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EDS5 enhanced disease susceptibility 5

sid1, eds5
Encodes an orphan multidrug and toxin extrusion transporter. Essential component of salicylic acid-dependent signaling for disease resistance. Member of the MATE-transporter family. Expression induced by salicylic acid. Mutants are salicylic acid-deficient. (from NCBI)
Top mentioned proteins: ACID, NPR1, V1a, CAN, prolactin receptor
Papers on sid1
Rutin-Mediated Priming of Plant Resistance to Three Bacterial Pathogens Initiating the Early SA Signal Pathway.
Chu et al., China. In Plos One, Dec 2015
We also demonstrated that the rutin-mediated priming resistance was attenuated in npr1, eds1, eds5, pad4-1, ndr1 mutants, and NahG transgenic Arabidopsis plant, while not in either snc1-11, ein2-5 or jar1 mutants.
RNAi-based inhibition of infectious myonecrosis virus replication in Pacific white shrimp Litopenaeus vannamei.
Marins et al., Rio Grande, Brazil. In Dis Aquat Organ, Jun 2015
Here, survival was tracked in L. vannamei injected with long synthetic dsRNAs targeted to IMNV open reading frame (ORF) 1a, ORF1b, and ORF2 genome regions prior to injection challenge with IMNV, and real-time RT-PCR was used to track the progress of IMNV infection and mRNA expression levels of the host genes sid1, dicer2, and argonaute2.
Opposite variations in fumarate and malate dominate metabolic phenotypes of Arabidopsis salicylate mutants with abnormal biomass under chilling.
Beale et al., Aberystwyth, United Kingdom. In Physiol Plant, 2014
In chilling conditions (5°C), salicylic acid (SA)-deficient mutants (sid2, eds5 and NahG) of Arabidopsis thaliana produced more biomass than wild type (Col-0), whereas the SA overproducer cpr1 was extremely stunted.
Localization and expression of EDS5H a homologue of the SA transporter EDS5.
Métraux et al., Fribourg, Switzerland. In Bmc Plant Biol, 2014
SA is produced in chloroplasts and the multidrug and toxin extrusion transporter ENHANCED DISEASE SUSCEPTIBILITY5 (EDS5; At4g39030) is necessary for the accumulation of SA after pathogen and abiotic stress.
RNAi silencing of the SoxE gene suppresses cell proliferation in silkworm BmN4 cells.
Lu et al., Chongqing, China. In Mol Biol Rep, 2014
Here, we used the silkworm ovary-derived BmN4-SID1 cell line to survey the roles of the silkworm SoxE protein (BmSoxE) and predict its candidate binding targets.
Atypical E2F transcriptional repressor DEL1 acts at the intersection of plant growth and immunity by controlling the hormone salicylic acid.
Wildermuth et al., Berkeley, United States. In Cell Host Microbe, 2014
Moreover, Enhanced Disease Susceptibility 5 (EDS5), a SA transporter required for elevated SA and immunity, is a direct repressed target of DEL1.
Biotrophy-specific downregulation of siderophore biosynthesis in Colletotrichum graminicola is required for modulation of immune responses of maize.
Deising et al., Halle, Germany. In Mol Microbiol, 2014
eGFP fusions with the key siderophore biosynthesis gene, SID1, encoding l-ornithine-N(5) -monooxygenase, suggested that siderophore biosynthesis is rigorously downregulated specifically during biotrophic development.
Establishment of Caenorhabditis elegans SID-1-dependent DNA delivery system in cultured silkworm cells.
Lee et al., Fukuoka, Japan. In Mol Biotechnol, 2014
The systemic RNA interference defective-1 (SID-1) can transport double-stranded RNA (dsRNA) into cytosol across the cytoplasmic membrane.
Establishing an in vivo assay system to identify components involved in environmental RNA interference in the western corn rootworm.
Tomoyasu et al., Chesterfield, United States. In Plos One, 2013
We also evaluated two WCR sid1- like (sil) genes with this assay system.
A large-scale genetic screen for mutants with altered salicylic acid accumulation in Arabidopsis.
Mou et al., Gainesville, United States. In Front Plant Sci, 2013
Complementation tests revealed that seven of the sln mutants are new alleles of eds5/sid1, two are sid2/eds16 alleles, one is allelic to pad4, and the remaining seven sln and two isn mutants are new non-allelic SA accumulation mutants.
The phosphate transporter PHT4;1 is a salicylic acid regulator likely controlled by the circadian clock protein CCA1.
Lu et al., Baltimore, United States. In Front Plant Sci, 2013
To further understand how PHT4;1 affects SA accumulation, here we analyzed the genetic interactions between the gain-of-function mutant pht4;1-1 and several known SA mutants, including sid2-1, ald1-1, eds5-3, and pad4-1.
Investigation of intercellular salicylic acid accumulation during compatible and incompatible Arabidopsis-pseudomonas syringae interactions using a fast neutron-generated mutant allele of EDS5 identified by genetic mapping and whole-genome sequencing.
Cameron et al., Hamilton, Canada. In Plos One, 2013
A whole-genome sequencing technique developed to identify fast neutron-induced deletion mutations revealed that iap1-1 is a new allele of EDS5 (eds5-5).
The variable hydroxamic acid siderophore metabolome of the marine actinomycete Salinispora tropica CNB-440.
Codd et al., Sydney, Australia. In Metallomics, 2013
Of these metabolites, bioinformatics analysis predicted that S. tropica CNB-440 could potentially biosynthesize, as high affinity Fe(iii) ligands, siderophores from the hydroxamic acid desferrioxamine class (sid1 gene cluster) and the phenolate-thia(oxa)zoli(di)ne class (sid2 and sid4 gene clusters).
Diverse responses are involved in the defence of Arabidopsis thaliana against Turnip crinkle virus.
Lin et al., Chengdu, China. In Z Naturforsch C, 2013
ABA can down-regulate the expression of the pathogenesis-related genes PR1 and PDF1.2, and compared to the wild type, it drastically reduces TCV accumulation in NahG transgenic plants and the eds5-p1 mutant, both of which do not accumulate SA.
Soaking RNAi in Bombyx mori BmN4-SID1 cells arrests cell cycle progression.
Kusakabe et al., Fukuoka, Japan. In J Insect Sci, 2012
Previously, the BmN4-SID1 cell expressing Caenorhabditis ele gans SID-1 was established, in which soaking RNAi could induce effective gene silencing.
Homo sapiens systemic RNA interference-defective-1 transmembrane family member 1 (SIDT1) protein mediates contact-dependent small RNA transfer and microRNA-21-driven chemoresistance.
Duxbury et al., Edinburgh, United Kingdom. In J Biol Chem, 2012
SIDT1, facilitates rapid, contact-dependent, bidirectional small RNA transfer between human cells, resulting in target-specific non-cell-autonomous RNAi
Preparation and characterization of the extracellular domain of human Sid-1.
MacRae et al., Los Angeles, United States. In Plos One, 2011
characterization of Sid-1 extracellular domain
Ascorbic acid deficiency in arabidopsis induces constitutive priming that is dependent on hydrogen peroxide, salicylic acid, and the NPR1 gene.
Barth et al., Morgantown, United States. In Mol Plant Microbe Interact, 2010
Data suggest hat vtc1-1 mutant requires functional PAD4, EDS5, and NPR1 for SA biosynthesis and pathogen resistance.
Overexpression of the Arabidopsis thaliana EDS5 gene enhances resistance to viruses.
Takahashi et al., Sendai, Japan. In Plant Biol (stuttg), 2008
Results suggest that, in addition to R gene-mediated gene-for-gene resistance, EDS5 is also important for basal resistance to viruses.
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