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7 documents found
1: Title: When the good go bad: Mutant NPM1 in acute myeloid leukemia.
Authors: Kunchala, Preethi, et.al. .
Journal: Blood reviews (Blood Rev), 2017 .
Snippet: We describe genetic abnormalities, the clinical significance of exon-12 mutations in the NPM1 gene, and chromosomal translocations including the recently discovered NPM1-TYK2, and NPM1-HAUS1.
Affiliation: Division of Hematologic Malignancies and Cellular Therapeutics, Department of Internal Medicine, University of Kansas Medical Center, Kansas City, KS, USA; The University of Kansas Cancer Center, Kansas City, KS, USA. The University of Kansas Cancer Center, Kansas City, KS, USA; Department of Pathology and Laboratory Medicine, University of Kansas Medical Center, Kansas City, KS, USA; Department of Cancer Biology, University of Kansas Medical Center, Kansas City, KS, USA. Division of Hematologic Malignancies and Cellular Therapeutics, Department of Internal Medicine, University of Kansas Medical Center, Kansas City, KS, USA; The University of Kansas Cancer Center, Kansas City, KS, USA; Department of Cancer Biology, University of Kansas Medical Center, Kansas City, KS, USA. Electronic address: rbalusu@kumc.edu. .
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2: Title: A novel mechanism of NPM1 cytoplasmic localization in acute myeloid leukemia: the recurrent gene fusion NPM1-HAUS1.
Journal: Haematologica, Vol. 101 (7): e287-90, 2016 .
No Abstract available.
Affiliation: Departments of Hematology and Clinical Pathology, and Research Institute, Hospital Israelita Albert Einstein, Department of Hematology, University of Campinas (Hemocentro - Unicamp), São Paulo, Brazil paulo.campregher@einstein.br. Research Institute, Hospital Israelita Albert Einstein, São Paulo, Brazil. Assistant physician and Chief, Hematology Service, Hospital das Clínicas, Faculdade de Medicina da Universidade de São Paulo, Chief of Cytogenetics Laboratories, Hospital Israelita Albert Einstein, São Paulo, Brazil. Department of Hematology, Hospital Israelita Albert Einstein, São Paulo, Brazil. Department of Clinical Pathology, Hospital Israelita Albert Einstein, São Paulo, Brazil. Laboratory of Health Physics, Radiobiology & Cytogenetics, National Centre for Scientific Research "Demokritos", Athens, Greece. Hematology and Hemotherapy Center, School of Medicine, University of Campinas, São Paulo, Brazil. .
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3: Title: Tissue-specific Gene Expression in Rat Hearts and Aortas in a Model of Vascular Nitrate Tolerance.
Authors: Csont, Tamás, et.al. .
Journal: Journal of cardiovascular pharmacology (J Cardiovasc Pharmacol), Vol. 65 (5): 485-93, 2015 .
Snippet: Of 7742 genes analyzed by DNA microarray, we found that although the expression of 25 genes changed significantly in the heart (increased: Tas2r119, Map6, Cd59, Kcnh2, Kcnh3, Senp6, Mcpt1, Tshb, Haus1, Vipr1, Lrn3, Lifr; decreased: Ihh, Fgfr1, Cryge, Krt9, Agrn, C4bpb, Fcer1a, Csf3, Hsd17b11, Hsd11b2, Ctnnbl1, Prpg1, Hsf1), only 14 genes were altered in the aorta (increased: Tas2r119, Ihh, Rrad, Npm1, Snai1; decreased: Tubb2b, Usp15, Sema6c, Wfdc2, Rps21, Ramp2, Galr1, Atxn1, Lhx1) in vascular nitrate tolerance.
Affiliation: *Department of Biochemistry, University of Szeged, Szeged, Hungary; †Pharmahungary Group, Szeged, Hungary; ‡Institute of Genetics, Biological Research Center, Hungarian Academy of Sciences, Szeged, Hungary; and §Department of Pharmacology and Pharmacotherapy, Semmelweis University, Budapest, Hungary. .
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4: Title: A novel insertion ins(18;5)(q21.1;q31.2q35.1) in acute myeloid leukemia associated with microdeletions at 5q31.2, 5q35.1q35.2 and 18q12.3q21.1 detected by oligobased array comparative genomic hybridization.
Journal: Molecular cytogenetics (Mol Cytogenet), Vol. 7 (1): 63, 2014 .
Snippet: A novel juxtaposition of the genes NPM1 and HAUS1 at 5q35.1 and 18q21.1,
Affiliation: Department of Hematology, HemoDiagnostic Laboratory, Cancer Cytogenetics Section, Aarhus University Hospital, Tage-Hansens Gade 2, Ent. 4A, DK-8000 Aarhus C, Denmark. .
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5: Title: Identification and characterization of genes related to the development of breast muscles in Pekin duck.
Authors: Xu, Tieshan, et.al. .
Journal: Molecular biology reports (Mol Biol Rep), Vol. 39 (7): 7647-55, 2012 .
Snippet: The results showed that unknown clone A233, C83 and C99 showed descending tendency as age increased; KBTBD10, HSPA8, MYL1, ZFP622, MARCH4, Nexilin, FABP4 and MUSTN1 had high expression levels at 6 weeks old; WAC, NT5C3, HSP90AA1, MRPL33, KLF6, TSNAX, CDC42EP3, HSPA4, TRAK1, NR2F2, HAUS1 and IGF1 had high expression levels at 8 weeks and showed ascending tendency as age increased.
Affiliation: State Key Laboratory of Animal Nutrition Science, Institute of Animal Science, CAAS, No 2, YuanMingYuan West Road, HaiDian, 100193 Beijing, People's Republic of China. .
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6: Title: Microarray analysis of gene expression profile of multidrug resistance in pancreatic cancer.
Authors: Zhao, Yu-pei, et.al. .
Journal: Chinese medical journal (Chinese Med J-peking), Vol. 120 (20): 1743-52, 2007 .
Snippet: According to Gene Ontology annotation, differentially expressed genes related to MDR in pancreatic cancer belong to many functional families and with diverse biological processes.
Affiliation: Department of General Surgery, Chinese Academy of Medical Sciences and Peking Union Medical College Hospital, Beijing 100730, China. zhao8028@yahoo.com.cn .
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7: Title: Human enhancer of invasion-cluster, a coiled-coil protein required for passage through mitosis.
Authors: Einarson, Margret B, et.al. .
Journal: Molecular and cellular biology (Mol Cell Biol), Vol. 24 (9): 3957-71, 2004 .
Snippet: These mitotic defects occur in spite of the fact that HEI-C-depleted cells retain functional cell cycle checkpoints, as these cells arrest normally following nocodazole or hydroxyurea treatment.
Affiliation: Division of Basic Science, Fox Chase Cancer Center, Philadelphia, Pennsylvania 19111, USA. .
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