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GoPubMed Proteins lists recent and important papers and reviews for proteins. Page last changed on 19 Dec 2016.

Zinc finger, DHHC-type containing 23

Top mentioned proteins: Insulin, IDD, AGE, iNOS, CAN
Papers on NIDD
Different stress-related gene expression in depression and suicide.
Swaab et al., Amsterdam, Netherlands. In J Psychiatr Res, Sep 2015
RESULTS: In the MDD-S group, expression levels of CRH and neuronal NOS-interacting DHHC domain-containing protein with dendritic mRNA (NIDD) were increased.
Altered expression of nNOS/NIDD in the retina of a glaucoma model of DBA/2J mice and the intervention by nNOS inhibition.
Guan et al., Nantong, China. In J Mol Neurosci, 2013
The NIDD gene, neuronal NOS (nNOS)-interacting DHHC domain-containing protein with dendritic mRNA, codes a protein that upregulates nNOS enzyme activity by the interaction with the postsynaptic density protein 95/discsslarge/zon occlusens-1 (PDZ) domain of nNOS.
Positional cloning of zinc finger domain transcription factor Zfp69, a candidate gene for obesity-associated diabetes contributed by mouse locus Nidd/SJL.
Joost et al., Potsdam, Germany. In Plos Genet, 2009
Here we report the identification of a candidate gene for the diabetogenic effect of a QTL (Nidd/SJL, Nidd1) contributed by the SJL, NON, and NZB strains in outcross populations with New Zealand Obese (NZO) mice.
Altered gene expression of NIDD in dorsal root ganglia and spinal cord of rats with neuropathic or inflammatory pain.
Shen et al., Nantong, China. In J Mol Histol, 2008
NIDD mRNA was predominantly expressed in nociceptive neurons and in neurons of the spinal dorsal horn and the number of NIDD-positive neurons in the corresponding dorsal root ganglia or spinal cord increased following induction of chronic hyperalgesia.
Effect of peripheral axotomy on gene expression of NIDD in rat neural tissues.
Shen et al., Nantong, China. In J Mol Neurosci, 2006
NIDD gene expression may be involved in the different pathological conditions including nerve regeneration, neuron loss or survival, and even pain processes.
Quantitative trait locus dissection in congenic strains of the Goto-Kakizaki rat identifies a region conserved with diabetes loci in human chromosome 1q.
Gauguier et al., Oxford, United Kingdom. In Physiol Genomics, 2004
To investigate the relationship between the human and rat diabetes loci, we fine mapped the rat locus Nidd/gk2 linked to hyperinsulinemia in an F2 cross derived from the diabetic (type 2) Goto-Kakizaki (GK) rat and the Brown Norway (BN) control rat, and carried out its genetic and pathophysiological characterization in BN.GK congenic strains.
NIDD, a novel DHHC-containing protein, targets neuronal nitric-oxide synthase (nNOS) to the synaptic membrane through a PDZ-dependent interaction and regulates nNOS activity.
Suzuki et al., Matsumoto, Japan. In J Biol Chem, 2004
These results suggest that NIDD plays an important role in the regulation of the nitric oxide signaling pathway at postsynaptic sites through targeting of nueronal nitric oxide synthase to the postsynaptic membrane.
Enhanced insulin secretion and cholesterol metabolism in congenic strains of the spontaneously diabetic (Type 2) Goto Kakizaki rat are controlled by independent genetic loci in rat chromosome 8.
Gauguier et al., Oxford, United Kingdom. In Diabetologia, 2004
The aims of this study were to refine the chromosomal mapping of a QTL ( Nidd/gk5) identified in chromosome 8 of the GK rat and to define a pathophysiological profile of GK gene variants underlying the QTL effects in congenics.
Structural changes of the erythrocyte as a marker of non-insulin-dependent diabetes: protective effects of N-acetylcysteine.
Malorni et al., Roma, Italy. In Biochem Biophys Res Commun, 2002
Prooxidant-antioxidant imbalance was considered as a hallmark of age-associated, non-insulin-dependent diabetes (NIDD).
Selenium and iodine in soil, rice and drinking water in relation to endemic goitre in Sri Lanka.
Dissanayake et al., Edinburgh, United Kingdom. In Sci Total Environ, 2001
The villages were characterised by low (< 10%), moderate (10-25%) and high (> 25%) goitre incidence (NIDD, MIDD and HIDD, respectively).
Type 2 diabetes-like hyperglycemia in a backcross model of NZO and SJL mice: characterization of a susceptibility locus on chromosome 4 and its relation with obesity.
Joost et al., Aachen, Germany. In Diabetes, 2000
In male NZO x F1 backcross mice, a major susceptibility locus for the development of hyperglycemia and hypoinsulinemia (Nidd/SJL) was identified on chromosome 4 between the markers D4Mit278 and D4Mit232, 10-28 cM distal of the previously described Nidd1 locus.
Preferred meal patterns in non-insulin-dependent diabetes.
Cooper et al., Melbourne, Australia. In Asia Pac J Clin Nutr, 1993
Current advice on the across-the-day distribution of energy and carbohydrate intakes in non-insulin-dependent diabetes (NIDD) is based on inadequate evidence.
[Diabetes and heredity].
Passa et al., Paris, France. In Rev Med Interne, 1991
Genetic factors are essential to the occurrence of insulin-dependent diabetes (IDD) and non-insulin-dependent diabetes (NIDD), and all that environmental factors do is facilitate the development of diabetes in genetically predisposed subjects.
Receptors of paraneurons, with special reference to glucoreceptors.
Hashioka et al., Nagoya, Japan. In Arch Histol Cytol, 1988
The pancreatic B cell of a non-insulin-dependent diabetes (NIDD) rat model, the NSZ rat, exhibited low insulin response to glucose and did not discriminate between the two anomers of glucose.
Blockade of chlorpropamide-alcohol flushing by indomethacin suggests an association between prostaglandins and diabetic vascular complications.
Pyke et al., In Lancet, 1980
Chlorpropamide/alcohol flushing (CPAF), found in many patients with non-insulin-dependent diabetes (NIDD), can be blocked by indomethacin in most patients who are free of vascular complications but not in those with such complications.
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