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GoPubMed Proteins lists recent and important papers and reviews for proteins. Page last changed on 19 Dec 2016.

Serine/threonine kinase 32B

Top mentioned proteins: LBN, CAN, GPR39, XRCC4, NCX3
Papers on STK32B
A genome-wide association study of suicidal behavior.
Mann et al., München, Germany. In Am J Med Genet B Neuropsychiatr Genet, Oct 2015
The top SNPs for the suicide attempt analysis included two from DPP10, one from CTNNA3 and one from STK32B.
Genome-Wide Specific Selection in Three Domestic Sheep Breeds.
Du et al., Beijing, China. In Plos One, 2014
For meat traits, ALDOA, STK32B and FAM190A are related to marbling.
Identification of Human N-Myristoylated Proteins from Human Complementary DNA Resources by Cell-Free and Cellular Metabolic Labeling Analyses.
Utsumi et al., Yamaguchi, Japan. In Plos One, 2014
As a result, the products of 13 cDNA clones (FBXL7, PPM1B, SAMM50, PLEKHN, AIFM3, C22orf42, STK32A, FAM131C, DRICH1, MCC1, HID1, P2RX5, STK32B) were found to be human N-myristoylated proteins.
Clinical relevance of breast cancer-related genes as potential biomarkers for oral squamous cell carcinoma.
Helou et al., Göteborg, Sweden. In Bmc Cancer, 2013
RESULTS: We found that CBX2, SCUBE2, and STK32B protein expression were associated with important clinicopathological features for OSCC (peritumoral inflammatory infiltration, metastatic spread to the cervical lymph nodes, and tumor size).
Two-marker association tests yield new disease associations for coronary artery disease and hypertension.
Elston et al., Cleveland, United States. In Hum Genet, 2011
For CAD, we detected single nucleotide polymorphisms (SNP) pairs in three genes showing genome-wide significance: HFE2, STK32B, and DIPC2.
Association between genes on chromosome 4p16 and non-syndromic oral clefts in four populations.
Beaty et al., Baltimore, United States. In Eur J Hum Genet, 2010
STK32B and EVC yielded consistent evidence from cleft lip, with or without cleft palate, trios in all four populations.
Long interspersed nuclear element-1 (LINE1)-mediated deletion of EVC, EVC2, C4orf6, and STK32B in Ellis-van Creveld syndrome with borderline intelligence.
Ruiz-Perez et al., Cairo, Egypt. In Hum Mutat, 2008
In a consanguineous pedigree diagnosed with EvC and borderline intelligence, a 520-kb homozygous deletion comprising EVC, EVC2, C4orf6, and STK32B, caused by recombination between LINE-1 elements, was detected
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