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30 documents found
1: Title: Gestational diabetes and maternal obesity are associated with epigenome-wide methylation changes in children.
Authors: Hjort, Line, et.al. .
Journal: JCI insight, Vol. 3 (17), 2018 .
Snippet: Three prepregnancy BMI-associated CpGs (cg00992687 and cg09452568 of ESM1 and cg14328641 of MS4A3) were validated in the replication cohort, while cg09109411 (PDE6A) was found to be associated with GDM status.
Affiliation: Department of Endocrinology (Diabetes and Metabolism), Rigshospitalet, Copenhagen, Denmark. Faculty of Health and Medical Sciences, University of Copenhagen, Copenhagen, Denmark. The Danish Diabetes Academy, Odense, Denmark. Centre for Food and Allergy Research, Murdoch Children's Research Institute, Melbourne, Victoria, Australia. Department of Pediatrics, Melbourne University, Melbourne, Victoria, Australia. Bioinformatics Group, Murdoch Children's Research Institute, Melbourne, Victoria, Australia. Monash Bioinformatics Platform, Monash University, Clayton, Victoria, Australia. Department of Computing and Information Systems, University of Melbourne, Melbourne, Victoria, Australia. Division of Intramural Population Health Research, Eunice Kennedy Shriver National Institute of Child Health and Human Development, Rockville, Maryland, USA. Epigenetics and Diabetes Unit, Department of Clinical Sciences, Lund University Diabetes Centre, CRC, Scania University Hospital, Malmö, Sweden. Centre for Fetal Programming, Statens Serum Institut, Copenhagen, Denmark. Cancer and Disease Epigenetics, Murdoch Children's Research Institute, Melbourne, Victoria, Australia. Cardiovascular and Metabolic Disease (CVMD) Translational Medicine Unit, Early Clinical Development, IMED Biotech Unit, AstraZeneca, Gothenburg, Sweden. .
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2: Title: Ecotropic viral integration site 1 regulates the progression of acute myeloid leukemia via MS4A3-mediated TGFβ/EMT signaling pathway.
Authors: Jiang, Min, et.al. .
Journal: Oncology letters (Oncol Lett), Vol. 16 (2): 2701-2708, 2018 .
Snippet: Previous studies have indicated that Evi1 may regulate cell proliferation, differentiation and apoptosis by inhibiting the membrane-spanning-4-domains subfamily-A member-3 (MS4A3) gene in AML.
Affiliation: Department of Anatomy, School of Basic Medical Sciences, Southern Medical University, Guangzhou, Guangdong 510515, P.R. China. Department of Pediatrics, The Fifth Hospital of Xiamen, Affiliated TongMin Hospital of Xiamen University, Xiamen, Fujian 361101, P.R. China. Department of Internal Medicine, The First College of Clinical Medical Science, China Three Gorges University, Yichang Central People's Hospital, Yichang, Hubei 443003, P.R. China. .
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3: Title: Identification of MS4A3 as a reliable marker for early myeloid differentiation in human hematopoiesis.
Authors: Ishibashi, Tomohiko, et.al. .
Journal: Biochemical and biophysical research communications (Biochem Biophys Res Commun), Vol. 495 (3): 2338-2343, 2018 .
Snippet: Further analysis revealed that a subset of CD34+CD38+CD33+ progenitor population in human adult bone marrow expressed MS4A3, and those MS4A3+ progenitors only produced granulocyte/macrophage colonies, losing erythroid colony- and mixed colony-forming capacity.
Affiliation: Department of Hematology and Oncology, Osaka University Graduate School of Medicine, Suita, Osaka 565-0871, Japan; Department of Vascular Physiology, National Cerebral and Cardiovascular Center Research Institute, Suita, Osaka 565-8565, Japan. Department of Hematology and Oncology, Osaka University Graduate School of Medicine, Suita, Osaka 565-0871, Japan. Electronic address: yokotat@bldon.med.osaka-u.ac.jp. Department of Hematology and Oncology, Osaka University Graduate School of Medicine, Suita, Osaka 565-0871, Japan; Department of Lifestyle Studies, Kobe Shoin Women's University, Kobe 657-0015, Japan. Department of Hematology and Oncology, Osaka University Graduate School of Medicine, Suita, Osaka 565-0871, Japan. Department of Hematology and Oncology, Osaka University Graduate School of Medicine, Suita, Osaka 565-0871, Japan; Department of Immunology and Cell Biology, Graduate School of Medicine and Frontier Biosciences, Osaka University, Suita, Osaka 565-0871, Japan. .
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4: Title: MS4A4A: a novel cell surface marker for M2 macrophages and plasma cells.
Authors: Sanyal, Ratna, et.al. .
Journal: Immunology and cell biology (Immunol Cell Biol), Vol. 95 (7): 611-619, 2017 .
Snippet: MS4A4A is a member of the membrane-spanning, four domain family, subfamily A (MS4A) that includes CD20 (MS4A1), FcRβ (MS4A2) and Htm4 (MS4A3).
Affiliation: Department of Biochemistry and Molecular Biology, and Snyder Institute for Chronic Diseases, University of Calgary, Calgary, Alberta, Canada. Departments of Medicine and Oncology, University of Calgary, Calgary, Alberta, Canada. Calgary Laboratory Services, Foothills Medical Centre, Calgary, Alberta, Canada. Department of Pathology and Laboratory Medicine, University of Calgary, Calgary, Alberta, Canada. Department of Oncology, University of Alberta and Cross Cancer Institute, Edmonton, Alberta, Canada. .
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5: Title: miR-125b and miR-532-3p predict the efficiency of rituximab-mediated lymphodepletion in chronic lymphocytic leukemia patients. A French Innovative Leukemia Organization study.
Authors: Gagez, Anne-Laure, et.al. .
Journal: Haematologica, Vol. 102 (4): 746-754, 2017 .
Snippet: Interestingly, both micro-ribonucleic acids were negatively correlated with MS4A1 expression, while they were positively correlated with MS4A3 and MSA47 Our results identify novel circulating predictive biomarkers for rituximab-mediated lymphodepletion efficacy in chronic lymphocytic leukemia, and suggest a novel molecular mechanism responsible for the rituximab mode of action that bridges miR-125b and miR-532-3p and CD20 family members.
Affiliation: CNRS UMR 5235, University of Montpellier, France. Department of Clinical Hematology, University Hospital Montpellier, France. INSERM, U1183, Institute of Regenerative Medicine and Biotherapy, University Hospital Montpellier, France. Henri Becquerel Center, Rouen, France. Department of Clinical Hematology, Hospital Center of Annecy, Pringy, France. Department of Biological Hematology, APHP, GHUPSSD, Avicenne Hospital, Bobigny, France. Department of Clinical Hematology, Pontchaillou Hospital, Rennes, France. Department of Bacteriology-Virology, University Hospital Montpellier, France. Department of Hematology, La Pitié Salpétrière Hospital, Paris, France. CNRS UMR 7292, François Rabelais University, University Hospital Tours, France. Department of Hematology, Cote Basque Hospital, Bayonne, France. Department of Clinical Hematology, University Hospital Estaing, Clermont-Ferrand, France. Unit of Lymphoid Hematologic Malignancies, Henri Mondor Hospital, Créteil, France. Clinical department for Osteoarticular Diseases, University Hospital Lapeyronie, Montpellier, France. CNRS UMR 5235, University of Montpellier, France g-cartron@chu-montpellier.fr. .
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6: Title: A 7-Gene Signature Depicts the Biochemical Profile of Early Prefibrotic Myelofibrosis.
Authors: Skov, Vibe, et.al. .
Journal: PloS one, Vol. 11 (8): e0161570, 2016 .
Snippet: The 7 genes included MPO, CEACAM8, CRISP3, MS4A3, CEACAM6, HEMGN, and MMP8, which are genes known to be involved in inflammation, cell adhesion, differentiation and proliferation.
Affiliation: Department of Hematology, Zealand University Hospital, Roskilde, Denmark. Department of Clinical Genetics, Odense University Hospital, Odense, Denmark. Department of Hematology X, Odense University Hospital, Odense, Denmark. Department of Pathology, Naestved Hospital, Naestved, Denmark. Department of Pathology, Lund University Hospital, Lund, Sweden. Department of Pathology, University of Aarhus, Aarhus, Denmark. Institute of Pathology, University of Cologne, Köln, Germany. Department of Hematology L, Rigshospitalet, University of Copenhagen, Copenhagen, Denmark. .
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7: Title: Gene-based aggregate SNP associations between candidate AD genes and cognitive decline.
Authors: Nettiksimmons, Jasmine, et.al. .
Journal: Age (Dordrecht, Netherlands) (Age (dordr)), Vol. 38 (2): 41, 2016 .
Snippet: Single nucleotide polymorphisms (SNPs) in and near ABCA7, BIN1, CASS4, CD2AP, CD33, CELF1, CLU, complement receptor 1 (CR1), EPHA1, EXOC3L2, FERMT2, HLA cluster (DRB5-DQA), INPP5D, MEF2C, MS4A cluster (MS4A3-MS4A6E), NME8, PICALM, PTK2B, SLC24A4, SORL1, and ZCWPW1 have been associated with Alzheimer's disease (AD) in large meta-analyses.
Affiliation: Department of Psychiatry, University of San Francisco - California, 4150 Clement Street, Box VAMC-116H, San Francisco, CA, 94121, USA. jasminen@gmail.com. California Pacific Medical Center Research Institute, Department of Epidemiology and Biostatistics, University of California - San Francisco, Mission Hall: Global Health & Clinical Sciences Building, 550 16th Street, 2nd floor, Box #0560, San Francisco, CA, 94158-2549, USA. California Pacific Medical Center Research Institute, Mission Hall: Global Health & Clinical Sciences Building, 550 16th Street, 2nd floor, Box #0560, San Francisco, CA, 94158-2549, USA. Memory and Aging Center, University of California - San Francisco, Sandler Neurosciences Center, 675 Nelson Rising Lane, Suite 190, San Francisco, CA, USA. Departments of Psychiatry, Neurology, and Epidemiology and Biostatistics, University of California - San Francisco, San Francisco Veterans Affairs Medical Center, 4150 Clement Street, Box 181, San Francisco, CA, 94121, USA. .
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8: Title: Decreased Expression of Innate Immunity-Related Genes in Peripheral Blood Mononuclear Cells from Patients with IgG4-Related Disease.
Authors: Nakajima, Akio, et.al. .
Journal: PloS one, Vol. 10 (5): e0126582, 2015 .
Snippet: CONCLUSIONS: The expression of genes related to allergy or innate immunity, including CLC, MS4A3, DEFA3, DEFA4, IL8RA and IL8RB, was lower in PBMCs from patients with IgG4-RD than from healthy controls.
Affiliation: Hematology and Immunology, Kanazawa Medical University, Uchinada, Ishikawa 920-0293, Japan. Medical Research Institute, Kanazawa Medical University, Uchinada, Ishikawa 920-0293, Japan. Division of Rheumatology, Department of Internal Medicine, Kanazawa University Hospital, Ishikawa 920-8641, Japan. Department of Medicine and Clinical Science, Gunma University Graduate School of Medicine, Gunma 371-8511, Japan. Health Administration Center University of Toyama, Toyama 930-0194, Japan. Department of Internal Medicine, Nagaoka Red Cross Hospital, Niigata 940-2085, Japan. Third Department of Internal Medicine, Division of Gastroenterology and Hepatology, Kansai Medical University, Osaka 573-1191, Japan. Department of Internal Medicine, Tokyo Metropolitan Komagome Hospital, Tokyo 113-8677, Japan. Department of Rheumatology, National Hospital Organization Nagasaki Medical center, Nagasaki 380-8582, Japan. Department of Clinical Oncology, Ehime Graduate School of Medicine, Ehime 791-0295, Japan. Department of Rheumatology, Japan Community Healthcare Organization, Isahaya General Hospital, Nagasaki 854-8501, Japan. Department of Gastroenterology, Rheumatology and Clinical Immunology, Sapporo Medical University School of Medicine, Hokkaido 060-8543, Japan. Medical Informatics Division and Department of Internal Medicine, Gastroenterology, Shinshu University School Hospital, Nagano 390-8621, Japan. First Department of Internal Medicine, Department of Immunology and Rheumatology, Nagasaki Graduate School of Health Sciences, Nagasaki 852-8520, Japan. First Department of Internal Medicine, School of Medicine, University of Occupational and Environmental Health, Japan, Fukuoka 807-8555, Japan. Department of Internal Medicine, Faculty of Medicine, University of Tsukuba, Ibaraki 305-8575, Japan. Division of Nephrology, Mitoyo General Hospital, Kagawa 769-1695, Japan. Hematology and Immunology, Kanazawa Medical University, Uchinada, Ishikawa 920-0293, Japan; Department of Clinical Immunology, Graduate School of Medicine and Faculty of Medicine, Kyoto University, Kyoto 606-8501, Japan. .
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9: Title: EVI1 promotes tumor growth via transcriptional repression of MS4A3.
Authors: Heller, Gerwin, et.al. .
Journal: Journal of hematology & oncology (J Hematol Oncol), Vol. 8, 2015 .
Snippet: Reporter gene assays and chromatin immunoprecipitation showed that EVI1 regulated MS4A3 via direct binding to a promoter proximal region.
Affiliation: Department of Medicine I, Medical University of Vienna, Währinger Gürtel 18-20, 1090, Vienna, Austria. gerwin.heller@meduniwien.ac.at. Comprehensive Cancer Center of the Medical University of Vienna, Vienna, Austria. gerwin.heller@meduniwien.ac.at. Department of Medicine I, Medical University of Vienna, Währinger Gürtel 18-20, 1090, Vienna, Austria. apm.rommer@gmail.com. Comprehensive Cancer Center of the Medical University of Vienna, Vienna, Austria. apm.rommer@gmail.com. Department of Medicine I, Medical University of Vienna, Währinger Gürtel 18-20, 1090, Vienna, Austria. katarina.steinleitner@meduniwien.ac.at. Comprehensive Cancer Center of the Medical University of Vienna, Vienna, Austria. katarina.steinleitner@meduniwien.ac.at. Department of Medicine I, Medical University of Vienna, Währinger Gürtel 18-20, 1090, Vienna, Austria. julia.etzler@meduniwien.ac.at. Comprehensive Cancer Center of the Medical University of Vienna, Vienna, Austria. julia.etzler@meduniwien.ac.at. Biocenter, Division of Bioinformatics, Medical University of Innsbruck, Innrain 80, 6020, Innsbruck, Austria. hubert.hackl@i-med.ac.at. Comprehensive Cancer Center of the Medical University of Vienna, Vienna, Austria. petra.heffeter@meduniwien.ac.at. Department of Medicine I, Institute of Cancer Research, and Research Platform "Translational Cancer Therapy Research", Borschkegasse 8A, 1090, Vienna, Austria. petra.heffeter@meduniwien.ac.at. Department of Medicine I, Medical University of Vienna, Währinger Gürtel 18-20, 1090, Vienna, Austria. erwin.tomasich@meduniwien.ac.at. Comprehensive Cancer Center of the Medical University of Vienna, Vienna, Austria. erwin.tomasich@meduniwien.ac.at. Comprehensive Cancer Center of the Medical University of Vienna, Vienna, Austria. martin.filipits@meduniwien.ac.at. Department of Medicine I, Institute of Cancer Research, and Research Platform "Translational Cancer Therapy Research", Borschkegasse 8A, 1090, Vienna, Austria. martin.filipits@meduniwien.ac.at. Department of Medicine I, Medical University of Vienna, Währinger Gürtel 18-20, 1090, Vienna, Austria. birgit.steinmetz@meduniwien.ac.at. Comprehensive Cancer Center of the Medical University of Vienna, Vienna, Austria. birgit.steinmetz@meduniwien.ac.at. Department of Medicine I, Medical University of Vienna, Währinger Gürtel 18-20, 1090, Vienna, Austria. thais.topakian@meduniwien.ac.at. Comprehensive Cancer Center of the Medical University of Vienna, Vienna, Austria. thais.topakian@meduniwien.ac.at. Department of Medicine I, Medical University of Vienna, Währinger Gürtel 18-20, 1090, Vienna, Austria. sim.kling@gmx.at. Comprehensive Cancer Center of the Medical University of Vienna, Vienna, Austria. sim.kling@gmx.at. Department of Medicine I, Medical University of Vienna, Währinger Gürtel 18-20, 1090, Vienna, Austria. barbara.ziegler@meduniwien.ac.at. Comprehensive Cancer Center of the Medical University of Vienna, Vienna, Austria. barbara.ziegler@meduniwien.ac.at. Core Facility Flow Cytometry & Surgical Research Laboratories, Medical University of Vienna, Währinger Gürtel 18-20, 1090, Vienna, Austria. andreas.spittler@meduniwien.ac.at. Department of Medicine I, Medical University of Vienna, Währinger Gürtel 18-20, 1090, Vienna, Austria. sabine.zoechbauer-mueller@meduniwien.ac.at. Comprehensive Cancer Center of the Medical University of Vienna, Vienna, Austria. sabine.zoechbauer-mueller@meduniwien.ac.at. Comprehensive Cancer Center of the Medical University of Vienna, Vienna, Austria. walter.berger@meduniwien.ac.at. Department of Medicine I, Institute of Cancer Research, and Research Platform "Translational Cancer Therapy Research", Borschkegasse 8A, 1090, Vienna, Austria. walter.berger@meduniwien.ac.at. Department of Medicine I, Medical University of Vienna, Währinger Gürtel 18-20, 1090, Vienna, Austria. rotraud.wieser@meduniwien.ac.at. Comprehensive Cancer Center of the Medical University of Vienna, Vienna, Austria. rotraud.wieser@meduniwien.ac.at. .
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10: Title: The MS4A family: counting past 1, 2 and 3.
Authors: Eon Kuek, Li, et.al. .
Journal: Immunology and cell biology (Immunol Cell Biol), Vol. 94 (1): 11-23, 2016 .
Snippet: The MS4A (membrane-spanning 4-domain family, subfamily A) family of proteins contains some well-known members including MS4A1 (CD20), MS4A2 (FcɛRIβ) and MS4A3 (HTm4).
Affiliation: Department of Pharmacology and Therapeutics, and Lung Health Research Centre, The University of Melbourne, Parkville, Victoria, Australia. John Curtin School of Medical Research, Australian National University, Canberra, Australian Capital Territory, Australia. Department of Biochemistry, La Trobe Institute for Molecular Science, La Trobe University, Bundoora, Victoria, Australia. .
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11: Title: Deficiency of GRP94 in the hematopoietic system alters proliferation regulators in hematopoietic stem cells.
Authors: Luo, Biquan, et.al. .
Journal: Stem cells and development (Stem Cells Dev), Vol. 22 (23): 3062-73, 2013 .
Snippet: A further examination comparing freshly isolated BM LSK cells with spleen LSK cells, as well as BM LSK cells cultured in vitro, revealed specific down-regulation of Ms4a3 in freshly isolated BM GRP94-null LSK cells.
Affiliation: 1 Department of Biochemistry and Molecular Biology, USC Norris Comprehensive Cancer Center, Keck School of Medicine, University of Southern California , Los Angeles, California. .
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12: Title: Whole-exome sequencing to identify novel somatic mutations in squamous cell lung cancers.
Authors: Zheng, Cui-Xia, et.al. .
Journal: International journal of oncology (Int J Oncol), Vol. 43 (3): 755-64, 2013 .
Snippet: Taken together, our results implicate TP53, EP300, LPHN2, C10orf137, MYH2, TGM2 and MS4A3 as potential driver genes of squamous cell lung cancer.
Affiliation: Department of Respiration, Ruijin Hospital Affiliated to Shanghai Jiaotong University School of Medicine, Shanghai 200025, P.R. China. .
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13: Title: Targeted ablation of miR-21 decreases murine eosinophil progenitor cell growth.
Authors: Lu, Thomas X, et.al. .
Journal: PloS one, Vol. 8 (3): e59397, 2013 .
Snippet: Using gene expression microarray analysis, we identified dysregulation of genes involved in cell proliferation (e,g, Ms4a3, Grb7), cell cycle and immune response as the most significant pathways affected by miR-21 in eosinophil progenitors.
Affiliation: Division of Allergy and Immunology, Department of Pediatrics, Cincinnati Children's Hospital Medical Center, University of Cincinnati College of Medicine, Cincinnati, Ohio, United States of America. .
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14: Title: Genome-wide association study identifies novel loci associated with serum level of vitamin B12 in Chinese men.
Authors: Lin, Xiaoling, et.al. .
Journal: Human molecular genetics (Hum Mol Genet), Vol. 21 (11): 2610-7, 2012 .
Snippet: These four loci were MS4A3 (11q12.1;
Affiliation: Fudan Institute of Urology, Huashan Hospital, School of Life Sciences, Fudan University, Shanghai, China. .
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15: Title: miR-451 enhances erythroid differentiation in K562 cells.
Journal: Leukemia & lymphoma (Leuk Lymphoma), Vol. 51 (4): 686-93, 2010 .
Snippet: Microarray data suggested potential targets regulated by miR-451: UBE2H, ARPP-19; and by miR-150: MS4A3, AGA, PTPRR.
Affiliation: Institute of Hematology and Blood Transfusion, Prague, Czech Republic. .
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16: Title: Phylogenetic analysis of the MS4A and TMEM176 gene families.
Authors: Zuccolo, Jonathan, et.al. .
Journal: PloS one, Vol. 5 (2): e9369, 2010 .
Snippet: BACKGROUND: The MS4A gene family in humans includes CD20 (MS4A1), FcRbeta (MS4A2), Htm4 (MS4A3), and at least 13 other syntenic genes encoding membrane proteins, most having characteristic tetraspanning topology.
Affiliation: Department of Biochemistry and Molecular Biology, University of Calgary, Calgary, Alberta, Canada. .
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17: Title: Characterization of the expression of HTm4 (MS4A3), a cell cycle regulator, in human peripheral blood cells and normal and malignant tissues.
Authors: Kutok, Jeffery L, et.al. .
Journal: Journal of cellular and molecular medicine (J Cell Mol Med), Vol. 15 (1): 86-93, 2011 .
Snippet: HTm4 (MS4A3) is a member of a family of four-transmembrane proteins designated MS4A.
Affiliation: Harvard Medical School, Department of Pathology, Brigham and Women's Hospital, Boston, MA 02115, USA. .
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18: Title: The CXCR4 antagonist AMD3100 releases a subset of G-CSF-primed peripheral blood progenitor cells with specific gene expression characteristics.
Authors: Fruehauf, Stefan, et.al. .
Journal: Experimental hematology (Exp Hematol), Vol. 34 (8): 1052-9, 2006 .
Snippet: Only increased expression was found in the categories antiapoptosis (e.g., MPO, HSPA1B), cell cycle (e.g., MS4A3, RRM2), replication/DNA repair (e.g., MPO, HSPA1B), cell motility (e.g., TNFSF4, HMMR), and oxygen transport.
Affiliation: Department of Internal Medicine V, University of Heidelberg, Heidelberg, Germany. Stefan.Fruehauf@med.uni-heidelberg.de .
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19: Title: siRNA-mediated AML1/MTG8 depletion affects differentiation and proliferation-associated gene expression in t(8;21)-positive cell lines and primary AML blasts.
Authors: Dunne, J, et.al. .
Journal: Oncogene, Vol. 25 (45): 6067-78, 2006 .
Snippet: Suppression of AML1/MTG8 results in the increased expression of genes associated with myeloid differentiation, such as AZU1, BPI, CTSG, LYZ and RNASE2 as well as of antiproliferative genes such as IGFBP7, MS4A3 and SLA both in blasts and in cell lines.
Affiliation: Cancer Research UK Medical Oncology Laboratory, Barts and the London School of Medicine, London, UK. .
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20: Title: Reversal of gene expression profile in the phenylketonuria mouse model after adeno-associated virus vector-mediated gene therapy.
Authors: Oh, Hyun-Jeong, et.al. .
Journal: Molecular genetics and metabolism (Mol Genet Metab), Vol. 86 Suppl 1, 2005 .
Snippet: Four are involved in defense and inflammatory responses of neutrophils (NE, MPO, NGP, and CRAMP), three other overexpressed genes are related to extracellular matrix organization and degradation (COL1A1, COL1A2, and MMP13); the remainder were a nociceptor in sensory neurons (MrgA1), a structural gene of P lysozyme (Lzp-s), an immunoglobulin alpha heavy chain constant region gene (Igh-2), an osteocalcin-related protein precursor (Bglap-rs1), and a membrane-spanning 4 domain, subfamily A, member 3 (Ms4a3).
Affiliation: Department of Biomedical Sciences, National Institute of Health, Seoul 122-701, Republic of Korea. .
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