, stem cell/cancer stem cell, and early development loci--including ETV4, FOXM1, LSR, CD9, RAB11FIP4, and FGFRL1.
Affiliation: Moores Cancer Center, Department of Pediatrics and Rady Children's Hospital. Moores Cancer Center, Bioinformatics and Systems Biology. Institute for Genomic Medicine. Department of Medicine. Moores Cancer Center, Department of Medicine, Sanford Consortium for Regenerative Medicine, University of California, San Diego, La Jolla, CA 92093 dcarson@ucsd.edu. Moores Cancer Center, Department of Pediatrics and Rady Children's Hospital, Institute for Genomic Medicine. .
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6: Title: Hypoxia upregulates Rab11-family interacting protein 4 through HIF-1α to promote the metastasis of hepatocellular carcinoma.
Authors: Hu, F, et.al. .
Journal: Oncogene, Vol. 34 (49): 6007-17, 2015 .
Snippet: Hypoxia-inducible factor 1α (HIF-1α), as a crucial regulator of transcriptional responses to hypoxia, induces the expression of multiple target genes involved in different steps of HCC metastatic process.
Affiliation: Shanghai Medical College of Fudan University, Shanghai, China. State Key Laboratory of Oncogenes and Related Genes, Shanghai Cancer Institute, Renji Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, China. Key Laboratory for Carcinogenesis and Cancer Invasion, The Chinese Ministry of Education, Liver Cancer Institute, Zhongshan Hospital and Shanghai Medical College, Fudan University, Shanghai, China. Department of Pathophysiology, Guangdong Medical College, Dongguan, China. Basic Medical Research Centre, Medical College of Nantong University, Nantong, China. .
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7: Title: HCMV-encoded glycoprotein M (UL100) interacts with Rab11 effector protein FIP4.
Journal: Traffic (Copenhagen, Denmark) (Traffic), Vol. 10 (10): 1439-57, 2009 .
Snippet: Recently, it has been shown that FIP4 interactions with Rab11 and additionally with Arf6/Arf5 are important for the vesicular transport of proteins in the endosomal recycling compartment (ERC) and during cytokinesis.
Affiliation: Department of Microbiology, University of Alabama at Birmingham, CHB160, Birmingham, AL 35233, USA. .
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8: Title: Expression analysis of genes lying in the NF1 microdeletion interval points to four candidate modifiers for neurofibroma formation.
Authors: Bartelt-Kirbach, B, et.al. .
Journal: Neurogenetics, Vol. 10 (1): 79-85, 2009 .
Snippet: Expression analysis of 13 genes of the microdeletion region in dermal neurofibromas and other tissues revealed four candidates for the modification of neurofibroma formation: CENTA2, RAB11FIP4, C17orf79, and UTP6.
Affiliation: Institute of Human Genetics, University of Ulm, Ulm, Germany. .
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9: Title: Mapping of a novel type III variant of Knobloch syndrome (KNO3) to chromosome 17q11.2.
Authors: Khaliq, Shagufta, et.al. .
Journal: American journal of medical genetics. Part A (Am J Med Genet A), Vol. 143A (23): 2768-74, 2007 .
Snippet: Mutation analysis of three candidate genes (UNC119, MYO1D, and RAB11FIP4) within the disease region did not identify any disease-associated mutation in affected individuals.
Affiliation: Biomedical and Genetic Engineering Division, Dr. A. Q. Khan Research Laboratories, Islamabad, Pakistan. .
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10: Title: Rab11-FIP4 interacts with Rab11 in a GTP-dependent manner and its overexpression condenses the Rab11 positive compartment in HeLa cells.
Authors: Wallace, Deborah M E, et.al. .
Journal: Biochemical and biophysical research communications (Biochem Bioph Res Co), Vol. 299 (5): 770-9, 2002 .
Snippet: Rab11-FIP4 interacts with Rab11 in a GTP-dependent manner and its overexpression condenses the Rab11 positive compartment in HeLa cells.
Affiliation: Cell and Molecular Biology Laboratory, Department of Biochemistry, University College Cork, Lee Maltings, Prospect Row, Cork, Ireland. .
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