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12 documents found
1: Title: Structure and function of the contactin-associated protein family in myelinated axons and their relationship with nerve diseases.
Authors: Zou, Yan, et.al. .
Journal: Neural regeneration research (Neural Regen Res), Vol. 12 (9): 1551-1558, 2017 .
Snippet: We analyzed the structures and functions of the Caspr family-CNTNAP1 (Caspr1), CNTNAP2 (Caspr2), CNTNAP3 (Caspr3), CNTNAP4 (Caspr4) and CNTNAP5 (Caspr5), Caspr1-5 is not only involved in the formation of myelinated axons, but also participates in maintaining the stability of adjacent connections.
Affiliation: Department of Neurology, The Second People's Hospital of Wuxi, Wuxi, Jiangsu Province, China. .
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2: Title: Elevated mRNA expression of CASPR3 in patients with schizophrenia.
Authors: Okita, Mitsuo, et.al. .
Journal: Nordic journal of psychiatry (Nord J Psychiatry), Vol. 71 (4): 312-314, 2017 .
Snippet: BACKGROUND: Recently, it was reported that antipsychotic treatment reverted Contactin Associated Protein-Like 3 (CASPR3, same as CNTNAP3) mRNA expressions in leukocytes of schizophrenia (SCZ) subjects to the same levels as healthy controls.
Affiliation: a Department of Neuropsychiatry, Molecules and Function , Ehime University Graduate School of Medicine , Toon , Ehime , Japan. .
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3: Title: Caspr3-Deficient Mice Exhibit Low Motor Learning during the Early Phase of the Accelerated Rotarod Task.
Authors: Hirata, Haruna, et.al. .
Journal: PloS one, Vol. 11 (1): e0147887, 2016 .
Snippet: Caspr3 (Contactin-associated protein-like 3, Cntnap3) is a neural cell adhesion molecule belonging to the Caspr family.
Affiliation: Department of Bioengineering, Nagaoka University of Technology, Nagaoka, Niigata, 940-2188, Japan. Mouse Genomics Resource Laboratory, National Institute of Genetics (NIG), Mishima, Shizuoka, 411-8540, Japan. Department of Genetics, The Graduate University for Advanced Studies (SOKENDAI), Mishima, Shizuoka, 411-8540, Japan. Transdisciplinary Research Integration Center, Research Organization of Information and Systems, Minato-ku, Tokyo, 105-0001, Japan. Laboratory of Behavioral Neuroendocrinology, University of Tsukuba, Tsukuba, Ibaraki, 305-8577, Japan. .
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4: Title: CNTNAP3 associated ATG16L1 expression and Crohn's disease.
Authors: Qiao, Yu Qi, et.al. .
Journal: Mediators of inflammation (Mediators Inflamm), Vol. 2015, 2015 .
Snippet: In vitro study revealed that CNTNAP3 could upregulate the expression of ATG16L1 and increase autophagy vacuoles.
Affiliation: Division of Gastroenterology and Hepatology, Renji Hospital, School of Medicine, Shanghai Jiao Tong University, Shanghai 200127, China ; Shanghai Institute of Digestive Disease, Shanghai 200127, China ; Shanghai Inflammatory Bowel Disease Research Center, Shanghai 200127, China ; Key Laboratory of Gastroenterology & Hepatology, Ministry of Health (Shanghai Jiao Tong University), Shanghai 200127, China. .
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5: Title: Schizophrenia gene expression profile reverted to normal levels by antipsychotics.
Journal: The international journal of neuropsychopharmacology / official scientific journal of the Collegium Internationale Neuropsychopharmacologicum (CINP) (Int J Neuropsychopharmacol), Vol. 18 (4), 2015 .
Snippet: Six genes (ADAMTS2, CD177, CNTNAP3, ENTPD2, RFX2, and UNC45B) out of the 17 are among the 200 genes that we characterized with differential expression in a previous study between antipsychotic-naïve schizophrenia patients and controls (Sainz et al., 2013).
Affiliation: University Hospital Marqués de Valdecilla. Department of Medicine & Psychiatry. School of Medicine University of Cantabria-IDIVAL, Santander, Spain (Dr Crespo-Facorro); CIBERSAM, Centro Investigación Biomédica en Red Salud Mental, Madrid, Spain (Dr Crespo-Facorro); Spanish National Research Council (CSIC), Institute of Biomedicine and Biotechnology of Cantabria, Santander, Spain (Drs Prieto and Sainz). University Hospital Marqués de Valdecilla. Department of Medicine & Psychiatry. School of Medicine University of Cantabria-IDIVAL, Santander, Spain (Dr Crespo-Facorro); CIBERSAM, Centro Investigación Biomédica en Red Salud Mental, Madrid, Spain (Dr Crespo-Facorro); Spanish National Research Council (CSIC), Institute of Biomedicine and Biotechnology of Cantabria, Santander, Spain (Drs Prieto and Sainz). sainzjv@unican.es. .
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6: Title: Genomic screening of testicular germ cell tumors from monozygotic twins.
Journal: Orphanet journal of rare diseases (Orphanet J Rare Dis), Vol. 9, 2014 .
Snippet: In peripheral blood samples, loss of 9p13.1-p12 was the unique novel CNV shared by the twins, confirming the involvement of CNTNAP3 gene in TGCTs development.
Affiliation: Neogene Laboratory, CIPE, A. C. Camargo Cancer Center, São Paulo, Brazil. sara_martorelli@yahoo.com.brl. Department of Pathology, A.C. Camargo Cancer Center, São Paulo, Brazil. iwcunha@accamargo.org.br. Institute of Mathematics and Statistics, Inter-Institutional Program on Bioinformatics, USP, São Paulo, Brazil. fmarchibr@hotmail.com. Neogene Laboratory, CIPE, A. C. Camargo Cancer Center, São Paulo, Brazil. ariane.b@ig.com.br. Nucleus of Sarcoma, Department of Pelvic Surgery, A.C. Camargo Cancer Center, São Paulo, Brazil. ademar-lopes@uol.com.br. Neogene Laboratory, CIPE, A. C. Camargo Cancer Center, São Paulo, Brazil. silvia.rogatto@cipe.accamargo.org.br. Department of Urology, Faculty of Medicine, UNESP, Botucatu, São Paulo, Brazil. silvia.rogatto@cipe.accamargo.org.br. .
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7: Title: Comparative gene array analysis of progenitor cells from human paired deep neck and subcutaneous adipose tissue.
Authors: Tews, D, et.al. .
Journal: Molecular and cellular endocrinology (Mol Cell Endocrinol), Vol. 395 (1-2): 41-50, 2014 .
Snippet: Analysis of highest regulated genes revealed that STMN2, MME, ODZ2, NRN1 and IL13RA2 genes were specifically expressed in white progenitor cells, whereas expression of LRRC17, CNTNAP3, CD34, RGS7BP and ADH1B marked brown progenitor cells.
Affiliation: Division of Pediatric Endocrinology and Diabetes, Department of Pediatrics and Adolescent Medicine, University Medical Center Ulm, Ulm, Germany. Clinic of Otorhinolaryngology, University Medical Center Ulm, Ulm, Germany. Department of Surgery, University Medical Center Ulm, Ulm, Germany. Else-Kröner-Fresenius Center for Nutritional Medicine, Technische Universität München, Munich, Germany. Department of Pathology, University Medical Center Ulm, Ulm, Germany. Core Facility Genomics, Ulm University, Ulm, Germany. Department of Pediatrics and Adolescent Medicine, University Medical Center Ulm, Ulm, Germany. Division of Pediatric Endocrinology and Diabetes, Department of Pediatrics and Adolescent Medicine, University Medical Center Ulm, Ulm, Germany. Electronic address: martin.wabitsch@uniklinik-ulm.de. .
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8: Title: Differences and similarities in the transcriptional profile of peripheral whole blood in early and late-onset preeclampsia: insights into the molecular basis of the phenotype of preeclampsiaa.
Journal: Journal of perinatal medicine (J Perinat Med), Vol. 41 (5): 485-504, 2013 .
Snippet: In contrast, only seven genes that encoded proteins involved in innate immunity (LTF, ELANE) and cell-to-cell recognition in the nervous system (CNTNAP3) were differentially expressed in late-onset PE alone.
Affiliation: Wayne State University/Hutzel Women’s Hospital, MI 48201, USA. tchaiwor@med.wayne.edu .
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9: Title: The clf2 gene has an epigenetic role in the multifactorial etiology of cleft lip and palate in the A/WySn mouse strain.
Authors: Plamondon, Jenna A, et.al. .
Journal: Birth defects research. Part A, Clinical and molecular teratology (Birth Defects Res A Clin Mol Teratol), Vol. 91 (8): 716-27, 2011 .
Snippet: RESULTS: The location of clf2 was redefined to 3.0 Mb between Cntnap3 and AK029746 containing 48 genes, of which 30 are Zfp genes.
Affiliation: Department of Medical Genetics, University of British Columbia, Vancouver, British Columbia, Canada. .
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10: Title: Identification of susceptibility loci at 7q31 and 9p13 for bipolar disorder in an isolated population.
Authors: Palo, Outi M, et.al. .
Journal: American journal of medical genetics. Part B, Neuropsychiatric genetics : the official publication of the International Society of Psychiatric Genetics (Am J Med Genet B Neuropsychiatr Genet), Vol. 153B (3): 723-35, 2010 .
Snippet: This region harbors several interesting candidate genes, including contactin associated protein-like 3 (CNTNAP3) and aldehyde dehydrogenase 1 (ALDH1B1).
Affiliation: FIMM, Institute for Molecular Medicine and National Institute for Health and Welfare, Helsinki, Finland. .
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11: Title: A reciprocal translocation 46,XY,t(8;9)(p11.2;q13) in a bladder exstrophy patient disrupts CNTNAP3 and presents evidence of a pericentromeric duplication on chromosome 9.
Authors: Boyadjiev, Simeon A, et.al. .
Journal: Genomics, Vol. 85 (5): 622-9, 2005 .
Snippet: Additionally, FISH analysis identified several genomic copies of CNTNAP3 on both sides of the chromosome 9 centromere flanking the polymorphic heterochromatin.
Affiliation: McKusick-Nathans Institute of Genetic Medicine, The Johns Hopkins University School of Medicine, 733 N. Broadway, BRB 469, Baltimore, MD 21205, USA. sboyd@mail.jhmi.edu .
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12: Title: Caspr3 and caspr4, two novel members of the caspr family are expressed in the nervous system and interact with PDZ domains.
Authors: Spiegel, Ivo, et.al. .
Journal: Molecular and cellular neurosciences (Mol Cell Neurosci), Vol. 20 (2): 283-97, 2002 .
Snippet: Similar to the neurexins, the cytoplasmic tails of Caspr3 and Caspr4 interacted differentially with PDZ domain-containing proteins of the CASK/Lin2-Veli/Lin7-Mint1/Lin10 complex.
Affiliation: Department of Molecular Cell Biology, The Weizmann Institute of Science, Rehovot 76100, Israel. .
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