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8 documents found
1: Title: Protein Acyltransferase DHHC3 Regulates Breast Tumor Growth, Oxidative Stress, and Senescence.
Authors: Sharma, Chandan, et.al. .
Journal: Cancer research (Cancer Res), Vol. 77 (24): 6880-6890, 2017 .
Snippet: Diminished DHHC3-dependent palmitoylation of ERGIC3 protein likely played a key role in TXNIP upregulation.
Affiliation: Department of Cancer Immunology and Virology, Dana-Farber Cancer Institute and Department of Pathology, Harvard Medical School, Boston, Massachusetts. Department of Cancer Immunology and Virology, Dana-Farber Cancer Institute and Department of Pathology, Harvard Medical School, Boston, Massachusetts. martin_hemler@dfci.harvard.edu. .
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2: Title: Endoplasmic reticulum-Golgi intermediate compartment protein 3 knockdown suppresses lung cancer through endoplasmic reticulum stress-induced autophagy.
Authors: Hong, Seong-Ho, et.al. .
Journal: Oncotarget, Vol. 7 (40): 65335-65347, 2016 .
Snippet: Moreover, non-invasive aerosol-delivery of shERGIC3 using the biocompatible carrier glycerol propoxylate triacrylate and spermine (GPT-SPE) inhibited lung tumorigenesis in the K-rasLA1 murine model of lung cancer.
Affiliation: Laboratory of Toxicology, BK21 PLUS Program for Creative Veterinary Science Research, Research Institute for Veterinary Science and College of Veterinary Medicine, Seoul National University, Seoul 08826, Korea. New Drug Development Center, Daegu-Gyeongbuk Medical Innovation Foundation, Daegu 41061, Korea. Department of Applied Chemistry, College of Applied Science, Kyung Hee University, Yongin 17104, Korea. Graduate Group of Tumor Biology, Seoul National University, Seoul 08826, Korea. Department of Pharmaceutics, State Key Laboratory of Natural Medicines, China Pharmaceutical University, Nanjing 210009, PR China. Laboratory of Pathology, College of Veterinary Medicine, Seoul National University, Seoul 08826, Korea. Department of Pharmacology and Medical Science, Infection Signaling Network Research Center, College of Medicine, Chungnam National University, Daejeon 35015, Korea. Graduate School of Convergence Science and Technology, Seoul National University, Suwon 16229, Korea. Advanced Institute of Convergence Technology, Seoul National University, Suwon 16229, Korea. Institute of GreenBio Science Technology, Seoul National University, Pyeongchang-gun 25354, Korea. .
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3: Title: Evolutionary diversification of the BetaM interactome acquired through co-option of the ATP1B4 gene in placental mammals.
Authors: Korneenko, Tatyana V, et.al. .
Journal: Scientific reports (Sci Rep), Vol. 6, 2016 .
Snippet: The inventory of identified BetaM interactors includes lamina-associated protein LAP-1, myocyte nuclear envelope protein Syne1, BetaM itself, heme oxidases HMOX1 and HMOX2; transcription factor LZIP/CREB3, ERGIC3, PHF3, reticulocalbin-3, and β-sarcoglycan.
Affiliation: Shemyakin and Ovchinnikov Institute of Bioorganic Chemistry, Moscow 117871, Russia. Department of Physiology and Pharmacology and Center for Diabetes and Endocrine Research, University of Toledo College of Medicine, 3000 Arlington Ave, Toledo, OH 43614, USA. .
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4: Title: ERGIC3, which is regulated by miR-203a, is a potential biomarker for non-small cell lung cancer.
Authors: Lin, Qing-Hai, et.al. .
Journal: Cancer science (Cancer Sci), Vol. 106 (10): 1463-73, 2015 .
Snippet: We also studied the mechanisms of ERGIC3 regulation in vitro and in vivo by means of bioinformatics analysis, luciferase reporter assay, miRNA expression profiling and miRNA transfection.
Affiliation: Laboratory of Molecular and Experimental Pathology, Kunming Institute of Zoology, Chinese Academy of Sciences, Kunming, China. Department of Pathology, Yunnan Tumor Hospital, Kunming, China. .
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5: Title: An integrative CGH, MSI and candidate genes methylation analysis of colorectal tumors.
Authors: Brim, Hassan, et.al. .
Journal: PloS one, Vol. 9 (1): e82185, 2014 .
Snippet: The most amplified markers were CD248, ERCC6, ERGIC3, GNAS, MMP2, NF1, P2RX7, SFRS6, SLC29A1 and TBX22.
Affiliation: Department of Pathology, Medicine and Cancer Center, Howard University, College of Medicine, Washington, DC, United States of America. Histopathology Core, National Eye Institute, National Institutes of Health, Bethesda, Maryland, United States of America. Section of Biomedical Computing, Clinical Center, National Institutes of Health, Bethesda, Maryland, United States of America. Drexel University College of Medicine, Monmouth Medical Center, Department of Medicine, Long Branch, New Jersey, United States of America. Biochemistry Department, Shiraz University of Medical Sciences, Shiraz, Iran. National Center for Biotechnology Information, National Library of Medicine, National Institutes of Health, Bethesda, Maryland, United States of America. .
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6: Title: Suppression subtractive hybridization identified differentially expressed genes in lung adenocarcinoma: ERGIC3 as a novel lung cancer-related gene.
Authors: Wu, Mingsong, et.al. .
Journal: BMC cancer, Vol. 13, 2013 .
Snippet: In the first stage of the screening for 16 genes, we compared lung cancer tissues with their adjacent non-malignant tissues at the mRNA level, and found six genes (ERGIC3, DDR1, HSP90B1, SDC1, RPSA, and LPCAT1) from the FSL were significantly up-regulated while two genes (GPX3 and TIMP3) from the RSL were significantly down-regulated (P < 0.05).
Affiliation: Key Laboratory of Animal Models and Human Disease Mechanism, Kunming Institute of Zoology, Chinese Academy of Sciences, Kunming 650223, China. .
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7: Title: miR-490-3p modulates cell growth and epithelial to mesenchymal transition of hepatocellular carcinoma cells by targeting endoplasmic reticulum-Golgi intermediate compartment protein 3 (ERGIC3).
Authors: Zhang, Ling-yun, et.al. .
Journal: The Journal of biological chemistry (J Biol Chem), Vol. 288 (6): 4035-47, 2013 .
Snippet: Unlike most miRNA-mRNA interactions, miR-490-3p increased ERGIC3 mRNA and protein levels as well as the intensity of expression of the EGFP reporter gene controlled by the 3'-UTR of ERGIC3 mRNA.
Affiliation: Tianjin Life Science Research Center and Basic Medical School, Tianjin Medical University, No. 22 Qi-Xiang-Tai Road, Tianjin 300070, China. .
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8: Title: Identification and characterization of endoplasmic reticulum-associated protein, ERp43.
Authors: Nishikawa, Manabu, et.al. .
Journal: Gene, Vol. 386 (1-2): 42-51, 2007 .
Snippet: Quantitative reverse transcriptase-polymerase chain reaction demonstrated that gene expression was relatively high in the neuronal tissues and in the kidney, with ERp43 protein highly expressed in the spinal cord and in the kidney.
Affiliation: Department of Biochemistry and Molecular Pathology, Osaka City University Medical School, Osaka 545-8585, Japan. nishikawa@med.osaka-cu.ac.jp .
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