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GoPubMed Proteins lists recent and important papers and reviews for proteins. Page last changed on 19 Dec 2016.

Low density lipoprotein receptor-related protein 12

Suppression of tumorigenicity, ST7
This gene encodes a member of the low-density lipoprotein receptor related protein family. The product of this gene is a transmembrane protein that is differentially expressed in many cancer cells. Alternate splicing results in multiple transcript variants. [provided by RefSeq, Feb 2010] (from NCBI)
Top mentioned proteins: HAD, ST1, ST5, CAN, St-4
Papers on Suppression of tumorigenicity
Changing epidemiology of group B streptococcal infections among adults in Iceland: 1975-2014.
Ramirez et al., Lisbon, Portugal. In Clin Microbiol Infect, Jan 2016
Nine cases of invasive GBS disease were caused by ST7, of possible zoonotic origin.
Prevalence and Genotype Characterization of Blastocystis hominis Among the Baghmalek People in Southwestern Iran in 2013 - 2014.
Alizadeh et al., Ahvāz, Iran. In Jundishapur J Microbiol, Oct 2015
The Blastocystis subtypes (ST) identified in this study were ST3, ST4, ST5, and ST7 with the most prevalent being ST4 (40.9%).
Genotypes of Clinical and Environmental Isolates of Cryptococcus neoformans and Cryptococcus gattii in Korea.
Hwang et al., Pusan, South Korea. In Mycobiology, Sep 2015
Three C. gattii strains were identified as ST57, ST7, and ST113.
ST2 and patient prognosis in chronic heart failure.
Ky et al., Barcelona, Spain. In Am J Cardiol, May 2015
Suppression of tumorigenicity 2 (ST2) is a marker of cardiomyocyte stress and fibrosis that provides incremental value to natriuretic peptides for risk stratification of patients with a wide spectrum of cardiovascular diseases.
Soluble ST2--analytical considerations.
Jaffe et al., Linz, Austria. In Am J Cardiol, May 2015
Suppression of tumorigenicity 2 (ST2, also known as interleukin [IL]-1 receptor-like-1) is an IL-1 receptor family member with transmembrane (ST2L) and soluble isoforms (sST2).
Maleewong et al., In Southeast Asian J Trop Med Public Health, Mar 2015
BLAST search and phylogenetic analysis demonstrated that Blastocystis subtypes ST1 (20%), ST3 (60%), ST6 (10%) and ST7 (10%) were present.
Seeing the Whole Elephant: Imaging Flow Cytometry Reveals Extensive Morphological Diversity within Blastocystis Isolates.
Tan et al., Singapore, Singapore. In Plos One, 2014
In this study, we used imaging flow cytometry to analyze morphological features of Blastocystis isolates representing 3 subtypes (ST1, ST4 and ST7).
[Target points: a discussion on acupuncture treatment of primary trigeminal neuralgia].
Xu et al., Beijing, China. In Zhong Xi Yi Jie He Xue Bao, 2012
This study summarized the method of using acupuncture at Xiaguan (ST7), Cuanzhu (BL2), Sibai (ST2) and Jiachengjiang acupoints to align with the spheno-palatine ganglion and additional nerve foramen (supraorbital, infraorbital and mental foramina) to treat primary trigeminal neuralgia.
Localization and characterization of ST7 in cancer.
Zenklusen et al., Nakhon Pathom, Thailand. In J Cancer Res Clin Oncol, 2011
ST7 mediates tumor suppression through the regulation of the genes involved in maintaining the cellular structure of the cell and involved in oncogenic pathways
Lrp12/Mig13a reveals changing patterns of preplate neuronal polarity during corticogenesis that are absent in reeler mutant mice.
Hatten et al., New York City, United States. In Cereb Cortex, 2011
As the preplate separates, Lrp12/Mig13a-positive neurons polarize in the radial plane and form a pseudocolumnar pattern, prior to moving to a deeper position within the emerging subplate layer.
Evidence based acupuncture practice recommendations for peripheral facial paralysis.
Chen et al., Chengdu, China. In Am J Chin Med, 2008
On the whole, when treating PFP, the best acupoints options are Dicang (ST4), Xiaguan (ST7), Jiache (ST6), Chengjiang (CV24), Yingxiang (LI20), Quanliao (SI18), Yifeng (TE17), Yangbai (GB14), Sibai (ST2), Fengchi (GB20), Shuigou (GV26), Yuyao (EX-HN4) and Hegu (LI4).
WNT2B: comparative integromics and clinical applications (Review).
Katoh, Tokyo, Japan. In Int J Mol Med, 2005
and WNT2-ST7-CAPZA2 locus at human chromosome 7q31.2
Human SWI/SNF-associated PRMT5 methylates histone H3 arginine 8 and negatively regulates expression of ST7 and NM23 tumor suppressor genes.
Sif et al., Columbus, United States. In Mol Cell Biol, 2004
suppressor of tumorigenicity 7 (ST7) and nonmetastatic 23 (NM23) are direct targets of PRMT5-containing BRG1 and hBRM complexes
Somatic mutation analysis of p53 and ST7 tumor suppressor genes in gastric carcinoma by DHPLC.
Zhang et al., Shenyang, China. In World J Gastroenterol, 2003
ST7 gene may not be the target gene of inactivation at 7q31 site in gastric carcinoma.
Mutational analysis of the ST7 gene in human myeloid tumor cell lines.
Hosokawa et al., Nagoya, Japan. In Oncol Rep, 2003
somatic mutations of ST7 do not commonly contribute to the molecular pathogenesis of human malignant myeloid tumors
Absence of ST7 mutations in tumor-derived cell lines and tumors.
Black et al., Glasgow, United Kingdom. In Nat Genet, 2001
The gene ST7 has been proposed as the multi-tissue tumor-suppressor gene (TSG) at chromosome 7q31.1.
Mutation of the ST7 tumor suppressor gene on 7q31.1 is rare in breast, ovarian and colorectal cancers.
Campbell et al., Melbourne, Australia. In Nat Genet, 2001
The gene ST7 has recently been implicated as the broad-range tumor suppressor on human chromosome 7q31.1.
Mutational and functional analyses reveal that ST7 is a highly conserved tumor-suppressor gene on human chromosome 7q31.
Green et al., Bethesda, United States. In Nat Genet, 2001
Using a positional cloning strategy and aided by the availability of near-complete sequence of this genomic interval, we have identified a TSG within 7q31, named ST7 (for suppression of tumorigenicity 7; this same gene was recently reported in another context and called RAY1).
Suppression of tumorigenicity in human colon carcinoma cells by introduction of normal chromosome 5 or 18.
Miyaki et al., Tokyo, Japan. In Nature, 1991
Development of colon carcinomas can be associated with allelic deletions on several chromosomes, including 5q and 18q.
Suppression of tumorigenicity of human prostate carcinoma cells by replacing a mutated RB gene.
Lee et al., San Diego, United States. In Science, 1990
Introduction of a normal retinoblastoma gene (RB) into retinoblastoma cells was previously shown to suppress several aspects of their neoplastic phenotype, including tumorigenicity in nude mice, thereby directly demonstrating a cancer suppression function of RB.
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