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GoPubMed Proteins lists recent and important papers and reviews for proteins. Page last changed on 19 Dec 2016.

Lipoprotein lipase

Lipoprotein Lipase, LPL
Top mentioned proteins: ACID, HAD, CAN, Insulin, HDL
Papers using Lipoprotein Lipase antibodies
Effect of adipocyte beta3-adrenergic receptor activation on the type 2 diabetic MKR mice
Luque Raul M., In PLoS ONE, 2005
... antibody (Cell Signaling Technology, Danvers, MA); mouse monoclonal lipoprotein lipase (LPL) antibody, and rabbit polyclonal HSL antibody (Abcam Inc, Cambridge, MA) ...
Molecular dissection of ligand binding sites on the low density lipoprotein receptor-related protein
Pedrini Michael T. et al., In Molecular and Cellular Endocrinology, 1993
... anti-GSK-3 (anti-glycogen synthase kinase 3) antibody was obtained from Upstate (Charlottesville, VA, USA) and the LPL antibody from Abcam (Cambridge, UK); anti-phospho-GSK-3/β-ser21/9, anti-phospho-Akt-ser473 ...
Papers on Lipoprotein Lipase
Interaction of lipoprotein lipase polymorphisms with body mass index and birth weight to modulate lipid profiles in children and adolescents: the CASPIAN-III Study.
Kelishadi et al., Eşfahān, Iran. In Sao Paulo Med J, Feb 2016
This study aimed to determine to what extent variants of lipoprotein lipase (LPL) might interact with birth weight or body weight in determining the lipid profile concentrations in children and adolescents.
Inhibition by Seeds of Phalaris canariensis Extracts of Key Enzymes Linked to Obesity.
Cruz Victoria et al., In Altern Ther Health Med, Jan 2016
Lipoprotein lipase (LPL) activity was measured by released (3H)-oleic acid.
Expression of lipases and lipid receptors in sperm storage tubules and possible role of fatty acids in sperm survival in the hen oviduct.
Yoshimura et al., Hiroshima, Japan. In Theriogenology, Jan 2016
Expression of genes encoding endothelial lipase (EL), lipase H (LIPH), adipose triglyceride lipase (ATGL), and lipoprotein lipase (LPL) were found in UVJ.
Disturbances in apoptosis of lamina propria lymphocytes in Crohn's disease.
Linke et al., Poznań, Poland. In Arch Med Sci, Jan 2016
INTRODUCTION: The aim of this study was to assess the potential mechanisms providing resistance to apoptosis of lamina propria lymphocytes (LPL) directlyin intestinal tissues from patients with Crohn's disease (CD).
Metabolism pathways in chronic lymphocytic leukemia.
Estrov et al., Petah Tikva, Israel. In Leuk Lymphoma, Jan 2016
STAT3, known to be constitutively activated in CLL, increases the levels of lipoprotein lipase (LPL) that mediates lipoprotein uptake and shifts the CLL cells' metabolism towards utilization of FFA.
Meta-analysis of lipid-traits in Hispanics identifies novel loci, population-specific effects, and tissue-specific enrichment of eQTLs.
Valladares-Salgado et al., Houston, United States. In Sci Rep, Dec 2015
Genome-wide significant signals were observed in or near CELSR2, ZNF259/APOA5, KANK2/DOCK6 and NCAN/MAU2 for total cholesterol, LPL, ABCA1, ZNF259/APOA5, LIPC and CETP for HDL cholesterol, CELSR2, APOB and NCAN/MAU2 for LDL cholesterol, and GCKR, TRIB1, ZNF259/APOA5 and NCAN/MAU2 for triglycerides.
Update on the molecular biology of dyslipidemias.
Ramasamy, Worcester, United Kingdom. In Clin Chim Acta, Dec 2015
Monogenic hypertriglyceridemia is the result of mutations in genes that regulate the metabolism of triglyceride rich lipoproteins (eg LPL, APOC2, APOA5, LMF1, GPIHBP1).
The Physiological Regulation of Skeletal Muscle Fatty Acid Supply and Oxidation During Moderate-Intensity Exercise.
van Hall, Copenhagen, Denmark. In Sports Med, Nov 2015
Moreover, NEFA from lipolysis via lipoprotein lipase (LPL) in the muscle of the very-low-density lipoproteins and in the (semi) post-prandial state chylomicrons may also contribute.
The many faces of small B cell lymphomas with plasmacytic differentiation and the contribution of MYD88 testing.
Campo et al., Pittsburgh, United States. In Virchows Arch, Nov 2015
UNASSIGNED: Plasmacytic differentiation may occur in almost all small B cell lymphomas (SBLs), although it varies from being uniformly present (as in lymphoplasmacytic lymphoma (LPL)) to very uncommon (as in mantle cell lymphomas (MCLs)).
Association of Genetic Variants with Polypoidal Choroidal Vasculopathy: A Systematic Review and Updated Meta-analysis.
Chen et al., Hong Kong, Hong Kong. In Ophthalmology, Sep 2015
Another 25 polymorphisms in 13 genes (ARMS2, HTRA1, C2, CFB, ELN, LIPC, LPL, ABCA1, VEGF-A, TLR3, LOXL1, SERPING1, and PEDF) had no significant association.
Targeting APOC3 in the familial chylomicronemia syndrome.
Witztum et al., Montréal, Canada. In N Engl J Med, 2015
The familial chylomicronemia syndrome is a genetic disorder characterized by severe hypertriglyceridemia and recurrent pancreatitis due to a deficiency in lipoprotein lipase (LPL).
The ER-associated degradation adaptor protein Sel1L regulates LPL secretion and lipid metabolism.
Qi et al., Ithaca, United States. In Cell Metab, 2014
Further analyses reveal that Sel1L is indispensable for the secretion of lipoprotein lipase (LPL), independent of its role in Hrd1-mediated ERAD and ER homeostasis.
The GPIHBP1-LPL complex is responsible for the margination of triglyceride-rich lipoproteins in capillaries.
Fong et al., Los Angeles, United States. In Cell Metab, 2014
Triglyceride-rich lipoproteins (TRLs) undergo lipolysis by lipoprotein lipase (LPL), an enzyme that is transported to the capillary lumen by an endothelial cell protein, GPIHBP1.
Circulating angiopoietin-like 4 links proteinuria with hypertriglyceridemia in nephrotic syndrome.
Chugh et al., Birmingham, United States. In Nat Med, 2014
But at the same time, in a local feedback loop, the elevated extrarenal pools of Angptl4 reduced tissue FFA uptake in skeletal muscle, heart and adipose tissue, subsequently resulting in hypertriglyceridemia, by inhibiting lipoprotein lipase (LPL)-mediated hydrolysis of plasma triglycerides to FFAs.
A largely random AAV integration profile after LPLD gene therapy.
Schmidt et al., Heidelberg, Germany. In Nat Med, 2013
We performed integration-site analysis after AAV1-LPL(S447X) intramuscular injection in five lipoprotein lipase-deficient subjects, revealing random nuclear integration and hotspots in mitochondria.
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