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Oligodendrocyte transcription factor 3

Olig3, Oligo-3
Top mentioned proteins: A20, PTPN22, TNF Receptor-Associated Factor 1, STAT4, DRB1
Papers on Olig3
Multiple sclerosis risk genotypes correlate with an elevated cerebrospinal fluid level of the suggested prognostic marker CXCL13.
Olsson et al., Stockholm, Sweden. In Mult Scler, 2013
RESULTS: Presence of the HLA-DRB1*15 and the MS risk genotypes for SNPs in the RGS1, IRF5 and OLIG3/TNFAIP3 gene regions correlated significantly with increased levels of CXCL13.
Structural characterization of biocompatible lipoic acid-oligo-(3-hydroxybutyrate) conjugates by electrospray ionization mass spectrometry.
Adamus et al., Zabrze, Poland. In Rapid Commun Mass Spectrom, 2013
Biodegradable and biocompatible conjugates of oligo-3-hydroxybutyrate with lipoic acid were obtained via the anionic ring-opening oligomerization of (R,S)-β-butyrolactone initiated by lipoic acid potassium salt.
Electrospray ionisation mass spectrometry molecular-level structural characterisation of novel phenoxycarboxylic acid-oligo(3-hydroxybutyrate) conjugates with potential agricultural applications.
Kowalczuk et al., Zabrze, Poland. In Rapid Commun Mass Spectrom, 2013
In the present work, we report the synthesis and structural studies of novel controlled-release pesticide-oligo-3-hydroxybutyrate systems with potential agricultural applications.
Comparison of methodologies for analysing the progression of joint destruction in rheumatoid arthritis.
van der Helm-van Mil et al., Leiden, Netherlands. In Scand J Rheumatol, 2012
As genetic variants are known to have small effect sizes, two genetic variants were studied as examples: rs675520 (located in the TNFAIP3-OLIG3 region) and the presence of the human leucocyte antigen (HLA) shared epitope (SE) alleles.
A genetic marker at the OLIG3/TNFAIP3 locus associates with methotrexate continuation in early inflammatory polyarthritis: results from the Norfolk Arthritis Register.
Thomson et al., Manchester, United Kingdom. In Pharmacogenomics J, 2012
SNP within the OLIG3/TNFAIP3 locus (rs6920220) was associated with being less likely to maintain MTX monotherapy
A comprehensive analysis of shared loci between systemic lupus erythematosus (SLE) and sixteen autoimmune diseases reveals limited genetic overlap.
International Consortium on the Genetics of Systemic Erythematosus et al., Charleston, United States. In Plos Genet, 2011
We observed that the loci shared among the most ADs include IL23R, OLIG3/TNFAIP3, and IL2RA.
Improving the estimation of celiac disease sibling risk by non-HLA genes.
Sacchetti et al., Napoli, Italy. In Plos One, 2010
All individuals were typed for HLA and 10 SNPs in non-HLA genes: CCR1/CCR3 (rs6441961), IL12A/SCHIP1 and IL12A (rs17810546 and rs9811792), TAGAP (rs1738074), RGS1 (rs2816316), LPP (rs1464510), OLIG3 (rs2327832), REL (rs842647), IL2/IL21 (rs6822844), SH2B3 (rs3184504).
Genetic variants in the prediction of rheumatoid arthritis.
Huizinga et al., Leiden, Netherlands. In Ann Rheum Dis, 2010
The following genetic variants were assessed HLA-DRB1 shared epitope (SE) alleles, rs2476601 (PTPN22), rs108184088 (TRAF1-C5), rs7574865 (STAT4), rs3087243 (CTLA4), rs4810485 (CD40), rs1678542 (KIF5A-PIP4K2C), rs2812378 (CCL21), rs42041 (CDK6), rs4750316 (PRKCQ), rs6684865 (MMEL1-TNFRSF14), rs2004640 (IRF5), rs6920220 and rs10499194 (TNFAIP3-OLIG3), interactions between HLA-SE alleles and rs2476601 (PTPN22) and between HLA-SE alleles and smoking.
Genetics in neuroendocrine immunology: implications for rheumatoid arthritis and osteoarthritis.
Rovenský et al., Regensburg, Germany. In Ann N Y Acad Sci, 2010
Recent genome-wide association studies (GWAS) on RA identified known and novel susceptibility genes like HLA-DRB1, PTPN22, STAT4, TRAF1/C5, OLIG3/TNFAIP3, CD40, CCL21, MMEL1-TNFRSF14, CDK6, PRKCQ, IL2RB, and KIF5A-PIP4K2C.
Pharmacogenetics of rheumatoid arthritis: Potential targets from susceptibility genes and present therapies.
Rahman et al., St. John's, Canada. In Pharmgenomics Pers Med, 2009
Outside the major histocompatibility complex (MHC) region, six additional risk loci have been identified and validated including PTPN22, STAT4, PADI4, CTLA4, TNFAIP3-OLIG3, and TRAF1/C5.
Unraveling the genetics of complex diseases: susceptibility genes for rheumatoid arthritis and psoriasis.
Begovich et al., Alameda, United States. In Semin Immunol, 2009
Recently the results of multiple, well powered, genetic case-control studies have begun to appear providing convincing statistical evidence for at least ten non-HLA related risk genes or loci (C5/TRAF1, CD40, CTLA4, KIF5A/PIP4K2C, MMEL1/TNFRSF14, PADI4, PRKCQ, PTPN22, STAT4, and TNFAIP3/OLIG3) for RA and six (IL12B, IL13, IL23R, STAT2/IL23A, TNFAIP3, and TNIP1) for psoriasis.
[Influence of genetic factors on development and severity of rheumatoid arthritis--part I].
Łacki et al., Warsaw, Poland. In Pol Merkur Lekarski, 2009
PTPN22 gene polymorphism, C5/TRAF1 genes region polymorphism and TNFAIP3-OLIG3 genes region polymorphism(s) are among newly identified and already confirmed genetic risk factors, whereas STAT 4, CTLA4, PADI4 and IRF5 genes polymorphisms are listed among probable RA development genetic risk factors.
The bHLH transcription factor Olig3 marks the dorsal neuroepithelium of the hindbrain and is essential for the development of brainstem nuclei.
Birchmeier et al., Berlin, Germany. In Development, 2009
The fate of class A neurons was not correctly determined in Olig3 mutants. As a consequence, the nucleus of the solitary tract did not form, and precerebellar nuclei, such as the inferior olivary nucleus, were absent or small.
Control of precerebellar neuron development by Olig3 bHLH transcription factor.
Qiu et al., Louisville, United States. In J Neurosci, 2008
Olig3 is specifically expressed in dorsal progenitor 1 domains of neural progenitor cells in the caudal hindbrain and regulates the fates of these neural progenitor cells.
Two independent alleles at 6q23 associated with risk of rheumatoid arthritis.
Altshuler et al., Cambridge, United States. In Nat Genet, 2007
After evaluating and adjusting for technical and population biases, we identified a SNP at 6q23 (rs10499194, approximately 150 kb from TNFAIP3 and OLIG3) that was reproducibly associated with rheumatoid arthritis both in the genome-wide association (GWA) scan and in 5,541 additional case-control samples (P = 10(-3), GWA scan; P < 10(-6), replication; P = 10(-9), combined).
Rheumatoid arthritis association at 6q23.
Worthington et al., Manchester, United Kingdom. In Nat Genet, 2007
This SNP maps to 6q23, between the genes oligodendrocyte lineage transcription factor 3 (OLIG3) and tumor necrosis factor-alpha-induced protein 3 (TNFAIP3).
Bmp and Wnt/beta-catenin signals control expression of the transcription factor Olig3 and the specification of spinal cord neurons.
Birchmeier et al., Berlin, Germany. In Dev Biol, 2007
Wnt/beta-catenin signals act upstream of Olig3 in the specification of dI2 and dI3 neurons.
Short-term lineage analysis of dorsally derived Olig3 cells in the developing spinal cord.
Ono et al., Okazaki, Japan. In Dev Dyn, 2005
dorsal Olig3 cells contribute to dorsal midline cells and commissural interneurons at intermediate and ventral levels
Potent antibacterial activity of oligo-3-aminopyridine against Staphylococcus aureus and Enterococcus faecalis.
Kaya et al., Çanakkale, Turkey. In Indian J Biochem Biophys, 2004
Oligo-3-aminopyridine (OAP) was tested for its antibacterial activity against two gram positive (Staphylococcus aureus and Enterococcus faecalis) and two gram negative (Escherchia coli and Pseudomonas aeruginosa) bacterial strains.
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