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GoPubMed Proteins lists recent and important papers and reviews for proteins. Page last changed on 19 Dec 2016.

Non-SMC condensin II complex, subunit G2

Condensin complexes I and II play essential roles in mitotic chromosome assembly and segregation. Both condensins contain 2 invariant structural maintenance of chromosome (SMC) subunits, SMC2 (MIM 605576) and SMC4 (MIM 605575), but they contain different sets of non-SMC subunits. NCAPG2 is 1 of 3 non-SMC subunits that define condensin II (Ono et al., 2003 [PubMed 14532007]).[supplied by OMIM, Mar 2008] (from NCBI)
Top mentioned proteins: CAN, ACID, V1a, HAD, CD4
Papers using Mtb antibodies
Integrated, nontargeted ultrahigh performance liquid chromatography/electrospray ionization tandem mass spectrometry platform for the identification and relative quantification of the small-molecule complement of biological systems.
Waldor Matthew K., In PLoS Biology, 2008
... (Mtb) was grown in Middlebrook 7H9 broth (Difco) supplemented with 0.05% Tween-80 and ADC enrichment, or on 7H10 agar with 10% OADC enrichment (Becton Dickinson) ...
A short-term model for preliminary screening of potential anti-tubercular compounds
Schaible Ulrich E. et al., In Journal of Antimicrobial Chemotherapy, 2008
... Mtb codon preferences, synthesized (Genscript USA Inc., Piscataway, USA) ...
Mycobacterium tuberculosis controls host innate immune activation through cyclopropane modification of a glycolipid effector molecule
Glickman Michael S. et al., In The Journal of Experimental Medicine, 1999
... Solid media for the growth of Mtb was Middlebrook 7H10 (Becton Dickinson) with 10% OADC and ...
Papers on Mtb
A luminescence assay for natural product inhibitors of the Mycobacterium tuberculosis proteasome.
Porter et al., Philadelphia, United States. In Phytochem Anal, Feb 2016
INTRODUCTION: Mycobacterium tuberculosis (Mtb) causes a large global burden of disease, with a high mortality rate in healthy and immuno-compromised patients.
Molecular principles behind pyrazinamide resistance due to mutations in panD gene in Mycobacterium tuberculosis.
Grover et al., New Delhi, India. In Gene, Feb 2016
Mutations have been detected in panD gene in the Mycobacterium tuberculosis (Mtb) strains.
Construction of Mycobacterium tuberculosis ESAT6 fused to human Fcγ of IgG1: To target FcγR as a delivery system for enhancement of immunogenicity.
Rezaee et al., Mashhad, Iran. In Gene, Feb 2016
UNASSIGNED: In order to prevent spreading of Mycobacterium tuberculosis (Mtb), it is necessary to discover effective vaccines, fast and reliable diagnosis and appropriate treatment schemes.
Lipoarabinomannan, and its related glycolipids, induce divergent and opposing immune responses to Mycobacterium tuberculosis depending on structural diversity and experimental variations.
Svenson et al., Stockholm, Sweden. In Tuberculosis (edinb), Jan 2016
Exposure to Mycobacterium tuberculosis (Mtb) may lead to active or latent tuberculosis, or clearance of Mtb, depending essentially on the quality of the host's immune response.
Mycobacterium tuberculosis EsxO (Rv2346c) promotes bacillary survival by inducing oxidative stress mediated genomic instability in macrophages.
Sonawane et al., Bhubaneshwar, India. In Tuberculosis (edinb), Jan 2016
Mycobacterium tuberculosis (Mtb) survives inside the macrophages by modulating the host immune responses in its favor.
Interdependence between Interleukin-1 and Tumor Necrosis Factor Regulates TNF-Dependent Control of Mycobacterium tuberculosis Infection.
Shayakhmetov et al., Atlanta, United States. In Immunity, Jan 2016
The interleukin-1 receptor I (IL-1RI) is critical for host resistance to Mycobacterium tuberculosis (Mtb), yet the mechanisms of IL-1RI-mediated pathogen control remain unclear.
Host-directed therapy targeting the Mycobacterium tuberculosis granuloma: a review.
Basaraba et al., Fort Collins, United States. In Semin Immunopathol, Nov 2015
UNASSIGNED: Infection by the intracellular bacterial pathogen Mycobacterium tuberculosis (Mtb) is a major cause of morbidity and mortality worldwide.
Pathology and immune reactivity: understanding multidimensionality in pulmonary tuberculosis.
Kaufmann et al., Berlin, Germany. In Semin Immunopathol, Nov 2015
UNASSIGNED: Heightened morbidity and mortality in pulmonary tuberculosis (TB) are consequences of complex disease processes triggered by the causative agent, Mycobacterium tuberculosis (Mtb).
Recent advances in the development of vaccines for tuberculosis.
Ahsan, Jaipur, India. In Ther Adv Vaccines, May 2015
There are nearly 15 vaccine candidates in various phases of clinical trials, includes five protein or adjuvant vaccines, four viral-vectored vaccines, three mycobacterial whole cell or extract vaccines, and one each of the recombinant live and the attenuated Mycobacterium tuberculosis (Mtb) vaccine.
The balance between protective and pathogenic immune responses in the TB-infected lung.
Cooper et al., Fort Collins, United States. In Nat Immunol, 2015
Mycobacterium tuberculosis (Mtb) is able to manipulate both the innate and acquired immune response of the host.
The application of tetracyclineregulated gene expression systems in the validation of novel drug targets in Mycobacterium tuberculosis.
Mizrahi et al., Cape Town, South Africa. In Front Microbiol, 2014
The elucidation of the genome sequence of Mycobacterium tuberculosis (Mtb) facilitated a shift to target-based approaches to drug design but these efforts have proven largely unsuccessful.
Tryptophan biosynthesis protects mycobacteria from CD4 T-cell-mediated killing.
Rubin et al., Boston, United States. In Cell, 2014
Here, we present a genome-scale study of Mycobacterium tuberculosis (Mtb) that uncovers the bacterial determinants of surviving host immunity, sets of genes we term "counteractomes."
Autophagy regulates phagocytosis by modulating the expression of scavenger receptors.
Eissa et al., Houston, United States. In Immunity, 2013
Atg7⁻/⁻ macrophages exhibited higher bacterial uptake when infected with Mycobacterium tuberculosis (Mtb) or with M. tuberculosis var.
Alteration of human macrophages microRNA expression profile upon infection with Mycobacterium tuberculosis.
Cirillo et al., Milano, Italy. In Int J Mycobacteriol, 2013
BACKGROUND: Mycobacterium tuberculosis (Mtb) has evolved multiple mechanisms to manipulate its cellular niche for its own advantage.
Pathogen-specific Treg cells expand early during mycobacterium tuberculosis infection but are later eliminated in response to Interleukin-12.
Urdahl et al., Seattle, United States. In Immunity, 2013
Here we show that pulmonary infection with Mycobacterium tuberculosis (Mtb), but not Listeria monocytogenes (Lm), induced robust lymph node expansion of a highly activated population of pathogen-specific Treg cells from the pre-existing pool of thymically derived Treg cells.
Microcephalin/MCPH1 associates with the Condensin II complex to function in homologous recombination repair.
Chen et al., New Haven, United States. In J Biol Chem, 2008
Microcephalin/MCPH1 associates with the Condensin II complex to function in homologous recombination repair
MTB, the murine homolog of condensin II subunit CAP-G2, represses transcription and promotes erythroid cell differentiation.
Crispino et al., Chicago, United States. In Leukemia, 2006
the condensin II subunit MTB/mCAP-G2 plays a novel function during erythropoiesis
Differential contributions of condensin I and condensin II to mitotic chromosome architecture in vertebrate cells.
Hirano et al., United States. In Cell, 2003
The CAP-G2 subunit of the condensin II complex implicated in chromosome assembly and segregation
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