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27 documents found
1: Title: Oncogene c-MYC Controls the Expression of PHF10 Subunit of PBAF Chromatin Remodeling Complex in SW620 Cell Line.
Authors: Tatarskiy, Eu V, et.al. .
Journal: Doklady. Biochemistry and biophysics (Dokl Biochem Biophys), Vol. 484 (1): 66-68, 2019 .
Snippet: We showed that the PHF10 expression in cells of different lines is activated by the c-MYC oncogene.
Affiliation: Institute of Gene Biology, Russian Academy of Sciences, 119334, Moscow, Russia. Institute of Gene Biology, Russian Academy of Sciences, 119334, Moscow, Russia. so2615nat@gmail.com. .
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2: Title: Stability of Chromatin Remodeling Complex Subunits Is Determined by Their Phosphorylation Status.
Authors: Azieva, A M, et.al. .
Journal: Doklady. Biochemistry and biophysics (Dokl Biochem Biophys), Vol. 479 (1): 66-68, 2018 .
Snippet: It was found that, in the differentiated cells of mouse brain, the level of core (Brg1 and BAF155) and specific (BRD7, BAF180, and PHF10) subunits of the chromatin-remodeling complex PBAF is reduced compared to the undifferentiated proliferating cells.
Affiliation: Institute of Gene Biology, Russian Academy of Sciences, Moscow, 119334, Russia. Kurchatov Institute National Research Center, Moscow, 123182, Russia. Engelhardt Institute of Molecular Biology, Russian Academy of Sciences, Moscow, Russia. Institute of Gene Biology, Russian Academy of Sciences, Moscow, 119334, Russia. so2615nat@gmail.com. .
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3: Title: Stability of the PHF10 subunit of PBAF signature module is regulated by phosphorylation: role of β-TrCP.
Authors: Tatarskiy, Victor V, et.al. .
Journal: Scientific reports (Sci Rep), Vol. 7 (1): 5645, 2017 .
Snippet: PHF10 have different domain structure and two of them (PHF10-S isoforms) lack C-terminal PHD domains, which enables their phosphorylation by CK-1.
Affiliation: Laboratory of Tumor Cell Death, N.N. Blokhin Russian Cancer Research Center, Kashirskoye Shosse 24, Moscow, 115478, Russia. Department of Transcription Factors, Engelhardt Institute of Molecular Biology, Russian Academy of Sciences, Vavilov Str. 32, Moscow, 119991, Russia. Laboratory of Structure Bioinformatics, Institute of Biomedical Chemistry (IBMC), Pogodinskaya street 10 building 8, Moscow, 119121, Russia. Department of Eukariotic Transcription Factors, Institute of Gene Biology, Russian Academy of Sciences, Vavilov Str. 34/5, Moscow, 119991, Russia. sonjag@molbiol.edu.ru. Department of Transcription Factors, Engelhardt Institute of Molecular Biology, Russian Academy of Sciences, Vavilov Str. 32, Moscow, 119991, Russia. sonjag@molbiol.edu.ru. Department of Eukariotic Transcription Factors, Institute of Gene Biology, Russian Academy of Sciences, Vavilov Str. 34/5, Moscow, 119991, Russia. so2615nat@gmail.com. .
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4: Title: The BAF45a/PHF10 subunit of SWI/SNF-like chromatin remodeling complexes is essential for hematopoietic stem cell maintenance.
Authors: Krasteva, Veneta, et.al. .
Journal: Experimental hematology (Exp Hematol), Vol. 48, 2017 .
Snippet: Here we provide evidence that BAF45a/PHF10, a subunit of SWI/SNF-like chromatin remodeling complexes, is essential for adult hemopoietic stem cell maintenance and myeloid lineage development.
Affiliation: IRIC, Institute for Research in Immunology and Cancer, Montreal, QC, Canada; Department of Pathology and Cell Biology, Faculty of Medicine, Université de Montréal, Montreal, QC, Canada. Department of Pathology, Stanford University School of Medicine, Stanford, CA; Developmental Biology, Stanford University School of Medicine, Stanford, CA. IRIC, Institute for Research in Immunology and Cancer, Montreal, QC, Canada; Department of Pathology and Cell Biology, Faculty of Medicine, Université de Montréal, Montreal, QC, Canada. Electronic address: j.lessard.1@umontreal.ca. .
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5: Title: [Ratio of transcription factor PHF10 splice variants in lymphocytes as a molecular marker of Parkinson's disease].
Authors: Soshnikova, N V, et.al. .
Journal: Molekuliarnaia biologiia (Mol Biol (mosk)), Vol. 50 (4): 695-702, 2016 Jul-Aug .
Snippet: PHF10 is one of the most important regulatory subunits of the SWI/SNF chromatin-remodeling protein complex, which controls the gene function and chromatin state in neurons.
Affiliation: Institute of Gene Biology, Russian Academy of Sciences, Moscow, 119334 Russia. Koltsov Institute of Developmental Biology, Russian Academy of Sciences, Moscow, 119334 Russia. annakolacheva@gmail.com. Kazan State Medical University, Ministry of Health of the Russian Federation, Kazan, 420012 Russia. Сenter of Early Diagnosis of Neurodegenerative Disorders, Kazan, 420100 Russia. Center for Extrapyramidal Pathology and Botulinum Therapy, Kazan Hospital for War Veterans, Ministry of Health of the Republic of Tatarstan, Kazan, 420039 Russia. Engelhardt Institute of Molecular Biology, Russian Academy of Sciences, Moscow, 119991 Russia. .
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6: Title: [PHF10 isoforms are phosphorylated in the PBAF mammalian chromatin remodeling complex].
Authors: Brechalov, A V, et.al. .
Journal: Molekuliarnaia biologiia (Mol Biol (mosk)), Vol. 50 (2): 320-6, 2016 Mar-Apr .
Snippet: Phosphorylation of PHF10 isoforms occurs while they are incorporated as a subunit of the PBAF complex, and therefore phosphorylation of PHF10 isoforms may play an essential role in regulation of PBAF complex's function and mechanism of action.
Affiliation: Institute of Gene Biology, Russian Academy of Sciences, Moscow, 119334, Russia. Engelhardt Institute of Molecular Biology, Russian Academy of Sciences, Moscow, 119991, Russia. so2615nat@gmail.com. .
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7: Title: The level of the Phf10 protein, a PBAF chromatin-remodeling complex subunit, correlates with the Mts1/S100A4 expression in human cancer cell lines.
Authors: Soshnikova, N V, et.al. .
Journal: Doklady. Biochemistry and biophysics (Dokl Biochem Biophys), Vol. 467 (1): 162-4, 2016 .
Snippet: We studied the expression of S100A4 and the total PHF10 protein in some cell lines.
Affiliation: Engelhardt Institute of Molecular Biology, Russian Academy of Sciences, ul. Vavilova 32, Moscow, 119991, Russia. so2615nat@gmail.com. Engelhardt Institute of Molecular Biology, Russian Academy of Sciences, ul. Vavilova 32, Moscow, 119991, Russia. .
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8: Title: The SWI/SNF Subunit INI1 Contains an N-Terminal Winged Helix DNA Binding Domain that Is a Target for Mutations in Schwannomatosis.
Authors: Allen, Mark D, et.al. .
Journal: Structure (London, England : 1993) (Structure), Vol. 23 (7): 1344-9, 2015 .
Snippet: Here, we show that mutations in INI1 that cause schwannomatosis target a hitherto unidentified N-terminal winged helix DNA binding domain that is also present in the BAF45a/PHF10 subunit of the SWI/SNF complex.
Affiliation: MRC Laboratory of Molecular Biology, Francis Crick Avenue, Cambridge Biomedical Campus, Cambridge CB2 0QH, UK. Centre for Advanced Cancer Therapies, Department of Microbiology, Cell and Tumour Biology and Science for Life Laboratory, Karolinska Institutet, Tomtebodavägen 23, Stockholm 171 65, Sweden. MRC Laboratory of Molecular Biology, Francis Crick Avenue, Cambridge Biomedical Campus, Cambridge CB2 0QH, UK. Electronic address: mxb@mrc-lmb.cam.ac.uk. .
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9: Title: Mammalian cells contain two functionally distinct PBAF complexes incorporating different isoforms of PHF10 signature subunit.
Authors: Brechalov, Alexander V, et.al. .
Journal: Cell cycle (Georgetown, Tex.) (Cell Cycle), Vol. 13 (12): 1970-9, 2014 .
Snippet: PBAF complexes containing different PHF10 isoforms can bind to the promoters of the same genes but produce different effects on the recruitment of Pol II to the promoter and on the level of gene transcription.
Affiliation: Department of Eukaryote Transcription Factors; Institute of Gene Biology; Russian Academy of Sciences; Moscow, Russia; Department of Transcription Factors; Institute of Molecular Biology; Russian Academy of Sciences; Moscow, Russia. Department of Eukaryote Transcription Factors; Institute of Gene Biology; Russian Academy of Sciences; Moscow, Russia. .
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10: Title: Delineation of candidate genes responsible for structural brain abnormalities in patients with terminal deletions of chromosome 6q27.
Authors: Peddibhotla, Sirisha, et.al. .
Journal: European journal of human genetics : EJHG (Eur J Hum Genet), Vol. 23 (1): 54-60, 2015 .
Snippet: The smallest region of overlap spans 1.7 Mb and contains DLL1, THBS2, PHF10, and C6orf70 (ERMARD) that are plausible candidates for the causation of structural brain abnormalities.
Affiliation: Department of Molecular and Human Genetics, Baylor College of Medicine, Houston, TX, USA. 1] Department of Molecular and Human Genetics, Baylor College of Medicine, Houston, TX, USA [2] Department of Biological Regulation, Weizmann Institute of Science, Rehovot, Israel. Department of Radiology, Baylor College of Medicine, Houston, TX, USA. 1] Department of Pediatrics, Division of Neurology and Developmental Neuroscience, Baylor College of Medicine, Houston, TX, USA [2] Texas Children's Hospital, Houston, TX, USA. Department of Pediatrics, University of Texas, Southwestern Medical Center, Dallas, TX, USA. Parkview Cytogenetics and Northeast Indiana Genetic Counseling Center, Fort Wayne, IN, USA. Regional Genetics Program, Conseillère en génétique agréée, Programme régional de Génétique, Ottawa, Ontario, Canada. Scott and White Memorial Hospital, Temple, TX, USA. Especially for Children, Austin, TX, USA. Department of Pediatrics, Vanderbilt School of Medicine, Nashville, TN, USA. Department of Neurosurgery, University of Texas Medical Branch, Galveston, TX, USA. 1] Department of Molecular and Human Genetics, Baylor College of Medicine, Houston, TX, USA [2] Allina Medical Laboratories, Minneapolis, MN, USA. .
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11: Title: Periventricular heterotopia in 6q terminal deletion syndrome: role of the C6orf70 gene.
Authors: Conti, Valerio, et.al. .
Journal: Brain : a journal of neurology (Brain), Vol. 136 (Pt 11): 3378-94, 2013 .
Snippet: Silencing of the contiguous Phf10 or Dll1 genes only produced slightly delayed migration but not periventricular nodular heterotopia.
Affiliation: 1 Paediatric Neurology and Neurogenetics Unit and Laboratories, A. Meyer Children's Hospital - Department of Neuroscience, Pharmacology and Child Health, University of Florence, 50139, Florence, Italy. .
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12: Title: SAYP and Brahma are important for 'repressive' and 'transient' Pol II pausing.
Authors: Vorobyeva, Nadezhda E, et.al. .
Journal: Nucleic acids research (Nucleic Acids Res), Vol. 40 (15): 7319-31, 2012 .
Snippet: Drosophila SAYP, a homologue of human PHF10/BAF45a, is a metazoan coactivator associated with Brahma and essential for its recruitment on the promoter.
Affiliation: Group of Transcription and mRNA Transport, Institute of Gene Biology, Russian Academy of Sciences, Moscow 119334, Russia. nvorobyova@gmail.com .
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13: Title: Genetic and functional evaluation of the role of DLL1 in susceptibility to visceral leishmaniasis in India.
Authors: Mehrotra, Sanjana, et.al. .
Journal: Infection, genetics and evolution : journal of molecular epidemiology and evolutionary genetics in infectious diseases (Infect Genet Evol), Vol. 12 (6): 1195-201, 2012 .
Snippet: Twenty-one single nucleotide polymorphisms (SNPs) at PHF10/C6orf70/DLL1/FAM120B/PSMB1/TBP were genotyped in 941 cases and 992 controls.
Affiliation: Institute of Medical Sciences, Banaras Hindu University, Varanasi, OS 221 005, India. anniebhu19@gmail.com .
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14: Title: SS18 together with animal-specific factors defines human BAF-type SWI/SNF complexes.
Authors: Middeljans, Evelien, et.al. .
Journal: PloS one, Vol. 7 (3): e33834, 2012 .
Snippet: By contrast,we demonstrate that human PHF10 is part of the PBAF complex, which harbors both ARID2/BAF200 and polybromo/BAF180 subunits, but not SS18 and nor the above BAF-specific subunits.
Affiliation: Department of Molecular Biology, Nijmegen Centre for Molecular Life Sciences, Radboud University, Nijmegen, The Netherlands. .
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15: Title: MicroRNA-409-3p regulates cell proliferation and apoptosis by targeting PHF10 in gastric cancer.
Authors: Li, Chenglong, et.al. .
Journal: Cancer letters (Cancer Lett), Vol. 320 (2): 189-97, 2012 .
Snippet: Taken together, these findings suggest that miR-409-3p may function as a novel tumor suppressor in GC and its anti-oncogenic activity may involve the direct targeting and inhibition of PHF10.
Affiliation: Shanghai Key Laboratory of Gastric Neoplasms, Department of Surgery, Shanghai Institute of Digestive Surgery, Ruijin Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai 200025, China. .
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16: Title: Double plant homeodomain (PHD) finger proteins DPF3a and -3b are required as transcriptional co-activators in SWI/SNF complex-dependent activation of NF-κB RelA/p50 heterodimer.
Authors: Ishizaka, Aya, et.al. .
Journal: The Journal of biological chemistry (J Biol Chem), Vol. 287 (15): 11924-33, 2012 .
Snippet: Using sensitive 293FT reporter cell clones that had integrated a SWI/SNF-dependent NF-κB reporter gene, we find in this study that the overexpression of DPF1, DPF2, DPF3a, DPF3b, and PHF10 significantly potentiates the transactivating activity of typical NF-κB dimers.
Affiliation: Division of Host-Parasite Interaction, Department of Microbiology and Immunology, Institute of Medical Science, University of Tokyo, 4-6-1 Shirokanedai, Minato-ku, Tokyo 108-8639, Japan. .
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17: Title: Transcription co-activator SAYP mediates the action of STAT activator.
Authors: Panov, Vladislav V, et.al. .
Journal: Nucleic acids research (Nucleic Acids Res), Vol. 40 (6): 2445-53, 2012 .
Snippet: We describe participation of metazoan co-activator SAYP/PHF10 in this pathway downstream of STAT.
Affiliation: Department of Regulation of Genes Expression, Institute of Gene Biology, Russian Academy of Sciences, Moscow 119334, Russia. .
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18: Title: A genome-wide meta-analysis of six type 1 diabetes cohorts identifies multiple associated loci.
Authors: Bradfield, Jonathan P, et.al. .
Journal: PLoS genetics (Plos Genet), Vol. 7 (9): e1002293, 2011 .
Snippet: The third most significantly associated SNP (rs924043, P = 8.06×10⁻⁹ lies in an intergenic region on 6q27, where the region of association is approximately 900 kb and harbors multiple genes including WDR27, C6orf120, PHF10, TCTE3, C6orf208, LOC154449, DLL1, FAM120B, PSMB1, TBP, and PCD2.
Affiliation: The Center for Applied Genomics, The Children's Hospital Philadelphia, Philadelphia, Pennsylvania, United States of America. .
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19: Title: Genetic and functional evidence implicating DLL1 as the gene that influences susceptibility to visceral leishmaniasis at chromosome 6q27.
Authors: Fakiola, Michaela, et.al. .
Journal: The Journal of infectious diseases (J Infect Dis), Vol. 204 (3): 467-77, 2011 .
Snippet: METHODS: Twenty-two single-nucleotide polymorphisms (SNPs) at genes PHF10, C6orf70, DLL1, FAM120B, PSMB1, and TBP were genotyped in 193 VL cases from 85 Sudanese families, and 8 SNPs at genes PHF10, C6orf70, DLL1, PSMB1, and TBP were genotyped in 194 VL cases from 80 Brazilian families.
Affiliation: Cambridge Institute for Medical Research and Department of Medicine, University of Cambridge School of Clinical Medicine, UK. .
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20: Title: [SAYP--a novel regulator of metazoan development].
Authors: Kuz'mina, Iu L, et.al. .
Journal: Genetika, Vol. 46 (8): 1033-40, 2010 .
Snippet: SAYP is a dual-function transcriptional coactivator of RNA polymerase II.
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