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GoPubMed Proteins lists recent and important papers and reviews for proteins. Page last changed on 19 Dec 2016.

HLA complex P5

Orf, HCP5, protein pVI
Top mentioned proteins: CAN, ACID, POLYMERASE, HAD, vascular endothelial growth factor
Papers on Orf
Orf virus IL-10 reduces monocyte, dendritic cell and mast cell recruitment to inflamed skin.
Wise et al., Dunedin, New Zealand. In Virus Res, Jan 2016
UNASSIGNED: Orf virus (ORFV) is a zoonotic parapoxvirus that causes pustular dermatitis of sheep, and occasionally humans.
A Genome Wide Study of Copy Number Variation Associated with Nasopharyngeal Carcinoma in Malaysian Chinese Identifies CNVs at 11q14.3 and 6p21.3 as Candidate Loci.
Ng et al., Kuala Lumpur, Malaysia. In Plos One, Dec 2015
RESULTS: Six putative CNVRs overlapping GRM5, MICA/HCP5/HCG26, LILRB3/LILRA6, DPY19L2, RNase3/RNase2 and GOLPH3 genes were jointly identified by PennCNV and cnvPartition.
MiRNA-891a-5p mediates HIV-1 Tat and KSHV Orf-K1 synergistic induction of angiogenesis by activating NF-κB signaling.
Lu et al., Nanjing, China. In Nucleic Acids Res, Nov 2015
Here, we explored the synergistic effect of HIV-1 Tat and KSHV oncogene Orf-K1 on angiogenesis.
Effect of ORF119 gene deletion on the replication and virulence of orf virus.
Chen et al., In Acta Virol, Sep 2015
Orf is a severe infectious disease of sheep and goats caused by orf virus (ORFV).
Phylogenetic analysis of B2L gene of Egyptian orf virus from naturally infected sheep.
Shiha et al., Al Manşūrah, Egypt. In Virusdisease, Sep 2015
Orf is a viral disease caused by a parapoxvirus, affecting primarily sheep and goats and causes severe economic losses.
Biochemistry and regulatory functions of bacterial glucose kinases.
Sánchez et al., Mexico. In Arch Biochem Biophys, Jul 2015
As reported for various microorganisms, in addition to their catalytic activity, some Glks, possessing ROK (Repressor Orf Kinases) motifs, also display a regulatory role.
Isolation and molecular characterization of Orf virus from natural outbreaks in goats of Assam.
Tamuly et al., Guwāhāti, India. In Virusdisease, Jun 2015
The major envelop protein (B2L) of Orf virus was targeted for molecular analysis.
Histopathological study and F1L gene sequence analysis of contagious ecthyma in small ruminants of Shiraz suburb, Iran.
Mohammadi et al., Shīrāz, Iran. In Trop Biomed, Jun 2015
Contagious ecthyma, also known as Orf, is a common viral skin disease of sheep and goats caused by a Parapoxvirus.
Molecular genetic analysis of orf virus: a poxvirus that has adapted to skin.
Mercer et al., Dunedin, New Zealand. In Viruses, Mar 2015
Orf virus is the type species of the Parapoxvirus genus of the family Poxviridae.
Genome analysis of orf virus isolates from goats in the Fujian Province of southern China.
Wang et al., Fuzhou, China. In Front Microbiol, 2014
Orf virus (ORFV), a species of the genus Parapoxvirus of the family Poxviridae, causes non-systemic, highly contagious, and eruptive disease in sheep, goat, and other wild and domestic ruminants.
[Molecular mechanism of ORFV intervention strategies based on the UPS of host cell: a review].
Cui et al., Daqing, China. In Bing Du Xue Bao, 2013
In order to compete the antiviral effects of the host cell in the process of infection, ORFV(known as Orf virus) relies on a series of functional genes developed through long-term population evolution, such as interferon resistance genes, Bcl-2 protein genes and cell cycle inhibitor gene and so on, with these weapons this virus is able to effectively counteract immune clearance and immune regulation from a host cell.
An Investigation of a Cluster of Parapoxvirus Cases in Missouri, Feb-May 2006: Epidemiologic, Clinical and Molecular Aspects.
Damon et al., Atlanta, United States. In Animals (basel), 2012
Forty-six percent of veterinarians reported having been consulted regarding suspected human orf infections.
Rising HIV-1 viral load set point at a population level coincides with a fading impact of host genetic factors on HIV-1 control.
Dutch HIV monitoring foundation HIV-1 Host Genetics study et al., Amsterdam, Netherlands. In Aids, 2011
assessed whether increases in viral load set points are associated with genetic markers in CCR5, HCP5, and HLA-C genes
Association of the genetic marker for abacavir hypersensitivity HLA-B*5701 with HCP5 rs2395029 in Mexican Mestizos.
Carnevale et al., Mexico. In Pharmacogenomics, 2011
Haplotype analysis revealed that HLA-B*5701 and the HCP5 rs2395029(G) allele are in complete LD (r(2) = 1) in this Mexican Mestizos sample.
Orf-I and orf-II-encoded proteins in HTLV-1 infection and persistence.
Franchini et al., Bethesda, United States. In Viruses, 2011
The 3' end of the human T-cell leukemia/lymphoma virus type-1 (HTLV-1) genome contains four overlapping open reading frames (ORF) that encode regulatory proteins.
A polymorphism in the HCP5 gene associated with HLA-B*5701 does not restrict HIV-1 in vitro.
NIAID Center for HIV/AIDS Vaccine Immunology et al., Durham, United States. In Aids, 2010
No significant HIV-1 restriction was observed in the cells throughout HIV-1 life cycle, suggesting that the association of HCP5 variant with viral control is likely due to HLA-B*5701-related effect or other functional variants in the haplotype or both
HLA status in patients with chronic spontaneous urticaria.
Jarzab et al., Zabrze, Poland. In Int Arch Allergy Immunol, 2009
HLA-B44 allele is prevalent in Polish patients with chronic spontaneous urticaria
HIV-1 disease-influencing effects associated with ZNRD1, HCP5 and HLA-C alleles are attributable mainly to either HLA-A10 or HLA-B*57 alleles.
Ahuja et al., San Antonio, United States. In Plos One, 2007
Data demonstrate that the influence of ZNRD1 alleles on disease progression rates are attributable to HLA-A10, help clarify the relationship between the HCP5, HLA-C and HLA-B*57 alleles, and reaffirm a critical role of HLA-B*57 alleles in HIV disease.
Differential roles of vascular endothelial growth factor receptor-1 and receptor-2 in angiogenesis.
Shibuya, Tokyo, Japan. In J Biochem Mol Biol, 2006
Recently, the VEGFR2-specific ligand VEGF-E (Orf-VEGF) was extensively characterized.
The adenovirus protease is activated by a virus-coded disulphide-linked peptide.
Kemp et al., Saint Andrews, United States. In Cell, 1993
Activity was reconstituted by a component of adenovirus virions, which was identified as GVQSLKRRRCF, a peptide derived from the virus protein pVI.
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