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GoPubMed Proteins lists recent and important papers and reviews for proteins. Page last changed on 19 Dec 2016.

ABI family, member 3

TARSH, Abi3bp, ABI gene family member 3 binding protein
Top mentioned proteins: Akt, p53, p21, V1a, lacZ
Papers on TARSH
Meta-analysis of Genome-Wide Association Studies Identifies Novel Loci Associated With Optic Disc Morphology.
Hammond et al., Rotterdam, Netherlands. In Genet Epidemiol, Mar 2015
We identified 10 new loci associated with disc area (CDC42BPA, F5, DIRC3, RARB, ABI3BP, DCAF4L2, ELP4, TMTC2, NR2F2, and HORMAD2) and another 10 new loci associated with cup area (DHRS3, TRIB2, EFEMP1, FLNB, FAM101, DDHD1, ASB7, KPNB1, BCAS3, and TRIOBP).
Novel identification of matrix proteins involved in calcitic biomineralization.
Hincke et al., Ottawa, Canada. In J Proteomics, Mar 2015
Among the most promising candidates for modulating protein-crystal interactions were extracellular matrix proteins, including ABI family member 3 (NESH) binding protein (ABI3BP), tiarin-like, hyaluronan and proteoglycan link protein 3 (HAPLN3), collagen alpha-1(X), collagen alpha-1(II) and fibronectin, in addition to the calcium binding proteins calumenin, EGF-like repeats and discoidin 1-like domains 3 (EDIL3), nucleobindin-2 and SPARC.
Abi3bp regulates cardiac progenitor cell proliferation and differentiation.
Dzau et al., Durham, United States. In Circ Res, 2015
We have recently identified ABI family, member 3 (NESH) binding protein (Abi3bp) as a protein important for mesenchymal stem cell biology.
Bioinformatics-Based Identification of MicroRNA-Regulated and Rheumatoid Arthritis-Associated Genes.
Yang et al., Chongqing, China. In Plos One, 2014
The result revealed that the expression of a number of genes, including ROR2, ABI3BP, SMOC2, etc., was not only affected by dysregulated miRNAs, but also altered in RA.
Genome-wide copy number variation study and gene expression analysis identify ABI3BP as a susceptibility gene for Kashin-Beck disease.
Deng et al., Xi'an, China. In Hum Genet, 2014
GCNVS found that CNV452 (P value = 7.78 × 10(-5)) overlapping with ABI3BP gene was significantly associated with KBD.
Genome-wide association study in NSAID-induced acute urticaria/angioedema in Spanish and Han Chinese populations.
Lee et al., Málaga, Spain. In Pharmacogenomics, 2013
RESULTS: Although no polymorphism reached genome-wide significance, we obtained suggestive associations for three clusters in the Spanish group (RIMS1, BICC1 and RAD51L 1) and one region in the Han Chinese population (ABI3BP).
Abi3bp is a multifunctional autocrine/paracrine factor that regulates mesenchymal stem cell biology.
Dzau et al., Durham, United States. In Stem Cells, 2013
In this study, we investigated the relevance of Abi3bp toward MSC biology.
ABI3 ectopic expression reduces in vitro and in vivo cell growth properties while inducing senescence.
Cerutti et al., São Paulo, Brazil. In Bmc Cancer, 2010
METHODS: The present study investigated ABI3 expression in a large panel of benign and malignant thyroid tumors and explored a correlation between the expression of ABI3 and its potential partner ABI3-binding protein (ABI3BP).
Genome-wide association study of suicide attempts in mood disorder patients.
Smoller et al., Boston, United States. In Am J Psychiatry, 2010
In major depression, strongest evidence of association was observed for a single nucleotide polymorphism in ABI3BP, with six loci also showing suggestive association.
A novel p53-dependent apoptosis function of TARSH in tumor development.
Maruyama et al., Ōbu, Japan. In Nagoya J Med Sci, 2009
a p53-dependent apoptosis function of the mouse TARSH gene.
Implication of p53-dependent cellular senescence related gene, TARSH in tumor suppression.
Maruyama et al., Ōbu, Japan. In Biochem Biophys Res Commun, 2009
These results suggest that TARSH plays an important role in proliferation of replicative senescence and may serve as a trigger of tumor development.
Secreted TARSH regulates olfactory mitral cell dendritic complexity.
Gong et al., Davis, United States. In Eur J Neurosci, 2009
Through a genome-wide microarray screen, we identified TARSH (Target of NESH SH3) as a transiently expressed molecule in mitral cells during the dendritic refinement period.
Re-expression of ABI3-binding protein suppresses thyroid tumor growth by promoting senescence and inhibiting invasion.
Cerutti et al., São Paulo, Brazil. In Endocr Relat Cancer, 2008
The loss of ABI3BP expression could play a functional role in thyroid tumorigenesis.
Generation and characterization of novel monoclonal antibodies against murine and human TARSH proteins.
Maruyama et al., Ōbu, Japan. In Hybridoma (larchmt), 2007
TARSH/Abi3bp was originally isolated as a novel target of NESH-SH3 by a two-hybrid yeast system.
NESH (Abi-3) is present in the Abi/WAVE complex but does not promote c-Abl-mediated phosphorylation.
Tani et al., Hachiōji, Japan. In Febs Lett, 2006
NESH (Abi-3), like Abi-1 and Abi-2, is a component of the Abi/WAVE complex, but likely plays a different role in the regulation of c-Abl.
Identification of candidates for tumor-specific alternative splicing in the thyroid.
Cerutti et al., São Paulo, Brazil. In Genes Chromosomes Cancer, 2006
Five genes (PTPN18, ABI3BP, PFDN5, SULF2, and ST5) presented new and/or additional unpredicted isoforms differentially expressed between malignant and benign or normal thyroid tissues, confirmed by sequencing.
Cancer-associated loss of TARSH gene expression in human primary lung cancer.
Fushiki et al., Kyoto, Japan. In J Cancer Res Clin Oncol, 2006
expression of TARSH mRNA is remarkably downregulated in all lung cancer cell lines examined; also TARSH expression was significantly low in all of the tumor specimens when compared to the expression in corresponding non-neoplastic lung tissue specimens
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