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GoPubMed Proteins lists recent and important papers and reviews for proteins. Page last changed on 19 Dec 2016.

SET binding factor 1

SBFI, Sbf1
Top mentioned proteins: ACID, V1a, ATPase, CAN, HAD
Papers on SBFI
Rare Earth Ion Mediated Fluorescence Accumulation on a Single Microbead: An Ultrasensitive Strategy for the Detection of Protein Kinase Activity at the Single-Cell Level.
Li et al., Xi'an, China. In Angew Chem Int Ed Engl, Jan 2016
A single microbead-based fluorescence imaging (SBFI) strategy that enables detection of protein kinase activity from single cell lysates is reported.
P2X7 receptor activation downmodulates Na(+)-dependent high-affinity GABA and glutamate transport into rat brain cortex synaptosomes.
Cordeiro et al., Porto, Portugal. In Neuroscience, Nov 2015
The cell-permeant sodium-selective fluorescent indicator, SBFI-AM, was used to estimate Na(+) influx across plasma membrane.
Breast cancer dissemination promoted by a neuregulin-collagenase 3 signalling node.
Pandiella et al., Salamanca, Spain. In Oncogene, Oct 2015
ERK1/2 increased collagenase 3 expression by controlling the activity of an SBF1-related transcription factor.
Na⁺ ions as spatial intracellular messengers for co-ordinating Ca²⁺ signals during pH heterogeneity in cardiomyocytes.
Vaughan-Jones et al., Salt Lake City, United States. In Cardiovasc Res, Mar 2015
Cytoplasmic [Na(+)] ([Na(+)]i) was imaged using SBFI.
Predose and Postdose Blood Gene Expression Profiles Identify the Individuals Susceptible to Acetaminophen-Induced Liver Injury in Rats.
Gao et al., Hangzhou, China. In Plos One, 2014
Among them, four genes, Incenp, Rpgrip1, Sbf1, and Mmp12, were found to be reproducibly in real-time PCR with an independent set of animals.
Recent advances in Charcot-Marie-Tooth disease.
Timmerman et al., Antwerp, Belgium. In Curr Opin Neurol, 2014
RECENT FINDINGS: Several new Charcot-Marie-Tooth disease-causing genes have been recently identified, further enlarging the genetic diversity and phenotypic variability, including: SBF1, DHTKD1, TFG, MARS, HARS, HINT1, TRIM1, AIFM1, PDK3 and GNB4.
Submicroscopic genomic rearrangements change gene expression in T-cell large granular lymphocyte leukemia.
Przybylski et al., Poznań, Poland. In Eur J Haematol, 2014
Two novel fusion transcripts were identified: CASP8-ERBB4 in MOTN-1 and SBF1-PKHD1L1 in T-LGL patient.
Non-preferential fuelling of the Na(+)/K(+)-ATPase pump.
Barros et al., Valdivia, Chile. In Biochem J, 2014
The activity of the Na(+) pump was assessed in parallel with the Na(+)-sensitive dye SBFI AM (Na(+)-binding benzofuran isophthalate acetoxymethyl ester).
Multi-photon intracellular sodium imaging combined with UV-mediated focal uncaging of glutamate in CA1 pyramidal neurons.
Rose et al., Düsseldorf, Germany. In J Vis Exp, 2013
Neurons were filled with the sodium-sensitive fluorescent dye SBFI through the patch-pipette, and multi-photon excitation of SBFI enabled the visualization of dendrites and adjacent spines.
DNA microarray reveals ZNF195 and SBF1 are potential biomarkers for gemcitabine sensitivity in head and neck squamous cell carcinoma cell lines.
Zheng et al., Shanghai, China. In Int J Clin Exp Pathol, 2013
The top seven positive genes responsible for gemcitabine sensitivity were validated by qPCR in these 10 HNSCC cell lines, while only two genes (SBF1 and ZNF195) were expression-correlated to gemcitabine response.
Regulation of myotubularin-related (MTMR)2 phosphatidylinositol phosphatase by MTMR5, a catalytically inactive phosphatase.
Dixon et al., Ann Arbor, United States. In Proc Natl Acad Sci U S A, 2003
MTMR2 interacts with MTMR5 via its coiled-coil domain and that mutations in the coiled-coil domain of either MTMR2 or MTMR5 abrogate this interaction.
Male infertility, impaired spermatogenesis, and azoospermia in mice deficient for the pseudophosphatase Sbf1.
Cleary et al., Stanford, United States. In J Clin Invest, 2002
expressed at high levels in seminiferous tubules of the testis; mice that are nullizygous for Sbf1 exhibit male infertility characterized by azoospermia.
Trithorax and dCBP acting in a complex to maintain expression of a homeotic gene.
Mazo et al., Philadelphia, United States. In Science, 2001
We have purified from Drosophila embryos a trithorax acetylation complex (TAC1) that contains Trx, dCBP, and Sbf1.
Pathogenesis and the role of Ca2+ overload during myocardial ischemia/reperfusion.
Hayashi, Nagoya, Japan. In Nagoya J Med Sci, 2000
To study the regulation of [Na+]i and [Ca2+]i during myocardial ischemia/reperfusion, [Na+]i and [Ca2+]i were measured simultaneously using guinea pig ventricular myocytes which were dual-loaded with SBFI/AM and fluo-3/AM.
MTM1 mutations in X-linked myotubular myopathy.
Mandel et al., Illkirch-Graffenstaden, France. In Hum Mutat, 1999
This gene encodes a dual-specificity phosphatase named myotubularin, defining a large gene family highly conserved through evolution (which includes the putative anti-phosphatase Sbf1/hMTMR5).
Characterization of the myotubularin dual specificity phosphatase gene family from yeast to human.
Mandel et al., Illkirch-Graffenstaden, France. In Hum Mol Genet, 1998
SBF1 (MTMR5) belongs to the myotubularin family of phosphoinositides phosphatases-like
Charcot-Marie-Tooth Neuropathy Type 4
Bird, Seattle, United States. In Unknown Journal, 1998
Detection of biallelic pathogenic variants in one of the following 11 genes establishes the diagnosis: GDAP1 (CMT4A), MTMR2 (CMT4B1), SBF2 (CMT4B2), SBF1 (CMT4B3), SH3TC2 (CMT4C), NDRG1 (CMT4D), EGR2 (CMT4E), PRX (CMT4F), HK1 (CMT4G), FGD4 (CMT4H), and FIG4 (CMT4J).
Association of SET domain and myotubularin-related proteins modulates growth control.
Cleary et al., Stanford, United States. In Nat Genet, 1998
These include myotubularin, the gene of which is mutated in a subset of patients with X-linked myotubular myopathy, and Sbf1, a newly isolated homologue of myotubularin.
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