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8 documents found
1: Title: Expression of micro-RNAs and genes related to angiogenesis in ccRCC and associations with tumor characteristics.
Journal: BMC urology (Bmc Urol), Vol. 17 (1): 113, 2017 .
Snippet: RESULTS: miR99a was overexpressed in most samples and its target gene mTOR was underexpressed, this also occurs for miRNAs 106a, 106b, and their target gene VHL.
Affiliation: Laboratory of Medical Investigation (LIM55), Urology Department, University of Sao Paulo Medical School, Av. Dr. Arnaldo 455, 2° floor, room 2145, Sao Paulo, 01246-903, Brazil. Uro-Oncology Group, Urology Department, University of Sao Paulo Medical School and Institute of Cancer Estate of Sao Paulo (ICESP), Sao Paulo, Brazil. Laboratory of Medical Investigation (LIM55), Urology Department, University of Sao Paulo Medical School, Av. Dr. Arnaldo 455, 2° floor, room 2145, Sao Paulo, 01246-903, Brazil. sasareis@gmail.com. .
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2: Title: Integrated analysis of the potential roles of miRNA‑mRNA networks in triple negative breast cancer.
Authors: Zhu, Huiru, et.al. .
Journal: Molecular medicine reports (Mol Med Report), Vol. 16 (2): 1139-1146, 2017 .
Snippet: To date, expression profiling of microRNA (miRNA/miR) and mRNA sequences have been widely applied for the diagnosis of TNBC.
Affiliation: Department of Galactophore, The Third Affiliated Hospital of Guangxi University of Chinese Medicine, Liuzhou, Guangxi 545001, P.R. China. Department of Clinical Laboratory, The Fourth Affiliated Hospital of Guangxi Medical University, Liuzhou, Guangxi 545005, P.R. China. .
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3: Title: Targeted sequencing of 96 renal developmental microRNAs in 1213 individuals from 980 families with congenital anomalies of the kidney and urinary tract.
Authors: Kohl, Stefan, et.al. .
Journal: Nephrology, dialysis, transplantation : official publication of the European Dialysis and Transplant Association - European Renal Association (Nephrol Dial Transplant), Vol. 31 (8): 1280-3, 2016 .
Snippet: Two out of 1213 unrelated individuals had potentially pathogenic variants with unknown biologic relevance affecting miRNAs MIR19B1 and MIR99A.
Affiliation: Department of Medicine, Boston Children's Hospital, Harvard Medical School, Boston, MA, USA Department of Pediatrics, Cologne Children's Hospital, Cologne, Germany. Department of Medicine, Boston Children's Hospital, Harvard Medical School, Boston, MA, USA. Department of Medicine, Boston Children's Hospital, Harvard Medical School, Boston, MA, USA Talpiot Medical Leadership Program, Sheba Medical Center, Tel-Hashomer, Israel. Department of Medicine, Boston Children's Hospital, Harvard Medical School, Boston, MA, USA Division of Nephrology, Department of Medicine, Kaohsiung Medical University Hospital, Kaohsiung Medical University, Kaohsiung, Taiwan. Department of Medicine, Boston Children's Hospital, Harvard Medical School, Boston, MA, USA Institute of Human Genetics, University of Bonn, Bonn, Germany. Department of Medicine, Columbia University, New York, NY, USA. Department of Urology, Boston Children's Hospital, Harvard Medical School, Boston, MA, USA. Department of Pediatrics, Kasr Al Ainy School of Medicine, Cairo University, Cairo, Egypt Egyptian Group for Orphan Renal Diseases (EGORD), Cairo, Egypt. Division of Urology, Department of Surgery, Kuwait University, Safat, Kuwait. Institute of Human Genetics, University of Bonn, Bonn, Germany Department of Neonatology, Children's Hospital, University of Bonn, Bonn, Germany. Medical Faculty Skopje, University Children's Hospital, Skopje, Macedonia. Department of Medicine, Boston Children's Hospital, Harvard Medical School, Boston, MA, USA Howard Hughes Medical Institute, Chevy Chase, MD, USA. .
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4: Title: Long noncoding RNA ANRIL promotes non-small cell lung cancer cell proliferation and inhibits apoptosis by silencing KLF2 and P21 expression.
Authors: Nie, Feng-qi, et.al. .
Journal: Molecular cancer therapeutics (Mol Cancer Ther), Vol. 14 (1): 268-77, 2015 .
Snippet: Our previous study showed that ANRIL was significantly upregulated in gastric cancer, and it could promote cell proliferation and inhibit cell apoptosis by silencing of miR99a and miR449a transcription.
Affiliation: Department of Oncology, First Affiliated Hospital, Nanjing Medical University, Nanjing, People's Republic of China. Department of Biochemistry and Molecular Biology, Nanjing Medical University, Nanjing, People's Republic of China. Department of Oncology, Nanjing First Hospital, Nanjing Medical University, People's Republic of China. Department of Oncology, First Affiliated Hospital, Nanjing Medical University, Nanjing, People's Republic of China. tgzy111@126.com yongqian_shu@163.com. .
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5: Title: [Genomics analysis of miRNA in colon cancer tissues].
Authors: Xu, Xuehu, et.al. .
Journal: Zhonghua wei chang wai ke za zhi = Chinese journal of gastrointestinal surgery (Zhonghua Wei Chang Wai Ke Za Zhi), Vol. 17 (11): 1130-2, 2014 .
Snippet: Cluster analysis showed that miR763-3, miR451 and miR99a had similar expression.
Affiliation: Department of Gastrointestinal Surgery, The Third Affiliated Hospital, Guangzhou Medical University, Guangzhou 510000, China. xxh@gzhmu.edu.cn. .
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6: Title: Loss of estrogen-regulated microRNA expression increases HER2 signaling and is prognostic of poor outcome in luminal breast cancer.
Authors: Bailey, Shannon T, et.al. .
Journal: Cancer research (Cancer Res), Vol. 75 (2): 436-45, 2015 .
Snippet: We found 78 miRNAs differentially expressed between the two cell lines, including a cluster comprising let-7c, miR99a, and miR125b, which is encoded in an intron of the long noncoding RNA LINC00478.
Affiliation: Center for Functional Cancer Epigenetics, Dana-Farber Cancer Institute, Boston, Massachusetts. Department of Medical Oncology, Dana-Farber Cancer Institute, Boston, Massachusetts. Department of Medicine, Brigham and Women's Hospital and Harvard Medical School, Boston, Massachusetts. Center for Functional Cancer Epigenetics, Dana-Farber Cancer Institute, Boston, Massachusetts. Department of Medical Oncology, Dana-Farber Cancer Institute, Boston, Massachusetts. Department of Medicine, Brigham and Women's Hospital and Harvard Medical School, Boston, Massachusetts. myles_brown@dfci.harvard.edu. .
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7: Title: microRNA‑99a inhibits cell proliferation, colony formation ability, migration and invasion by targeting fibroblast growth factor receptor 3 in prostate cancer.
Authors: Wu, Deyao, et.al. .
Journal: Molecular medicine reports (Mol Med Report), Vol. 11 (2): 1469-75, 2015 .
Snippet: microRNA‑99a (miR‑99a) was reported to be among the most frequently downregulated miRNAs in numerous types of human cancers, including prostate, bladder, hepatocellular and ovarian carcinoma, squamous cell carcinoma of the tongue, squamous cell lung carcinoma as well as childhood adrenocortical tumors.
Affiliation: Department of Urology, The Fourth Affiliated Hospital of Nantong Medical College, Yancheng City First People's Hospital, Yancheng, Jiangsu 224001, P.R. China. .
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8: Title: MiR99a modulates MMP7 and MMP13 to regulate invasiveness of Kaposi's sarcoma.
Authors: Zhang, Jun, et.al. .
Journal: Tumour biology : the journal of the International Society for Oncodevelopmental Biology and Medicine (Tumour Biol), Vol. 35 (12): 12567-73, 2014 .
Snippet: Inhibition of PI3k/Akt signaling pathway significantly abolished the effect of miR99a-knockdown on MMP7, but not MMP13 activation, while inhibition of ERK/MAPK signaling pathway significantly abolished the effect of miR99a-knockdown on MMP13, but not MMP7 activation.
Affiliation: Department of Surgical Oncology, Children's Hospital of Chongqing Medical University, Chongqing, China. .
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