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GoPubMed Proteins lists recent and important papers and reviews for proteins. Page last changed on 19 Dec 2016.

Primase, DNA, polypeptide 2

may play a role in DNA replication; V288M mutation is detected in a hepatoma cell line [RGD, Feb 2006] (from NCBI)
Top mentioned proteins: CAN, ACID, POLYMERASE, HAD, p70
Papers on p58
Y-Box Binding Protein 1 Stabilizes Hepatitis C Virus NS5A via Phosphorylation-Mediated Interaction with NS5A To Regulate Viral Propagation.
Wu Lee et al., Taipei, Taiwan. In J Virol, Dec 2015
Moreover, knockdown of YB-1 in HCV-infected cells prevented infectious virus production and reduced the ratio of hyperphosphorylated (p58) to hypophosphorylated (p56) forms of NS5A, whereas DDX3 silencing did not affect the ratio of the p58 and p56 phosphoforms of NS5A.
Application of multiplexing technology to the analysis of the intrathecally released immunoglobulins against B. burgdorferi antigens in neuroborreliosis.
Lewczuk et al., Białystok, Poland. In Immunol Lett, Nov 2015
IgG-ASI against either VlsE, p100, p58, p39, p18, or OspC, and none of these patients showed positive OspA-IgG-ASI.
Differential expression of endoplasmic reticulum stress-response proteins in different renal tubule subtypes of OVE26 diabetic mice.
Klein et al., Louisville, United States. In Cell Stress Chaperones, Nov 2015
Expression of ER stress-induced PERK inhibitor p58(IPK) was decreased and then increased in all tubule subtypes of young and older mice, respectively.
Erp29 Attenuates Cigarette Smoke Extract-Induced Endoplasmic Reticulum Stress and Mitigates Tight Junction Damage in Retinal Pigment Epithelial Cells.
Zhang et al., Oklahoma City, United States. In Invest Ophthalmol Vis Sci, Oct 2015
Overexpression of ERp29 increased the levels of GRP78, p58(IPK), and Nrf-2, while reducing p-eIF2α and C/EBP homologous protein (CHOP), and protected RPE cells from CSE-induced apoptosis.
Critical role of CDK11(p58) in human breast cancer growth and angiogenesis.
Wu et al., Shanghai, China. In Bmc Cancer, 2014
CDK11(p58), a Ser/Thr kinase that belongs to the Cell Division Cycle 2-like 1 (CDC2L1) subfamily is associated with cell cycle progression, tumorigenesis, sister chromatid cohesion and apoptotic signaling.
Activation of calcium/calmodulin-dependent protein kinase II in obesity mediates suppression of hepatic insulin signaling.
Tabas et al., New York City, United States. In Cell Metab, 2014
The mechanism of improvement begins with induction of ATF6 and the ATF6 target p58(IPK), a chaperone that suppresses the PERK-p-eIF2α-ATF4 branch of the UPR.
14-3-3sigma controls mitotic translation to facilitate cytokinesis.
Yaffe et al., Cambridge, United States. In Nature, 2007
This defective switch in the mechanism of translation results in reduced mitotic-specific expression of the endogenous internal ribosomal entry site (IRES)-dependent form of the cyclin-dependent kinase Cdk11 (p58 PITSLRE), leading to impaired cytokinesis, loss of Polo-like kinase-1 at the midbody, and the accumulation of binucleate cells.
Epidemiology and diagnosis of Lyme borreliosis.
Wilske, München, Germany. In Ann Med, 2004
The sensitivity and standardization of immunoblots has been considerably enhanced by use of recombinant antigens (p100, p58, p41i, VlsE, OspC, DbpA) including those expressed primarily in vivo (VlsE and DbpA) instead of whole cell lysates.
ER-to-Golgi transport and cytoskeletal interactions in animal cells.
Presley et al., Montréal, Canada. In Cell Mol Life Sci, 2004
Many proteins (including vSNAREs, ERGIC53/p58 and the KDEL receptor) must cycle back to the ER from pre-Golgi intermediates or the Golgi.
Microbiological diagnosis in Lyme borreliosis.
Wilske, München, Germany. In Int J Med Microbiol, 2002
Progress has been made in the sensitivity of the recombinant blot using additional antigens as p58 and homologues of Osp17.
HLA-class I-specific inhibitory receptors in HIV-1 infection.
Moretta et al., Genova, Italy. In Hum Immunol, 2000
In HIV-1 infected patients, relevant proportions of peripheral blood CD8+ T lymphocytes express iNKRs belonging to the Ig superfamily (p58/p70/p140) and CD94/NKG2A.
The non-structural 5A protein of hepatitis C virus.
Germanidis et al., Créteil, France. In J Viral Hepat, 1999
The released, mature NS5A protein is a 56 000 MW phosphoprotein (p56), which also exists within infected cells in a hyperphosphorylated form (p58).
Regulating the yeast kinetochore by ubiquitin-dependent degradation and Skp1p-mediated phosphorylation.
Sorger et al., Cambridge, United States. In Cell, 1997
Thus, p58 appears to be activated by phosphorylation in a p23Skp1-dependent step and degraded by the proteosome in a ubiquitin-dependent step.
Structure of the inhibitory receptor for human natural killer cells resembles haematopoietic receptors.
Wiley et al., Cambridge, United States. In Nature, 1997
In the human killer-cell inhibitory receptor family, individual p58 members are specific for a subset of class I human leukocyte antigen (HLA)-C molecules.
Recruitment of tyrosine phosphatase HCP by the killer cell inhibitor receptor.
Long et al., Rockville, United States. In Immunity, 1996
Human NK cells express a p58 inhibitory receptor specific for HLA-C.
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