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22 documents found
1: Title: Differential Proteomic Analysis between Small Cell Lung Carcinoma (SCLC) and Pulmonary Carcinoid Tumors Reveals Molecular Signatures for Malignancy in Lung Cancer.
Authors: Fujii, Kiyonaga, et.al. .
Journal: Proteomics. Clinical applications (Proteomics Clin Appl), 2018 .
Snippet: Particularly, 11 proteins involved in tumor proliferation (MCM2, 4, 6, 7, and MSH2), metastasis (RCC2, CORO1C, CHD4, and IPO9), and cancer metabolism (PHGDH and TYMP) were identified as SCLC-specific proteins.
Affiliation: Department of Translational Medicine Informatics, St. Marianna University School of Medicine, Kawasaki, Japan. Nissha Co., Ltd., Kyoto, Japan. Department of Pathology, St. Marianna University School of Medicine, Kawasaki, Japan. Department of Chest Surgery, St. Marianna University School of Medicine, Kawasaki, Japan. .
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2: Title: [Genetic diversity of the Pacific walrus (Odobenus rosmarus divergens) in the western part of the Chukchi Sea].
Authors: Shitova, M V, et.al. .
Journal: Genetika, Vol. 53 (2): 223-32, 2017 .
Snippet: Two microsatellite loci which are described as microsatellites for the first time are used in this study: repeated sequences within introns of Coro1c and Plod2 genes.
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3: Title: Transcriptomic signatures of cellular and humoral immune responses in older adults after seasonal influenza vaccination identified by data-driven clustering.
Authors: Voigt, Emily A, et.al. .
Journal: Scientific reports (Sci Rep), Vol. 8 (1): 739, 2018 .
Snippet: CORO1C, C8orf83) likely related to influenza vaccine-induced immunity due to their expression patterns.
Affiliation: Mayo Clinic Vaccine Research Group, Mayo Clinic, Rochester, MN 55905, USA. Division of Biomedical Statistics and Informatics Mayo Clinic, Rochester, MN 55905, USA. Mayo Clinic Vaccine Research Group, Mayo Clinic, Rochester, MN 55905, USA. poland.gregory@mayo.edu. .
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4: Title: Identification of miRNA and genes involving in osteosarcoma by comprehensive analysis of microRNA and copy number variation data.
Authors: Luo, Tao, et.al. .
Journal: Oncology letters (Oncol Lett), Vol. 14 (5): 5427-5433, 2017 .
Snippet: miRNAs (hsa-miR-27a-3p, hsa-miR-124-3p, hsa-miR-9-5p, hsa-miR-182-5p, hsa-miR-26a-5p) and the genes [Fibroblast growth factor receptor substrate 2 (FRS2), coronin 1C (CORO1C), forkhead box P1 (FOXP1), cytoplasmic polyadenylation element binding protein 4 (CPEB4) and glucocorticoid induced 1 (GLCCI1)] with the highest degrees of association with osteosarcoma development were identified.
Affiliation: Department of Blood Transfusion, Tianjin Hospital, Tianjin 300211, P.R. China. Department of Intensive Care Unit, Tianjin Medical University Cancer Institute and Hospital, Tianjin 300060, P.R. China. .
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5: Title: WD40-repeat 47, a microtubule-associated protein, is essential for brain development and autophagy.
Authors: Kannan, Meghna, et.al. .
Journal: Proceedings of the National Academy of Sciences of the United States of America (Proc Natl Acad Sci U S A), Vol. 114 (44): E9308-E9317, 2017 .
Snippet: Remarkably, all seven genes showed corpus callosum defects, including thicker (Atg16l1, Coro1c, Dmxl2, and Herc1), thinner (Kif21b and Wdr89), or absent corpus callosum (Wdr47), revealing a common role for WDR genes in brain connectivity.
Affiliation: Department of Translational Medicine and Neurogenetics, Institut de Génétique et de Biologie Moléculaire et Cellulaire, 67404 Illkirch, France. Centre National de la Recherche Scientifique, UMR7104, 67404 Illkirch, France. Institut National de la Santé et de la Recherche Médicale, U964, 67404 Illkirch, France. Université de Strasbourg, 67404 Illkirch, France. Center for Integrative Genomics, University of Lausanne, CH-1015 Lausanne, Switzerland. Génétique Moléculaire Génomique Microbiologie, UMR7156, Université de Strasbourg, CNRS, 67000 Strasbourg, France. Department of Physiological Sciences, University of Stellenbosch, 7600 Stellenbosch, South Africa. South African Medical Research Council Centre for Tuberculosis Research, Department of Biomedical Sciences, University of Stellenbosch, 7505 Tygerberg, South Africa. ICube, UMR 7357, Fédération de Médecine Translationnelle, University of Strasbourg, 67085 Strasbourg, France. Neuropsychiatry and Neurorehabilitation Unit, Scientific Institute, Istituto di Ricovero e Cura a Carattere Scientifico Eugenio Medea, 23842 Bosisio Parini, Lecco, Italy. Wellcome Trust Sanger Institute, Hinxton, CB10 1SA Cambridge, United Kingdom. Centre des Sciences du Goût et de l'Alimentation, Université de Bourgogne-Franche Comté, 21000 Dijon, France. Department of Translational Medicine and Neurogenetics, Institut de Génétique et de Biologie Moléculaire et Cellulaire, 67404 Illkirch, France; Binnaz.Yalcin@igbmc.fr. .
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6: Title: Proteomic alterations of fibroblasts induced by ovarian cancer cells reveal potential cancer targets.
Authors: Zhang, X Y, et.al. .
Journal: Neoplasma, 2017 .
Snippet: Our data showed that the level of CENPE, BAG2, SOD2, GDI2, CORO1C, CFL1, DSTN, CALD1, PHGDH, PDHA1, AKR1B1, TST and TBCA proteins were significantly up-regulated in the fibroblasts co-cultured with ovarian cancer cells, whereas HSPB1, P4HB and VIM were significantly down-regulated.
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7: Title: Modifier genes: Moving from pathogenesis to therapy.
Journal: Molecular genetics and metabolism (Mol Genet Metab), Vol. 122 (1-2): 1-3, 2017 .
Snippet: A recent article explored protective modifiers, including plastin 3 (PLS3) and coronin 1C (CORO1C), in spinal muscular atrophy (SMA).
Affiliation: March of Dimes Foundation, United States; Department of Pediatrics, David Geffen School of Medicine at UCLA, United States. Electronic address: emccabe@mednet.ucla.edu. .
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8: Title: YBX1 gene silencing inhibits migratory and invasive potential via CORO1C in breast cancer in vitro.
Authors: Lim, Jia Pei, et.al. .
Journal: BMC cancer, Vol. 17 (1): 201, 2017 .
Snippet: Global gene expression profiling in the YBX1 silenced MDA-MB-231 cells identified differential expression of several genes, including CORO1C (which encodes for an actin binding protein, coronin-1C) as a potential downstream target of YB-1.
Affiliation: Department of Anatomy, Yong Loo Lin School of Medicine, National University of Singapore, 4 Medical Drive, Blk MD10, Singapore, 117594, Singapore. Quantitative Proteomics Group, Institute of Molecular and Cell Biology, Agency for Science, Technology and Research, 61 Biopolis Drive, Proteos, Singapore, 138673, Singapore. Laboratory of Cellular Biochemistry, RIKEN, 2-1 Hirosawa, Wako, Saitama, 351-0198, Japan. Department of Anatomy, Yong Loo Lin School of Medicine, National University of Singapore, 4 Medical Drive, Blk MD10, Singapore, 117594, Singapore. boon_huat_bay@nuhs.edu.sg. .
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9: Title: The Power of Human Protective Modifiers: PLS3 and CORO1C Unravel Impaired Endocytosis in Spinal Muscular Atrophy and Rescue SMA Phenotype.
Journal: American journal of human genetics (Am J Hum Genet), Vol. 99 (3): 647-65, 2016 .
Snippet: Similar to PLS3 overexpression, CORO1C overexpression restored fluid-phase endocytosis in SMN-knockdown cells by elevating F-actin amounts and rescued the axonal truncation and branching phenotype in Smn-depleted zebrafish.
Affiliation: Institute of Human Genetics, University of Cologne, 50931 Cologne Germany; Institute for Genetics, University of Cologne, 50674 Cologne, Germany; Center for Molecular Medicine Cologne, University of Cologne, 50931 Cologne, Germany. Center for Biochemistry, Institute of Biochemistry I, Medical Faculty, University of Cologne, 50931 Cologne, Germany. Cologne Excellence Cluster on Cellular Stress Responses in Aging Associated Diseases (CECAD), University of Cologne, 50931 Cologne, Germany. IONIS Pharmaceuticals, 2855 Carlsbad, CA 92008, USA. Biocenter, Institute for Zoology, Neurophysiology, University of Cologne, 50674 Cologne, Germany. Center for Molecular Medicine Cologne, University of Cologne, 50931 Cologne, Germany; Cologne Excellence Cluster on Cellular Stress Responses in Aging Associated Diseases (CECAD), University of Cologne, 50931 Cologne, Germany; Biocenter, Institute for Zoology, Developmental Biology, University of Cologne, 50674 Cologne, Germany. Institute of Human Genetics, University of Cologne, 50931 Cologne Germany. Institute of Human Genetics, University of Cologne, 50931 Cologne Germany; Institute for Genetics, University of Cologne, 50674 Cologne, Germany; Center for Molecular Medicine Cologne, University of Cologne, 50931 Cologne, Germany. Electronic address: brunhilde.wirth@uk-koeln.de. .
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10: Title: New molecular insights into modulation of platelet reactivity in aspirin-treated patients using a network-based approach.
Authors: Zufferey, Anne, et.al. .
Journal: Human genetics (Hum Genet), Vol. 135 (4): 403-14, 2016 .
Snippet: The predicted targets of these miRNAs were mapped onto the network, allowing the identification of seven overlapping genes (THBS1, CDC42, CORO1C, SPTBN1, TPM3, GTPBP2, and MAPRE2), suggesting a synergistic effect of these two miRNAs on these predicted targets.
Affiliation: Division of Angiology and Haemostasis, University Hospitals of Geneva, Rue Gabrielle-Perret-Gentil 4, 1205, Geneva, Switzerland. Translational Biomarker Group, Human Protein Sciences Department, University of Geneva, Geneva, Switzerland. Geneva Platelet Group, University of Geneva, Geneva, Switzerland. Vital-IT Group, SIB Swiss Institute of Bioinformatics, University of Lausanne, Lausanne, Switzerland. Division of Internal Medicine, and Rehabilitation, Trois-Chêne Hospital, University Hospitals of Geneva, Geneva, Switzerland. iGE3 Genomics Platform, University of Geneva, Geneva, Switzerland. Swiss-Prot Group, SIB Swiss Institute of Bioinformatics, Geneva, Switzerland. Division of Angiology and Haemostasis, University Hospitals of Geneva, Rue Gabrielle-Perret-Gentil 4, 1205, Geneva, Switzerland. pierre.fontana@hcuge.ch. Geneva Platelet Group, University of Geneva, Geneva, Switzerland. pierre.fontana@hcuge.ch. .
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11: Title: A common variant association study reveals novel susceptibility loci for low HDL-cholesterol levels in ethnic Arabs.
Authors: Wakil, S M, et.al. .
Journal: Clinical genetics (Clin Genet), Vol. 90 (6): 518-525, 2016 .
Snippet: Seven other variants including rs1147169 in the PLCL1 gene, rs10248618 in the DNAH11, rs476155 in the GLIS3, rs7024300 in the ABCA1, intergenic rs10836699, rs11603691 in P2RX3 and rs750134 in CORO1C gene exhibited borderline protective properties.
Affiliation: Genetics Department, King Faisal Specialist Hospital & Research Centre, Riyadh, Saudi Arabia. Western Australian Institute for Medical Research, University of Western Australia, Perth, Australia. King Faisal Heart Institute, King Faisal Specialist Hospital and Research Centre, Riyadh, Saudi Arabia. .
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12: Title: Proteomic screening and identification of microRNA-128 targets in glioma cells.
Authors: Yang, Bin, et.al. .
Journal: Proteomics, Vol. 15 (15): 2602-17, 2015 .
Snippet: A luciferase assay validated that 11 of 13 selected genes were potential targets of miR-128, and a mutagenesis experiment confirmed CBFB, CORO1C, GLTP, HnRNPF, and TROVE2 as the target genes.
Affiliation: The State Key Laboratory of Medical Molecular Biology, Department of Molecular Biology and Biochemistry, Institute of Basic Medical Sciences, Chinese Academy of Medical Sciences and Peking Union Medical College, Beijing, P. R. China. The State Key Laboratory of Proteomics, Beijing Proteome Research Center, Beijing Institute of Radiation Medicine, Beijing, P. R. China. .
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13: Title: Downregulation of the microRNA-1/133a cluster enhances cancer cell migration and invasion in lung-squamous cell carcinoma via regulation of Coronin1C.
Authors: Mataki, Hiroko, et.al. .
Journal: Journal of human genetics (J Hum Genet), Vol. 60 (2): 53-61, 2015 .
Snippet: Coronin-1C (CORO1C) was a common target gene of the miR-1/133a cluster, as shown by the genome-wide gene expression analysis and the luciferase reporter assay.
Affiliation: Department of Pulmonary Medicine, Graduate School of Medical and Dental Sciences, Kagoshima University, Kagoshima, Japan. Department of Urology, Graduate School of Medical and Dental Sciences, Kagoshima University, Kagoshima, Japan. Department of Functional Genomics, Graduate School of Medicine, Chiba University, Chiba, Japan. .
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14: Title: Coronin-1C and RCC2 guide mesenchymal migration by trafficking Rac1 and controlling GEF exposure.
Authors: Williamson, Rosalind C, et.al. .
Journal: Journal of cell science (J Cell Sci), Vol. 127 (Pt 19): 4292-307, 2014 .
Snippet: Coro1C mediates release of inactive Rac1 from non-protrusive membrane and is necessary for Rac1 redistribution to a protrusive tip and fibronectin-dependent Rac1 activation.
Affiliation: School of Biochemistry, University of Bristol, University Walk, Bristol BS8 1TD, UK. Department of Engineering, University of Bristol, University Walk, Bristol BS8 1TD, UK. School of Biochemistry, University of Bristol, University Walk, Bristol BS8 1TD, UK mark.bass@bristol.ac.uk. .
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15: Title: miR-206 inhibits cell migration through direct targeting of the actin-binding protein coronin 1C in triple-negative breast cancer.
Authors: Wang, Jun, et.al. .
Journal: Molecular oncology (Mol Oncol), Vol. 8 (8): 1690-702, 2014 .
Snippet: Further, silencing of CORO1C reduced tumor cell migration and affected the actin skeleton and cell morphology, similar to miR-206 expression, but did not reduce proliferation.
Affiliation: Center for Nuclear Receptors and Cell Signaling, Department of Biology and Biochemistry, University of Houston, TX 77204-5506, USA. Department of Oncology and Pathology, Karolinska Institutet and University Hospital, S-171 76 Stockholm, Sweden. Center for Nuclear Receptors and Cell Signaling, Department of Biology and Biochemistry, University of Houston, TX 77204-5506, USA. Electronic address: ceciliawilliams@uh.edu. .
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16: Title: Cortisol treatment of prespawning female cod affects cytogenesis related factors in eggs and embryos.
Authors: Kleppe, Lene, et.al. .
Journal: General and comparative endocrinology (Gen Comp Endocrinol), Vol. 189, 2013 .
Snippet: Among these differentially expressed genes, some were found to be linked to cytogenesis (stxbp6, fbxw2, capn12, thbs4, sytl2, coro1c, sel1l3), induction of mesodermal fate (fgfrl1) and import of the glucocorticoid receptor to the cell nucleus (ipo7).
Affiliation: Institute of Marine Research, P. O. Box 1870, 5817 Bergen, Norway. lene.kleppe@imr.no .
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17: Title: Role of coronin 1B in PDGF-induced migration of vascular smooth muscle cells.
Authors: Williams, Holly C, et.al. .
Journal: Circulation research (Circ Res), Vol. 111 (1): 56-65, 2012 .
Snippet: Downregulation of Coro1B by siRNA increases PDGF-induced migration, while downregulation of Coro1C has no effect.
Affiliation: Department of Medicine, Division of Cardiology, Emory University School of Medicine, Atlanta, Georgia, USA. .
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18: Title: Proteomic analysis of 1α,25-dihydroxyvitamin D3 action on human colon cancer cells reveals a link to splicing regulation.
Authors: Cristobo, Iván, et.al. .
Journal: Journal of proteomics (J Proteomics), Vol. 75 (2): 384-97, 2011 .
Snippet: In addition, a smaller group of proteins (ERM (Ezrin, Radixin, Moesin) family, VCL, CORO1C, ACTB) were cytoskeleton-associated and played a role in cell adhesion and morphology.
Affiliation: Functional Proteomics Laboratory, Centro de Investigaciones Biológicas, Consejo Superior de Investigaciones Científicas, Madrid, Spain. .
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19: Title: Profiling gene expression induced by protease-activated receptor 2 (PAR2) activation in human kidney cells.
Authors: Suen, Jacky Y, et.al. .
Journal: PloS one, Vol. 5 (11): e13809, 2010 .
Snippet: Both PAR2 and PAR1 activation resulted in up-regulated expression of several genes (CD44, FOSL1, TNFRSF12A, RAB3A, COPEB, CORO1C, THBS1, SDC4) known to be important in cancer.
Affiliation: Institute for Molecular Bioscience, The University of Queensland, Brisbane, Queensland, Australia. .
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20: Title: Primary effusion lymphoma: genomic profiling revealed amplification of SELPLG and CORO1C encoding for proteins important for cell migration.
Authors: Luan, Shi-Lu, et.al. .
Journal: The Journal of pathology (J Pathol), Vol. 222 (2): 166-79, 2010 .
Snippet: SELPLG is critical for cell migration and chemotaxis, while CORO1C regulates actin-dependent processes, thus important for cell motility.
Affiliation: Department of Pathology, University of Cambridge, Cambridge, CB2 0QQ, UK. .
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