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Melanoma antigen family A, 9B

MAGEA9B is a duplication of the MAGEA9 gene (MIM 300342) on chromosome Xq28. The 2 copies are separated by about 194 kb (Hartz, 2009).[supplied by OMIM, Mar 2009] (from NCBI)
Top mentioned proteins: OUT, CAN, PHD2, MAGE-B2, Ubiquitin
Papers on MAGE-9
High expression of MAGE-A9 in tumor and stromal cells of non-small cell lung cancer was correlated with patient poor survival.
Huang et al., Nantong, China. In Int J Clin Exp Pathol, 2014
We concluded that the molecular assessment of MAGEA9 could be considered to improve prognostic evaluation and to identify eligible patients for potential target therapy.
Expression of cancer-testis antigens MAGEA1, MAGEA3, ACRBP, PRAME, SSX2, and CTAG2 in myxoid and round cell liposarcoma.
Iwenofu et al., Columbus, United States. In Mod Pathol, 2014
Gene expression studies have reported the expression of various cancer-testis antigens in liposarcoma, with mRNA expression of CTAG1B, CTAG2, MAGEA9, and PRAME described specifically in myxoid and round-cell liposarcoma.
Recurrent X chromosome-linked deletions: discovery of new genetic factors in male infertility.
Krausz et al., Barcelona, Spain. In J Med Genet, 2014
MAGEA9, an ampliconic gene reported as independently acquired on the human X chromosome with exclusive physiological expression in the testis, is likely to be involved in CNV67.
Cancer testis antigens in newly diagnosed and relapse multiple myeloma: prognostic markers and potential targets for immunotherapy.
Sonneveld et al., Rotterdam, Netherlands. In Haematologica, 2011
Multivariate analysis demonstrated that presence of MAGEA6 and CDCA1 were clearly associated with shorter progression free survival, and presence of MAGEA9 with shorter overall survival in the set of newly diagnosed cases.
High frequency of MAGE-A4 and MAGE-A9 expression in high-risk bladder cancer.
Fradet et al., Québec, Canada. In Int J Cancer, 2009
Overexpression of MAGE-A9 is associated with bladder cancer.
Melanoma antigen-11 inhibits the hypoxia-inducible factor prolyl hydroxylase 2 and activates hypoxic response.
Niederhuber et al., Bethesda, United States. In Cancer Res, 2009
Furthermore, MAGE-9, the closest homologue of MAGE-11, was also found to interact with PHD2.
Double complex mutations involving F8 and FUNDC2 caused by distinct break-induced replication.
Chen et al., Christchurch, New Zealand. In Hum Mutat, 2007
The copy number of several genes is affected by this rearrangement, with deletion of part of the Factor VIII gene (F8, causing hemophilia A) and the FUNDC2 gene, and duplication of the TMEM185A, HSFX1, MAGEA9, and MAGEA11 genes.
Global expression analysis of cancer/testis genes in uterine cancers reveals a high incidence of BORIS expression.
Barrett et al., Bethesda, United States. In Clin Cancer Res, 2007
The expression of the two most commonly expressed CT genes on the arrays, MAGEA9 (24 of 122 cancers and 0 of 10 normal tissues) and Down syndrome critical region 8 (DSCR8)/MMA1 (16 if 122 cancers and 0 of 10 normal tissues), was confirmed by reverse transcription-PCR methods, validating the array screening approach.
Generation of RAGE-1 and MAGE-9 peptide-specific cytotoxic T-lymphocyte lines for transfer in patients with renal cell carcinoma.
Zöller et al., Heidelberg, Germany. In Int J Cancer, 2005
May provide suitable targets for immunotherapy of renal cell carcinoma.
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