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5 documents found
1: Title: Decreased expression of MS4A12 inhibits differentiation and predicts early stage survival in colon cancer.
Authors: He, L, et.al. .
Journal: Neoplasma, Vol. 64 (1): 65-73, 2017 .
Snippet: We used BS (4 mmol/L) inducing differentiation of LoVo cell and found after BS treated over 48h MS4A12 variant-1 (one of MS4A12 gene transcripts) as well as ALP and E-cadherin of LoVo cells were all increased significantly.
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2: Title: CAV3 mutation in a patient with transient hyperCKemia and myalgia.
Authors: Macias, Anna, et.al. .
Journal: Neurologia i neurochirurgia polska (Neurol Neurochir Pol), Vol. 50 (6): 468-473, 2016 Nov - Dec .
Snippet: Here, we describe a patient with Klinefelter syndrome, ulcerative colitis and Sjögren syndrome, who developed transient hyperCKemia, myalgia and mild muscular weakness.
Affiliation: Department of Neurology, Medical University of Warsaw, Warsaw, Poland. Department of Molecular and Human Genetics, Baylor College of Medicine, Houston, USA; Institute of Computer Science, Warsaw University of Technology, Warsaw, Poland. Department of Molecular and Human Genetics, Baylor College of Medicine, Houston, USA. 2nd Department of Clinical Radiology, Medical University of Warsaw, Warsaw, Poland. Department of Medical Genetics, Institute of Mother and Child, Warsaw, Poland. Department of Molecular and Human Genetics, Baylor College of Medicine, Houston, USA; Department of Pediatrics; Human Genome Sequencing Center, Baylor College of Medicine, Houston, USA; Texas Children's Hospital, Houston, USA. Department of Neurology, Medical University of Warsaw, Warsaw, Poland. Electronic address: amkaminska@wum.edu.pl. .
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3: Title: Selective activation of tumor growth-promoting Ca2+ channel MS4A12 in colon cancer by caudal type homeobox transcription factor CDX2.
Authors: Koslowski, Michael, et.al. .
Journal: Molecular cancer (Mol Cancer), Vol. 8, 2009 .
Snippet: Moreover, this is the first report establishing CDX2 as transactivator of tumor growth-promoting gene expression in colon cancer, adding to untangle the complex and conflicting biological functions of CDX2 in colon cancer and supporting MS4A12 as important factor for normal colonic development as well as for the biology and treatment of colon cancer.
Affiliation: Department of Internal Medicine III, Experimental and Translational Oncology, Johannes Gutenberg University, Obere Zahlbacherstr, 63, 55131 Mainz, Germany. koslowsk@uni-mainz.de .
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4: Title: MS4A12 is a colon-selective store-operated calcium channel promoting malignant cell processes.
Authors: Koslowski, Michael, et.al. .
Journal: Cancer research (Cancer Res), Vol. 68 (9): 3458-66, 2008 .
Snippet: Expression analysis and immunohistochemistry revealed MS4A12 to be a colonic epithelial cell lineage gene confined to the apical membrane of colonocytes with strict transcriptional repression in all other normal tissue types.
Affiliation: Department of Internal Medicine III, Division of Experimental and Translational Oncology, Johannes Gutenberg University, Mainz, Germany. .
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5: Title: Structural organization of the human MS4A gene cluster on Chromosome 11q12.
Authors: Liang, Y, et.al. .
Journal: Immunogenetics, Vol. 53 (5): 357-68, 2001 .
Snippet: In contrast to other MS4A genes, MS4A4E, MS4A6E, and MS4A10 transcripts were rare and not detected among hematopoietic cells and most nonlymphoid tissues.
Affiliation: Department of Immunology, Room 353 Jones Building, Research Drive, Duke University Medical Center, Durham, NC 27710, USA. .
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