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TNFAIP3 interacting protein 3

Lind, Listeria-induced, ABIN-3
Top mentioned proteins: CAN, HAD, A20, IL-10, IL-1beta
Papers on Lind
Phenotype and transcriptome analysis reveals chloroplast development and pigment biosynthesis together influenced the leaf color formation in mutants of Anthurium andraeanum 'Sonate'.
Wang et al., Nanjing, China. In Front Plant Sci, 2014
Leaf color is one of the well-sought traits in breeding program for Anthurium andraeanum Lind.
Cloning and expression of the sucrose transporter gene PsSUT1 from tree peony leaf.
He et al., Zhengzhou, China. In Genet Mol Res, 2014
This study reports the cloning of a sucrose transporter gene, PsSUT1, from the leaf of tree peony (Paeonia suffruticosa Lind.
Co-modification of class B genes TfDEF and TfGLO in Torenia fournieri Lind. alters both flower morphology and inflorescence architecture.
Ohtsubo et al., Tsukuba, Japan. In Plant Mol Biol, 2014
Here, we examine the cooperative functions of TfDEF and TfGLO on floral organ development in the ornamental plant torenia (Torenia fournieri Lind.).
The biology of A20-binding inhibitors of NF-kappaB activation (ABINs).
Beyaert et al., In Adv Exp Med Biol, 2013
The family of A20-Binding Inhibitors of NF-kappaB (ABINs) consists of three proteins, ABIN-1, ABIN-2 and ABIN-3, which were originally identified as A20-binding proteins and inhibitors of cytokines and Lipopolysaccharide (LPS) induced NF-kappaB activation.
Molecular characterization of Anthurium genotypes by using DNA fingerprinting and SPAR markers.
Silva et al., Wuhan, China. In Genet Mol Res, 2013
We characterized single primer amplification reaction (SPAR) molecular markers from 20 genotypes of Anthurium andraeanum Lind., including 3 from commercial varieties and 17 from 2 communities in the State of Espírito Santo, Brazil.
Molecular characterization of mutations in white-flowered torenia plants.
Nakatsuka et al., Kitakami, Japan. In Bmc Plant Biol, 2013
BACKGROUND: Torenia (Torenia fournieri Lind.) is a model plant increasingly exploited in studies in various disciplines, including plant engineering, biochemistry, physiology, and ecology.
Tumor necrosis factor α induces sustained signaling and a prolonged and unremitting inflammatory response in rheumatoid arthritis synovial fibroblasts.
Kalliolias et al., New York City, United States. In Arthritis Rheum, 2013
Furthermore, FLS expressed low levels of the feedback inhibitors A20-binding inhibitor of NF-κB activation 3 (ABIN-3), IL-1 receptor-associated kinase M (IRAK-M), suppressor of cytokine signaling 3 (SOCS-3), and activating transcription factor 3 (ATF-3), which terminate inflammatory responses in macrophages.
β(2) integrins inhibit TLR responses by regulating NF-κB pathway and p38 MAPK activation.
Hamerman et al., Seattle, United States. In Eur J Immunol, 2013
TLR hypersensitivity was not the result of insufficient ABIN-3, A20, Hes-1, or IRAK-M expression, nor to changes in IL-10 production or sensitivity, though Itgb2(-/-) macrophages had reduced p38 MAPK phosphorylation after LPS treatment.
Listeria monocytogenes triggers the cell surface expression of Gp96 protein and interacts with its N terminus to support cellular infection.
Sousa et al., Porto, Portugal. In J Biol Chem, 2013
Our findings unravel the Listeria-induced surface expression of Gp96 and the topology of its insertion on the plasma membrane and improve our knowledge on the Vip-Gp96 interaction during Listeria infection.
Simvastatin enhances protection against Listeria monocytogenes infection in mice by counteracting Listeria-induced phagosomal escape.
Brombacher et al., Cape Town, South Africa. In Plos One, 2012
Statins are well-known cholesterol lowering drugs targeting HMG-CoA-reductase, reducing the risk of coronary disorders and hypercholesterolemia. Statins are also involved in immunomodulation, which might influence the outcome of bacterial infection.
A pro-inflammatory role for A20 and ABIN family proteins in human fibroblast-like synoviocytes in rheumatoid arthritis.
Ishihara et al., Kurashiki, Japan. In Immunol Lett, 2012
recombinant transgene facilitates the transcription of pro-inflammatory cytokines in fibroblast-like synoviocytes in rheumatoid arthritis
Indirect inhibition of Toll-like receptor and type I interferon responses by ITAM-coupled receptors and integrins.
Ivashkiv et al., New York City, United States. In Immunity, 2010
We show that high-avidity ligation of ITAM-coupled beta2 integrins and FcgammaRs in macrophages inhibited type I interferon receptor and Toll-like receptor (TLR) signaling and induced expression of interleukin-10 (IL-10); signaling inhibitors SOCS3, ABIN-3, and A20; and repressors of cytokine gene transcription STAT3 and Hes1.
Adiponectin inhibits pro-inflammatory signaling in human macrophages independent of interleukin-10.
Libby et al., Boston, United States. In J Biol Chem, 2009
adiponectin augmented the expression of A20, suppressor of cytokine signaling (SOCS) 3, B-cell CLL/lymphoma (BCL) 3, TNF receptor-associated factor (TRAF) 1, and TNFAIP3-interacting protein (TNIP) 3.
ABINs: A20 binding inhibitors of NF-kappa B and apoptosis signaling.
Beyaert et al., Gent, Belgium. In Biochem Pharmacol, 2009
This review defines ABIN-3 based on three different parameters: ability to bind A20; ability to inhibit NF-kappaB activation upon overexpression; the presence of specific short amino acid regions of strong homology, designated ABIN homology domains.
Expression of the NF-kappaB inhibitor ABIN-3 in response to TNF and toll-like receptor 4 stimulation is itself regulated by NF-kappaB.
Beyaert et al., Gent, Belgium. In J Cell Mol Med, 2008
Enhanced expression of ABIN-3 in monocytes is associated with sepsis
ABIN-3: a molecular basis for species divergence in interleukin-10-induced anti-inflammatory actions.
Schreiber et al., Saint Louis, United States. In Mol Cell Biol, 2007
ABIN-3 is, thus, an IL-10-induced gene product capable of attenuating NF-kappaB in human macrophages yet is inoperative in mice and represents a basis for species-specific differences in IL-10 actions.
Murine model of pregnancy-associated Listeria monocytogenes infection.
Deckert et al., Rijeka, Croatia. In Fems Immunol Med Microbiol, 2003
This resulted in severe necrotizing hemorrhagic hepatitis and Listeria-induced placental necrosis, increasing the incidence of postimplantation loss and poor pregnancy outcome.
[Listeria arthritis in chronic polyarthritis during low dose prednisolone and methotrexate therapy. Case report and review of the literature].
Becker-Capeller et al., Cuxhaven, Germany. In Z Rheumatol, 2001
Clinically, the Listeria-induced septic arthritis could not be differentiated from rheumatoid arthritis; fever was not present.
Development of TH1 CD4+ T cells through IL-12 produced by Listeria-induced macrophages.
Murphy et al., Saint Louis, United States. In Science, 1993
Development of the appropriate CD4+ T helper (TH) subset during an immune response is important for disease resolution.
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