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GoPubMed Proteins lists recent and important papers and reviews for proteins. Page last changed on 19 Dec 2016.

Aldehyde dehydrogenase 3 family, member A2

Top mentioned proteins: ACID, CAN, HAD, V1a, OUT
Papers using SLS antibodies
Design of a new multiparticulate system for potential site-specific and controlled drug delivery to the colonic region
Dorkoosh F. et al., In Research in Pharmaceutical Sciences, 1997
... grade hydrochloric acid, potassium dihydrogen phosphate, sodium dihydrogen phosphate, sodium hydroxide and sodium lauryl sulphate (SLS) were purchased from Merck (Darmstadt, Germany) ...
Papers on SLS
How the cation 1-butyl-3-methylimidazolium impacts the interaction between the entrapped water and the reverse micelle interface created with an ionic liquid-like surfactant.
Falcone et al., Río Cuarto, Argentina. In Soft Matter, Feb 2016
The behavior of the interfacial water entrapped in reverse micelles (RMs) formed by the ionic liquid-like surfactant 1-butyl-3-methylimidazolium 1,4-bis-2-ethylhexylsulfosuccinate (bmim-AOT) dissolved in benzene (or chlorobenzene) was investigated using noninvasive techniques such as dynamic light scattering (DLS), static light scattering (SLS), FT-IR and (1)H NMR.
Improving Oral Bioavailability of Sorafenib by Optimizing the "Spring" and "Parachute" Based on Molecular Interaction Mechanisms.
Qian et al., Beijing, China. In Mol Pharm, Feb 2016
In this study, we observed that the solubility of sorafenib could be increased ∼50-fold in the coexistence of poly(vinylpyrrolidone-vinyl acetate) (PVP-VA) and sodium lauryl sulfate (SLS), due to the formation of PVP-VA/SLS complexes at a lower critical aggregation concentration.
Cerebral venous thrombosis in a Caribbean population.
Landais et al., Pointe-à-Pitre, Guadeloupe. In Rev Neurol (paris), Dec 2015
Thrombosis of the SLS (60%), and of the lateral sinuses (64.4%) were the most frequently encountered.
Plasmalogens and fatty alcohols in rhizomelic chondrodysplasia punctata and Sjögren-Larsson syndrome.
Brites et al., Porto, Portugal. In J Inherit Metab Dis, 2015
Sjögren-Larsson syndrome (SLS) is caused by defects in the microsomal fatty aldehyde dehydrogenase (FALDH) leading to the accumulation of fatty alcohols and fatty aldehydes.
The response of trypanosomes and other eukaryotes to ER stress and the spliced leader RNA silencing (SLS) pathway in Trypanosoma brucei.
Michaeli, Ramat Gan, Israel. In Crit Rev Biochem Mol Biol, 2014
Here, I describe UPR in yeast and mammals, and compare it to what we know about pathogenic fungi and the parasitic protozoans from the order kinetoplastida, focusing on the novel pathway the spliced leader silencing (SLS) in Trypanosoma brucei.
Penetration of titanium dioxide nanoparticles through slightly damaged skin in vitro and in vivo.
Lu et al., Shanghai, China. In J Appl Biomater Function Mater, 2014
METHODS: TiO2-NPs with a diameter of 20 nm was labeled with 125I.The skin of rat was treated with 2% SLS solution and obtained as slightly damaged skin.
Increased cytotoxicity and streptolysin O activity in group G streptococcal strains causing invasive tissue infections.
Norrby-Teglund et al., Stockholm, Sweden. In Sci Rep, 2014
The opposite was true for Streptolysin S (SLS).
Endoplasmic reticulum stress responses in Leishmania.
Adak et al., Calcutta, India. In Mol Biochem Parasitol, 2014
Three different signaling pathways in the ER stress response have been described in trypanosomatids: these pathways involve (i) the down-regulation of translation by a protein kinase RNA-like ER kinase (PERK), (ii) the ER-associated degradation (ERAD) pathway, and (iii) the spliced leader silencing (SLS) pathway and its target mRNAs.
Induction of endoplasmic reticulum stress and unfolded protein response constitutes a pathogenic strategy of group A streptococcus.
Hanski et al., Jerusalem, Israel. In Front Cell Infect Microbiol, 2013
GAS acts extracellularly and during adherence to host cells it delivers the hemolysin toxins; streptolysin O (SLO) and streptolysin S (SLS).
Roles of gap junctions, connexins, and pannexins in epilepsy.
Carlen et al., Toronto, Canada. In Front Physiol, 2013
Specific Cx43 mimetic peptides, Gap 27 and SLS, inhibit the docking of astrocytic connexin Cx43 proteins from forming intercellular gap junctions (GJs), diminishing spontaneous seizures.
Streptolysin S and necrotising infections produced by group G streptococcus.
Nizet et al., San Diego, United States. In Lancet, 2002
The group G streptococcus chromosome contained a homologue of the nine-gene group A streptococcus sag operon encoding the beta-haemolysin streptolysin S (SLS).
Resolution of cutaneous inflammation after local elimination of macrophages.
van De Winkel et al., Utrecht, Netherlands. In Nat Biotechnol, 2000
We examined the activity of our immunotoxin in a sodium lauryl sulfate (SLS)-induced cutaneous inflammation model, using transgenic mice expressing human CD64.
Sjögren-Larsson syndrome is caused by mutations in the fatty aldehyde dehydrogenase gene.
Rizzo et al., Bethesda, United States. In Nat Genet, 1996
Sjögren-Larsson syndrome (SLS) is an inherited neurocutaneous disorder characterized by mental retardation, spasticity and ichthyosis.
The Sjögren-Larsson syndrome gene is close to D17S805 as determined by linkage analysis and allelic association.
Wadelius et al., Uppsala, Sweden. In Nat Genet, 1994
Sjögren-Larsson Syndrome (SLS) is characterized by congenital ichthyosis, spastic dior tetraplegia and mental retardation.
A gene for familial juvenile nephronophthisis (recessive medullary cystic kidney disease) maps to chromosome 2p.
Broyer et al., Paris, France. In Nat Genet, 1993
NPH is frequently (16% of cases) associated with Leber amaurosis (termed Senior-Løken syndrome, SLS).
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