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GoPubMed Proteins lists recent and important papers and reviews for proteins. Page last changed on 19 Dec 2016.

Zinc finger protein 42 homolog

Rex-1, Zfp42
Top mentioned proteins: Nanog, Sox2, CAN, OCT, TRAF2
Papers on Rex-1
A repeat sequence domain of the ring-exported protein-1 of Plasmodium falciparum controls export machinery architecture and virulence protein trafficking.
Tilley et al., Melbourne, Australia. In Mol Microbiol, Dec 2015
The Ring-EXported Protein-1 (REX1) is a Maurer's cleft resident protein.
WNT/β-catenin signaling affects cell lineage and pluripotency-specific gene expression in bovine blastocysts: prospects for bovine embryonic stem cell derivation.
Perkowska et al., Poznań, Poland. In Stem Cells Dev, Nov 2015
WNT activity clearly exerted a positive effect on pluripotency gene expression in developing bovine embryos, manifested by upregulation of OCT4, NANOG, REX1, SOX2, c-MYC, and KLF4 in the ICM and downregulation of CDX2 in the TE.
Putative embryonic stem cells derived from porcine cloned blastocysts using induced pluripotent stem cells as donors.
Hyun et al., Ch'ŏngju, South Korea. In Theriogenology, Nov 2015
Based on quantitative polymerase chain reaction, the expression levels of REX1 and FGFR2 in iPS-NT lines were higher than those of cells of other origins.
Expression profile of FGF receptors in preimplantation ovine embryos and the effect of FGF2 and PD173074.
Nasr-Esfahani et al., Eşfahān, Iran. In Growth Factors, Oct 2015
Assessment of expression profiles of lineage-associated markers revealed that FGF2 (500 ng/ml) supplementation: (i) significantly increased expression of putative hypoblast marker (GATA4), (ii) significantly decreased expression of putative epiblast (EPI) marker (NANOG) and (iii) did not change TE markers (CDX2 and IFNT) and pluripotency makers (OCT4, SOX2 and REX1).
Molecular and phenotypic characterization of CD133 and SSEA4 enriched very small embryonic-like stem cells in human cord blood.
Bhartiya et al., Mumbai, India. In Leukemia, Sep 2015
These enriched cells were small in size (4-6 μm), spherical, exhibited telomerase activity and expressed pluripotent stem cell (OCT4A, OCT4, SSEA4, NANOG, SOX2, REX1), primordial germ cell (STELLA, FRAGILIS) as well as primitive hematopoietic (CD133, CD34) markers at protein and transcript levels.
Development, Characterization, and Pluripotency Analysis of Buffalo (Bubalus bubalis) Embryonic Stem Cell Lines Derived from In Vitro-Fertilized, Hand-Guided Cloned, and Parthenogenetic Embryos.
Chauhan et al., Karnāl, India. In Cell Reprogram, Aug 2015
All the cell lines expressed stem cell markers, such as Alkaline Phosphatase, OCT4, NANOG, SSEA1, SSEA4, TRA-1-60, TRA-1-81, SOX2, REX1, CD-90, STAT3, and TELOMERASE.
Cellular stage specific functional analysis of REX1: In human embryonic stem cells.
Lee et al., Baltimore, United States. In Proteomics, Jul 2015
The new proteome analysis by Son et al. [Proteomics 2015, 15, 2220-2229] identified the functions of the pluripotency marker protein, REX1 in hESCs, and unraveling its regulatory network orchestrating pluripotency.
Markers of Pluripotency in Human Amniotic Epithelial Cells and Their Differentiation to Progenitor of Cortical Neurons.
Díaz et al., Ciudad López Mateos, Mexico. In Plos One, 2014
We analyzed qualitatively and quantitatively the expression of the transcription factors of pluripotency (OCT4, SOX2, NANOG, KLF4 and REX1) by RT-PCR and RT-qPCR in hAEC.
Generation of Naïve Bovine Induced Pluripotent Stem Cells Using PiggyBac Transposition of Doxycycline-Inducible Transcription Factors.
Imai et al., Kyoto, Japan. In Plos One, 2014
Both types of biPSCs had strong alkaline phosphatase activity, expressed pluripotent markers (OCT3/4, NANOG, REX1, ESRRβ, STELLA, and SOCS3), and formed embryoid bodies that gave rise to differentiated cells from all three embryonic germ layers.
Regulation of Mouse Retroelement MuERV-L/MERVL Expression by REX1 and Epigenetic Control of Stem Cell Potency.
Muniesa et al., Zaragoza, Spain. In Front Oncol, 2013
This review will focus on the role of MERVL-derived sequences as controlling elements of gene expression specific for pre-implantation development, two-cell stage-specific gene expression, and stem cell pluripotency, the epigenetic mechanisms that control their expression, and the contributions of the pluripotency marker REX1 and the related Yin Yang 1 family of transcription factors to this regulation process.
Role of tumor suppressor genes in the cancer-associated reprogramming of human induced pluripotent stem cells.
Saito et al., In Stem Cell Res Ther, 2013
They express embryonic stem cell markers, such as OCT4, SOX2, NANOG, SSEA-3, SSEA-4, and REX1, and can differentiate into all adult tissue types, both in vitro and in vivo.
RNF12 initiates X-chromosome inactivation by targeting REX1 for degradation.
Gribnau et al., Rotterdam, Netherlands. In Nature, 2012
RNF12 causes REX1 breakdown through dose-dependent catalysis, thereby representing an important pathway to initiate X-chromosome inactivation
Embryonic stem cell factors undifferentiated transcription factor-1 (UFT-1) and reduced expression protein-1 (REX-1) are widely expressed in human skin and may be involved in cutaneous differentiation but not in stem cell fate determination.
Pammer et al., Vienna, Austria. In Int J Exp Pathol, 2011
reduced expression protein-1 (REX-1) is widely expressed in human skin and may be involved in cutaneous differentiation but not in stem cell fate determination.
Rex1 (Zfp42) null mice show impaired testicular function, abnormal testis morphology, and aberrant gene expression.
Gudas et al., New York City, United States. In Dev Biol, 2011
loss of Rex1 leads to impaired testicular function.
Association of Rex-1 to target genes supports its interaction with Polycomb function.
Schoorlemmer et al., Zaragoza, Spain. In Stem Cell Res, 2011
the involvement of Rex-1 in control of Polycomb target genes during pluripotency or differentiation.
Rex1/Zfp42 as an epigenetic regulator for genomic imprinting.
Kim et al., Baton Rouge, United States. In Hum Mol Genet, 2011
the functional connection of Rex1 to genomic imprinting represents another case where newly made genes have co-evolved with lineage-specific phenomena.
The coupling of X-chromosome inactivation to pluripotency.
Avner et al., Paris, France. In Annu Rev Cell Dev Biol, 2010
In murine embryonic stem cells, both genes are under the transcriptional control of a series of critical pluripotency factors, namely, OCT3/4, NANOG, SOX2, KLF4, C-MYC and REX1.
Live cell imaging distinguishes bona fide human iPS cells from partially reprogrammed cells.
Schlaeger et al., Boston, United States. In Nat Biotechnol, 2009
Proviral silencing and expression of TRA-1-60, DNMT3B and REX1 can be used to distinguish the fully reprogrammed state, whereas alkaline phosphatase, SSEA-4, GDF3, hTERT and NANOG are insufficient as markers.
Chlamydomonas reinhardtii: a convenient model system for the study of DNA repair in photoautotrophic eukaryotes.
Miadoková et al., Bratislava, Slovakia. In Curr Genet, 2008
Among others, the involvement of UVSE1 gene in recombinational repair and uniparental inheritance of chloroplast genome, the specific role of TRXH1 gene in strand break repair, the requirement of PHR1 gene for full activity of PHR2 gene, or encoding of two excision repair proteins by the single REX1 gene.
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