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GoPubMed Proteins lists recent and important papers and reviews for proteins. Page last changed on 19 Dec 2016.

Tripartite motif containing 59

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Top mentioned proteins: p53, Trim, Ubiquitin, V1a, POLYMERASE
Papers on TRIM59
Development of a forensically useful age prediction method based on DNA methylation analysis.
Branicki et al., Warsaw, Poland. In Forensic Sci Int Genet, Jul 2015
TRIM59 on 3q25.33,
TRIM59 Promotes the Proliferation and Migration of Non-Small Cell Lung Cancer Cells by Upregulating Cell Cycle Related Proteins.
Wang et al., Nanchang, China. In Plos One, 2014
In an effort to profile the expression patterns of TRIM superfamily in several non-small cell lung cancer (NSCLC) cell lines, we found that the expression of 10 TRIM genes including TRIM3, TRIM7, TRIM14, TRIM16, TRIM21, TRIM22, TRIM29, TRIM59, TRIM66 and TRIM70 was significantly upregulated in NSCLC cell lines compared with the normal human bronchial epithelial (HBE) cell line, whereas the expression of 7 other TRIM genes including TRIM4, TRIM9, TRIM36, TRIM46, TRIM54, TRIM67 and TRIM76 was significantly down-regulated in NSCLC cell lines compared with that in HBE cells.
TRIM59 is up-regulated in gastric tumors, promoting ubiquitination and degradation of p53.
Gao et al., Shanghai, China. In Gastroenterology, 2014
We found expression of tripartite motif 59 (TRIM59), which encodes a putative ubiquitin ligase, to be increased, and investigated its effects in gastric cancer cell lines.
TRIM59 interacts with ECSIT and negatively regulates NF-κB and IRF-3/7-mediated signal pathways.
Hatakeyama et al., Sapporo, Japan. In Biochem Biophys Res Commun, 2012
These findings indicate that TRIM59 may serve as a multifunctional regulator for innate immune signaling pathways.
TRIM59, a novel multiple cancer biomarker for immunohistochemical detection of tumorigenesis.
Xuan et al., London, Canada. In Bmj Open, 2011
OBJECTIVES AND DESIGN: We identified a novel TRIM59 gene, as an early signal transducer in two (SV40Tag and Ras) oncogene pathways in murine prostate cancer (CaP) models.
Characterization of the oncogenic activity of the novel TRIM59 gene in mouse cancer models.
Xuan et al., London, Canada. In Mol Cancer Ther, 2011
A novel TRIM family member, TRIM59 gene was characterized to be upregulated in SV40 Tag oncogene-directed transgenic and knockout mouse prostate cancer models as a signaling pathway effector.
Transcriptional regulation of Wnt inhibitory factor-1 by Miz-1/c-Myc.
Herman et al., Baltimore, United States. In Oncogene, 2010
In a genome-wide screen, DNAJA4, TGFβ-induced and TRIM59 were repressed by c-Myc overexpression and DNA promoter hypermethylation.
Identification and characterization of a long isoform of human IFT80, IFT80-L.
Li et al., Charlottesville, United States. In Biochem Biophys Res Commun, 2008
Sequence analysis indicates that IFT80-L is likely an evolutionarily merged product of genes IFT80 and TRIM59, a RING finger gene we reported previously.
Molecular cloning, mapping and characterization of a novel mouse RING finger gene, Mrf1.
Li et al., Memphis, United States. In Gene, 2002
PMID:12095697 pertains to mouse Mrf1 gene which appears to be the homolog of human TSBF1.
Circulating immune complexes in serum and in cerebrospinal fluid of patients with multiple sclerosis. Characterization and correlation with the clinical course.
Blasio et al., Milano, Italy. In Acta Neurol Scand, 1988
No significant increase in serum or in CSF were found using the mRF-I test.
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