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GoPubMed Proteins lists recent and important papers and reviews for proteins. Page last changed on 19 Dec 2016.

MYC induced nuclear antigen

Mina, Mina53
MINA is a c-Myc (MYC; MIM 190080) target gene that may play a role in cell proliferation or regulation of cell growth. (Tsuneoka et al., 2002 [PubMed 12091391]; Zhang et al., 2005 [PubMed 15897898]).[supplied by OMIM, May 2008] (from NCBI)
Top mentioned proteins: c-Myc, HAD, acetylcholinesterase, CAN, fibrillin-1
Papers on Mina
Histone Modifier Genes Alter Conotruncal Heart Phenotypes in 22q11.2 Deletion Syndrome.
Morrow et al., United States. In Am J Hum Genet, Jan 2016
Eighteen case subjects (20%) had rdSNVs in four genes (JMJD1C, RREB1, MINA, KDM7A) all involved in demethylation of histones (H3K9, H3K27).
The proteomic investigation reveals interaction of mdig protein with the machinery of DNA double-strand break repair.
Chen et al., Detroit, United States. In Oncotarget, Oct 2015
To investigate how mineral dust-induced gene (mdig, also named as mina53, MINA, or NO52) promotes carcinogenesis through inducing active chromatin, we performed proteomics analyses for the interacting proteins that were co-immunoprecipitated by anti-mdig antibody from either the lung cancer cell line A549 cells or the human bronchial epithelial cell line BEAS-2B cells.
Current understanding of mdig/MINA in human cancers.
Chen et al., Detroit, United States. In Genes Cancer, Jul 2015
UNLABELLED: Mineral dust-induced gene, mdig has recently been identified and is known to be overexpressed in a majority of human cancers and holds predictive power in the poor prognosis of the disease.
Integrative network analysis for survival-associated gene-gene interactions across multiple genomic profiles in ovarian cancer.
Sohn et al., Suwŏn, South Korea. In J Ovarian Res, 2014
METHODS: We propose a mutual information-based integrative network analysis framework (MINA) that identifies gene pairs associated with clinical outcome and systematically analyses the resulting networks over multiple genomic profiles.
Mina: a Th2 response regulator meets TGFβ.
Bix et al., Memphis, United States. In Curr Opin Immunol, 2014
The JmjC protein Mina is an important immune response regulator.
Ribosomal oxygenases are structurally conserved from prokaryotes to humans.
Schofield et al., Oxford, United Kingdom. In Nature, 2014
In Escherichia coli, YcfD catalyses arginine hydroxylation in the ribosomal protein L16; in humans, MYC-induced nuclear antigen (MINA53; also known as MINA) and nucleolar protein 66 (NO66) catalyse histidine hydroxylation in the ribosomal proteins RPL27A and RPL8, respectively.
Mina53, a novel molecular marker for the diagnosis and prognosis of gastric adenocarcinoma.
Chen et al., Shanghai, China. In Oncol Rep, 2014
Mina53 is a direct novel target protein of Myc.
Paradoxical roles of mineral dust induced gene on cell proliferation and migration/invasion.
Chen et al., Detroit, United States. In Plos One, 2013
Increased expression of mineral dust-induced gene (mdig, also named as mina53, MINA, or NO52) has been observed in a number of human cancers.
Bacteria and genes involved in arsenic speciation in sediment impacted by long-term gold mining.
Nascimento et al., Belo Horizonte, Brazil. In Plos One, 2013
Sediment was collected from the historically gold mining impacted Mina stream, located in one of the world's largest mining regions known as the "Iron Quadrangle".
Survey of predictive value of 4-hour urine collection for diagnosis of proteinuria in preeclampsia.
Moslehi et al., Rasht, Iran. In Iran J Reprod Med, 2013
(Mina Moslehi).
Acetylcholinesterase reactivators (HI-6, obidoxime, trimedoxime, K027, K075, K127, K203, K282): structural evaluation of human serum albumin binding and absorption kinetics.
Kuca et al., Hradec Králové, Czech Republic. In Int J Mol Sci, 2012
Current efforts at treatments for OPNA exposure are focused on non-quaternary reactivators, monoisonitrosoacetone oximes (MINA), and diacylmonoxime reactivators (DAM).
Ablation of Mina53 in mice reduces allergic response in the airways.
Tsuneoka et al., Takasaki, Japan. In Cell Struct Funct, 2012
The mina53 (myc-induced nuclear antigen with a 53 kDa molecular mass; also known as mina) was identified as a direct transcriptional target of the oncoprotein Myc and encodes a conserved protein in vertebrates.
Transcriptional activation of Mina by Sp1/3 factors.
Bix et al., Memphis, United States. In Plos One, 2012
Mina is an epigenetic gene regulatory protein known to function in multiple physiological and pathological contexts, including pulmonary inflammation, cell proliferation, cancer and immunity.
Associations of the single-nucleotide polymorphisms of the Mina gene with the development of asthma in Chinese Han children: a case-control study.
Wang et al., Chongqing, China. In Genet Test Mol Biomarkers, 2011
The single nucleotide polymorphism of the Mina gene is associated with the development of asthma in Chinese Han children.
An outsider's perspective--ecotaxis revisited: an integrative review of cancer environment, iron and immune system cells.
de Sousa, Porto, Portugal. In Integr Biol (camb), 2011
Finally, I conclude with wondering how much longer what I call the 'Hunter Paradigm' will dominate cancer research and immunology and how timely it is to acknowledge in the first decade of a new century, Mina Bissell as a pioneer in the change of that paradigm in Cancer Research.
Expression of Mina53, a novel c-Myc target gene, is a favorable prognostic marker in early stage lung cancer.
Sueoka et al., Saga, Japan. In Lung Cancer, 2010
favorable prognostic marker in early stage lung cancer
Mina53, a novel c-Myc target gene, is frequently expressed in lung cancers and exerts oncogenic property in NIH/3T3 cells.
Sueoka et al., Saga, Japan. In J Cancer Res Clin Oncol, 2010
Mina53 plays an important role in carcinogenesis and may be a target for cancer prevention.
Mina, an Il4 repressor, controls T helper type 2 bias.
Bix et al., Yokohama, Japan. In Nat Immunol, 2009
Mina specifically bound to and repressed the Il4 promoter. Mina overexpression in transgenic mice impaired Il4 expression, whereas its knockdown in primary CD4(+) T cells led to Il4 derepression.
Lung cancer-associated JmjC domain protein mdig suppresses formation of tri-methyl lysine 9 of histone H3.
Chen et al., Morgantown, United States. In Cell Cycle, 2009
The MINA protein alters histone H3 methylation contributes to the initiation or development of the human lung cancer.
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