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8 documents found
1: Title: MAGE genes in the kidney: identification of MAGED2 as upregulated during kidney injury and in stressed tubular cells.
Authors: Valiño-Rivas, Lara, et.al. .
Journal: Nephrology, dialysis, transplantation : official publication of the European Dialysis and Transplant Association - European Renal Association (Nephrol Dial Transplant), 2018 .
Snippet: Results: Five MAGE genes (MAGED1, MAGED2, MAGED3, MAGEH1, MAGEE1) were expressed at the mRNA level in healthy adult mouse kidneys, as assessed by RNA-Seq.
Affiliation: Nephrology, IIS-Fundacion Jimenez Diaz-Universidad Autonoma de Madrid and Fundacion Renal Iñigo Alvarez de Toledo-IRSIN, Madrid, Spain. Nephrology, REDINREN, Madrid, Spain. Institute of Cardiovascular and Medical Sciences, University of Glasgow, Glasgow, UK. Department of Diabetes and Cardiovascular Science, University of the Highlands and Islands, Inverness, UK. Mosaiques diagnostics GmbH, Hannover, Germany. .
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2: Title: Downregulation of MAGE family member H1 enhances hepatocellular carcinoma progression and serves as a biomarker for patient prognosis.
Authors: Wang, Peng-Cheng, et.al. .
Journal: Future oncology (London, England) (Future Oncol), 2018 .
Snippet: RESULTS:  MAGEH1 expression was downregulated in HCC tumor tissues compared with adjacent normal liver tissues and in samples from patients with tumor recurrence.
Affiliation: Liver Cancer Institute, Zhongshan Hospital, Fudan University, Key Laboratory of Carcinogenesis & Cancer Invasion, Fudan University, Ministry of Education, Shanghai 200032, China. .
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3: Title: Molecular evolution of type II MAGE genes from ancestral MAGED2 gene and their phylogenetic resolution of basal mammalian clades.
Authors: De Donato, Marcos, et.al. .
Journal: Mammalian genome : official journal of the International Mammalian Genome Society (Mamm Genome), 2017 .
Snippet: Type II genes can be divided into two: those with 13 exons (MAGED1, MAGED2, TRO, and MAGED4) and those with only one exon (MAGEE1, MAGEE2, MAGEF1, NSMCE3, MAGEH1, MAGEL2, and NDN) with different evolutionary patterns.
Affiliation: Animal Genetics and Genomics Laboratory, International Programs, College of Agriculture and Life Science, Cornell University, Ithaca, NY, 14853, USA. Escuela de Escuela de Bioingenierias, Instituto Tecnologico y de Estudios Superiores de Monterrey, Queretaro, Mexico. Department of Animal Science, Berry College, Mount Berry, GA, 30149, USA. Department of Molecular Biology, Virtual University of Pakistan, Lahore, 54000, Pakistan. Department of Biomedical Sciences, Rochester Institute of Technology, Rochester, NY, 14623, USA. Animal Genetics and Genomics Laboratory, International Programs, College of Agriculture and Life Science, Cornell University, Ithaca, NY, 14853, USA. igi2@cornell.edu. School of Biological Sciences, Georgia Institute of Technology, Atlanta, GA, 30332, USA. igi2@cornell.edu. .
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4: Title: Transcriptional Landscape of Human Tissue Lymphocytes Unveils Uniqueness of Tumor-Infiltrating T Regulatory Cells.
Authors: De Simone, Marco, et.al. .
Journal: Immunity, Vol. 45 (5): 1135-1147, 2016 .
Snippet: Remarkably, high expression in whole-tumor samples of Treg cell signature genes, such as LAYN, MAGEH1, or CCR8, correlated with poor prognosis.
Affiliation: Istituto Nazionale Genetica Molecolare INGM 'Romeo ed Enrica Invernizzi,' Milan 20122, Italy. Istituto Nazionale Genetica Molecolare INGM 'Romeo ed Enrica Invernizzi,' Milan 20122, Italy; Division of Pathology, IRCCS Ca' Granda Ospedale Maggiore Policlinico, Milan 20122, Italy; Department of Pathophysiology and Organ Transplantation, Università degli Studi di Milano, Milano 20122, Italy. Division of Pathology, IRCCS Ca' Granda Ospedale Maggiore Policlinico, Milan 20122, Italy; Department of Pathophysiology and Organ Transplantation, Università degli Studi di Milano, Milano 20122, Italy. Division of Thoracic Surgery, IRCCS Ca' Granda Ospedale Maggiore Policlinico, Milan 20122, Italy. Division of Thoracic Surgery, IRCCS Ca' Granda Ospedale Maggiore Policlinico, Milan 20122, Italy; Department of Pathophysiology and Organ Transplantation, Università degli Studi di Milano, Milano 20122, Italy. Department of Pathology, San Gerardo Hospital, Monza 20900, Italy. Department of Surgery, San Gerardo Hospital, Monza 20900, Italy. Department of Surgery, San Gerardo Hospital, Monza 20900, Italy; School of Medicine and Surgery, Milano-Bicocca University, Monza 20900 Italy. School of Medicine and Surgery, Milano-Bicocca University, Monza 20900 Italy. UO Chirurgia Epatobiliopancreatica e Digestiva Ospedale San Paolo, Milan 20142, Italy. UO Chirurgia Epatobiliopancreatica e Digestiva Ospedale San Paolo, Milan 20142, Italy; Department of Health Sciences, Università degli Studi di Milano, Milano 20122, Italy. Department of Molecular Biology, Faculty of Science, Radboud University, Nijmegen, The Netherlands. Istituto Nazionale Genetica Molecolare INGM 'Romeo ed Enrica Invernizzi,' Milan 20122, Italy; Department of Clinical Sciences and Community Health, Università degli Studi di Milano, Milano 20122, Italy. Electronic address: abrignani@ingm.org. Istituto Nazionale Genetica Molecolare INGM 'Romeo ed Enrica Invernizzi,' Milan 20122, Italy; Department of Medical Biotechnology and Translational Medicine, Università degli Studi di Milano, Milano 20129, Italy. Electronic address: pagani@ingm.org. .
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5: Title: Tumor-specific mutations in low-frequency genes affect their functional properties.
Authors: Erdem-Eraslan, Lale, et.al. .
Journal: Journal of neuro-oncology (J Neurooncol), Vol. 122 (3): 461-70, 2015 .
Snippet: NTN4 and MAGEH1) showed that the mutation affects the subcellular localization of the protein (n = 2/12).
Affiliation: Department of Neurology, Be 430A, Erasmus Medical Center, PO Box 2040, 3000 CA, Rotterdam, The Netherlands. .
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6: Title: Neonatal detection of Turner syndrome by real-time PCR gene quantification of the ARSE and MAGEH1 genes.
Authors: Corrêa, S C, et.al. .
Journal: Genetics and molecular research : GMR (Genet Mol Res), Vol. 13 (4): 9068-76, 2014 .
Snippet: Two study genes (ARSE and MAGEH1) and 1 normalizing gene (HBB) were used to detect the second X chromosome.
Affiliation: Laboratório de Medicina Molecular, Departamento de Ciências Fisiológicas, Faculdade de Ciências Médicas da Santa Casa de São Paulo, São Paulo, SP, Brasil. Departamento de Obstetrícia e Ginecologia, Irmandade Santa Casa de Misericórdia de São Paulo, São Paulo, SP, Brasil. Unidade de Neonatologia, Departamento de Pediatria, Irmandade Santa Casa de Misericórdia de São Paulo, São Paulo, SP, Brasil. Laboratório de Medicina Molecular, Departamento de Ciências Fisiológicas, Faculdade de Ciências Médicas da Santa Casa de São Paulo, São Paulo, SP, Brasil carloslongui@msn.com. .
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7: Title: The irreversible pan-HER inhibitor PF00299804 alone or combined with gemcitabine has an antitumor effect in biliary tract cancer cell lines.
Authors: Nam, Hyun-Jin, et.al. .
Journal: Investigational new drugs (Invest New Drugs), Vol. 30 (6): 2148-60, 2012 .
Snippet: Moreover, PF00299804 exerted synergistic effects with gemcitabine in seven of the eight BTC cell lines, possibly through the regulation of the genes involved in the response to gemcitabine, such as TS (thymidylate synthase), RRM1 (ribonucleotide reductase), and MAGEH1, which is negatively correlated with gemcitabine sensitivity.
Affiliation: Cancer Research Institute, Seoul National University College of Medicine, Seoul, South Korea. .
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8: Title: Establishment of six new human biliary tract carcinoma cell lines and identification of MAGEH1 as a candidate biomarker for predicting the efficacy of gemcitabine treatment.
Authors: Ojima, Hidenori, et.al. .
Journal: Cancer science (Cancer Sci), Vol. 101 (4): 882-8, 2010 .
Snippet: Based on their gemcitabine sensitivity, 10 BTC cell lines (including six new and four publicly available ones) were clearly categorized into two groups, and MAGEH1 mRNA expression in the tumor cells showed a significant negative correlation with their sensitivity to gemcitabine.
Affiliation: Pathology Division, Clinical Trials and Practice Support Division, Center for Cancer Control and Information Services, National Cancer Center, Chuo-ku, Tokyo, Japan. .
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