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GoPubMed Proteins lists recent and important papers and reviews for proteins. Page last changed on 19 Dec 2016.

XRCC6 binding protein 1

KUB3, Ku70 binding protein 3, XRCC6BP1
Top mentioned proteins: IRBP, V1a, p53, mdm2, CDK4
Papers on KUB3
Biological effects of passive versus active scanning proton beams on human lung epithelial cells.
Luo-Owen et al., Loma Linda, United States. In Technol Cancer Res Treat, Feb 2015
In contrast, many more genes had >2-fold difference with ABS protons: BRCA1, BRCA2, CDC25A, CDC25C, CCNB2, CDK1, DMC1, DNMT1, E2F1, EXO1, FEN1, GADD45A, GTSE1, IL-6, JUN, KRAS, MDM4, PRC1, PTTG1, RAD51, RPA1, TNF, WT1, XRCC2, XRCC3 and XRCC6BP1.
Glioma-amplified sequence KUB3 influences double-strand break repair after ionizing radiation.
Meese et al., Homburg, Germany. In Int J Oncol, 2013
Previously, we cloned Ku70-binding protein 3 (KUB3) from the amplified domain at 12q13-15.
Mitomycin C modulates DNA-double strand break repair genes in cervical carcinoma cells.
Yoon et al., Seoul, South Korea. In Amino Acids, 2010
Ku70-binding protein (KUB3), MHC class I antigen, MHC class I chain-related protein A or multi-PDZ domain protein 1, MAGUK P55 subfamily member 3 or lamda/iota protein kinase C-interacting protein, and GL014 or Sad1/unc-84 protein-like 1 were suppressed by mitomycin C treatment.
Amplicons on chromosome 12q13-21 in glioblastoma recurrences.
Meese et al., Homburg, Germany. In Int J Cancer, 2010
We recognized a significant longer survival time in glioblastoma patients that lack amplifications of either CDK4, CYP27B1, XRCC6BP1 (KUB3), or MDM2.
The effect of single nucleotide polymorphisms from genome wide association studies in multiple sclerosis on gene expression.
Ramagopalan et al., Oxford, United Kingdom. In Plos One, 2009
Several SNPs were found to be associated with changes in expression: in particular two with HLA-DQA1, HLA-DQA2, HLA-DQB1, HLA-DRB1, HLA-DRB4 and HLA-DRB5, one with ZFP57, one with CD58, two with IL7 and FAM164A, and one with FAM119B, TSFM and KUB3.
A different view on DNA amplifications indicates frequent, highly complex, and stable amplicons on 12q13-21 in glioma.
Meese et al., Homburg, Germany. In Mol Cancer Res, 2008
Expression analysis of the amplified region revealed 10 overexpressed genes (i.e., KUB3, CTDSP2, CDK4, OS-9, DCTN2, RAB3IP, FRS2, GAS41, MDM2, and RAP1B) that were consistently overexpressed in all cases that carried this amplification.
Glioblastoma multiforme: the role of DSB repair between genotype and phenotype.
Meese et al., Homburg, Germany. In Oncogene, 2008
Specifically, we address the role of DNA-PKcs and the novel potential end-joining factor KUB3 in maintaining the radioresistant phenotype, the interrelationship between genetic lesions and repair mechanisms, and new perspectives that emerge from the identification of glioblastoma stem cells.
KUB3 amplification and overexpression in human gliomas.
Meese et al., Homburg, Germany. In Glia, 2001
In this article, we demonstrate that GAS16 results from an alternative splicing process of the Ku70 binding protein 3 (KUB3) that is essential for DNA double-strand break repair.
Isolation of Ku70-binding proteins (KUBs).
Boothman et al., Cleveland, United States. In Nucleic Acids Res, 1999
KUB3 and KUB4 are unknown, >10 kb trans-cripts.
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