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GoPubMed Proteins lists recent and important papers and reviews for proteins. Page last changed on 19 Dec 2016.

Odd-skipped related 1

Top mentioned proteins: HIF-1alpha, CAN, AGE, HAD, VHL
Papers using ODD antibodies
Non-invasive detection of a small number of bioluminescent cancer cells in vivo.
Agoulnik Irina, In PLoS ONE, 2007
... ODD-luciferase transgenic mice were bred with ...
Papers on ODD
An oxygen-insensitive Hif-3α isoform inhibits Wnt signaling by destabilizing the nuclear β-catenin complex.
Duan et al., Ann Arbor, United States. In Elife, Feb 2016
Hif-3α2 lacks the bHLH, PAS, PAC, and ODD domains and is expressed in embryonic and adult tissues independently of oxygen availability.
A multidomain cascade model of early childhood risk factors associated with oppositional defiant disorder symptoms in a community sample of 6-year-olds.
Bryant et al., Chicago, United States. In Dev Psychopathol, Jan 2016
UNASSIGNED: The present study examined a cascade model of age 4 and 5 contextual, parent, parenting, and child factors on symptoms of oppositional defiant disorder (ODD) at age 6 in a diverse community sample of 796 children.
Combining Optical Reporter Proteins with Different Half-lives to Detect Temporal Evolution of Hypoxia and Reoxygenation in Tumors.
Bhujwalla et al., Baltimore, United States. In Neoplasia, Dec 2015
For this purpose, human prostate cancer PC3 cells were transfected with the luciferase gene fused with an oxygen-dependent degradation domain (ODD-luc) and a variant of the enhanced green fluorescent protein (EGFP).
Correlates and Consequences of Suicidal Cognitions and Behaviors in Children Ages 3 to 7 Years.
Luby et al., Amherst Center, United States. In J Am Acad Child Adolesc Psychiatry, Nov 2015
RESULTS: Controlling for a range of clinical and demographic variables, early-childhood SI (as defined as suicidal thoughts, behavior, or any expression of plans/attempts occurring before age 7) and suicidal themes in play were concurrently associated with childhood attention-deficit/hyperactivity (ADHD) and oppositional defiant/conduct disorders (ODD/CD).
Oxidative DNA damage enhances the carcinogenic potential of in vitro chronic arsenic exposures.
Hernández et al., Barcelona, Spain. In Arch Toxicol, Nov 2015
Our previous work demonstrated that environmentally relevant arsenic exposures generate an accelerated accumulation of pre-carcinogen 8-OH-dG DNA lesions under Ogg1-deficient backgrounds, but it remains unproved whether this observed arsenic-induced oxidative DNA damage (ODD) is certainly important in terms of cancer.
Pharmacological Management of Pediatric Patients with Comorbid Attention-Deficit Hyperactivity Disorder Oppositional Defiant Disorder.
Connor, Farmington, United States. In Paediatr Drugs, Oct 2015
Oppositional defiant disorder (ODD) is an externalizing behavior disorder characterized by difficulties with emotional and behavioral regulation that frequently brings the child into conflict with authority figures.
Gene-set and multivariate genome-wide association analysis of oppositional defiant behavior subtypes in attention-deficit/hyperactivity disorder.
van Hulzen et al., Zürich, Switzerland. In Am J Med Genet B Neuropsychiatr Genet, Aug 2015
UNASSIGNED: Oppositional defiant disorder (ODD) is a frequent psychiatric disorder seen in children and adolescents with attention-deficit-hyperactivity disorder (ADHD).
Hypoxia fate mapping identifies cycling cardiomyocytes in the adult heart.
Sadek et al., Dallas, United States. In Nature, Aug 2015
Here we report fate mapping of hypoxic cells and their progenies by generating a transgenic mouse expressing a chimaeric protein in which the oxygen-dependent degradation (ODD) domain of Hif-1α is fused to the tamoxifen-inducible CreERT2 recombinase.
Heterologous expression and transcript analysis of gibberellin biosynthetic genes of grasses reveals novel functionality in the GA3ox family.
Phillips et al., Davis, United States. In Bmc Plant Biol, 2014
Some additional paralogs of 2-ODD genes were identified: notably, a novel gene in the wheat B genome related to GA3ox2 was shown to encode a GA 1-oxidase, named as TaGA1ox-B1.
Neuropsychological Profiles Correlated with Clinical and Behavioral Impairments in a Sample of Brazilian Children with Attention-Deficit Hyperactivity Disorder.
Bueno et al., São Paulo, Brazil. In Front Psychiatry, 2014
Attention-deficit hyperactivity disorder (ADHD) is a complex neurodevelopmental disorder that implies several-step process, and there is no single test to diagnose both ADHD and associated comorbidities, such as oppositional-defiant disorder (ODD), anxiety disorder, depression, and certain types of learning disabilities.
Hypoxia as a target for tissue specific gene therapy.
Lee et al., Seoul, South Korea. In J Control Release, 2014
In addition to transcriptional and translational regulations, post-translational regulation strategies have been developed using the HIF-1α ODD domain.
The function and catalysis of 2-oxoglutarate-dependent oxygenases involved in plant flavonoid biosynthesis.
Lou et al., Jinan, China. In Int J Mol Sci, 2013
During the synthesis of the tricyclic flavonoid natural products in plants, oxidative modifications to the central C ring are catalyzed by four of FeII and 2-oxoglutarate dependent (2-ODD) oxygenases, namely flavone synthase I (FNS I), flavonol synthase (FLS), anthocyanidin synthase (ANS) and flavanone 3β-hydroxylase (FHT).
[Development of incurable cancer cell targeted agents using viral technology].
Takahashi et al., Ōsaka, Japan. In Yakugaku Zasshi, 2012
Since tumor cells present in a hypoxic environment are known to be resistant to chemotherapy and radiotherapy, we also engineered a novel oncolytic herpes virus targeting a specific tumor microenvironment, which harbors a gene encoding a fusion protein of oxygen-dependent degradation (ODD) domain of HIF1α and ICP4, a master viral transcription factor required for replication.
A variant OSR1 allele which disturbs OSR1 mRNA expression in renal progenitor cells is associated with reduction of newborn kidney size and function.
Goodyer et al., Montréal, Canada. In Hum Mol Genet, 2011
OSR1 is expressed in human mesenchymal stem cells, the blastemal component of Wilms tumors and CD24+/CD133+ progenitor cells isolated from the mature kidney.
Selective, reversible inhibitors of the AAA ATPase p97
Deshaies et al., Bethesda, United States. In Unknown Journal, 2011
By contrast, these probe compounds were far less potent in blocking degradation of the p97-independent substrate oxygen-dependent degradation domain (ODD)-luciferase (IC50 > 28 μM), underscoring their specificity towards p97.
Regulation of the NKCC2 ion cotransporter by SPAK-OSR1-dependent and -independent pathways.
Alessi et al., Dundee, United Kingdom. In J Cell Sci, 2011
Data demonstrate that hypotonic low-chloride conditions that activate the WNK1-SPAK and OSR1 pathway promote phosphorylation of NKCC2 isoforms.
Odd-skipped maintains prohemocyte potency and blocks blood cell development in Drosophila.
Fossett et al., Baltimore, United States. In Genesis, 2011
Decapentaplegic signaling limits Odd-skipped expression to promote prohemocyte differentiation.
Hypoxia targeting gene expression for breast cancer gene therapy.
Lee, Seoul, South Korea. In Adv Drug Deliv Rev, 2009
In addition, post-translational regulation with the oxygen-dependent degradation (ODD) domain is effective to eliminate therapeutic gene products and reduce side effects in normal tissue.
Odd-skipped genes encode repressors that control kidney development.
Gómez-Skarmeta et al., Sevilla, Spain. In Dev Biol, 2007
Odd genes may also be required for proper development of the Malpighian tubules, the Drosophila renal organs
An eh1-like motif in odd-skipped mediates recruitment of Groucho and repression in vivo.
Paroush et al., Jerusalem, Israel. In Mol Cell Biol, 2005
Protein Odd-skipped, requires its eh1-like sequence to bind Groucho for repressing specific target genes in segmentation.
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