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GoPubMed Proteins lists recent and important papers and reviews for proteins. Page last changed on 19 Dec 2016.

Interferon, alpha 5

IFNA5, interferon alpha-C, interferon-alpha 5
Top mentioned proteins: Interferon-alpha, IFNA6, HAD, SET, Hoxa5
Papers on IFNA5
The relationship between IFNA1, IFNA5, IFNA10 and IFNA17 gene polymorphisms and Crimean-Congo Hemorrhagic Fever prognosis in a Turkish population range.
Dokmetas et al., Sivas, Turkey. In J Med Virol, Jan 2016
Hence, the relationship between SNPs in genes encoding cytokines (IFNA1 -1823G/A (rs1332190), IFNA5 -2529T/A (rs758236), IFNA10 Cys20stop (rs10119910), and IFNA17 Ile184Arg (rs9298814) SNPs and disease susceptibility were investigated.
Messenger RNAs bearing tRNA-like features exemplified by interferon alfa 5 mRNA.
Gómez et al., Granada, Spain. In Cell Mol Life Sci, Oct 2015
One of these cleavage sites was found in interferon-alpha 5 (IFNA5) mRNA between specific base positions and with the characteristic RNase-P chemistry of cleavage.
Expression patterns of immune-associated genes in external genital and perianal warts treated with sinecatechins.
Tyring et al., Houston, United States. In Viral Immunol, May 2015
Genes that were significantly regulated between VRs and VNRs were CREB3L4, HIST1H3A, HIST1H3H, IFNA1, IFNA4, IFNA5, IFNA6, IFNA8, IFNA14, IFNG, IFNAR1, IL6, IRF9, MAPK4, MAPK5, MAPK14, NET1, and PIK3C2A in the IFN array.
Expression of B-cell-associated genes in peripheral blood mononuclear cells of patients with symptomatic pulmonary embolism.
Song et al., Shanghai, China. In Mol Med Report, Mar 2015
Furthermore, the expression levels of B‑cell‑activation‑associated cytokine genes demonstrated a significant upregulation of LTA and IL10 and downregulation of L1A, IFNA5, IFNA6, IFNA8, IFNA14, IL2, IL13 and IFNG in PE patients compared to those of the control group.
Association of interferon-alpha gene polymorphisms with chronic hepatitis B virus infection.
Hirankarn et al., Bangkok, Thailand. In Int J Immunogenet, 2013
By multifactor dimensionality reduction analysis, we found the interaction between IFNA5 (-2529) and IFNA1 (-1823) genes that gave the risk to chronic HBV infection, with the OR (95% CI) of the high-risk to low-risk group was 2.79 (1.77-4.40),
Epidemiological and genetic characteristics associated with the severity of acute viral bronchiolitis by respiratory syncytial virus.
Ribeiro et al., Campinas, Brazil. In J Pediatr (rio J), 2013
Polymorphisms of the TLRs, RANTES, JUN, IFNA5, NOS2, CX3CR1, ILs, and VDR genes have been shown to be associated with more severe evolution of AVB by RSV.
Synthesis and expression of recombinant interferon alpha-5 gene in tobacco chloroplasts, a non-edible plant.
Nurjis et al., Faisalābād, Pakistan. In Mol Biol Rep, 2012
In the present study the interferon alpha 5 gene was synthesized by using complex set of oligos.
Genetic association of interferon-alpha subtypes 1, 2 and 5 in systemic lupus erythematosus.
Avihingsanon et al., Bangkok, Thailand. In Tissue Antigens, 2008
This is the first report of positive association of IFNA gene in SLE, especially the role of specific subtypes IFNA1 and IFNA5.
Distinct signature type I interferon responses are determined by the infecting virus and the target cell.
Fish et al., Toronto, Canada. In Antivir Ther, 2007
Infecting virus and the target cell type dictate the extent and signature of inducible IFN-alpha5 gene expression.
Genetic susceptibility to respiratory syncytial virus bronchiolitis is predominantly associated with innate immune genes.
Hoebee et al., Netherlands. In J Infect Dis, 2007
RESULTS: SNPs in the innate immune genes VDR (rs10735810; P=.0017), JUN (rs11688; P=.0093), IFNA5 (rs10757212; P=.0093), and NOS2 (rs1060826; P=.0031) demonstrated the strongest association with bronchiolitis.
Differential activities of alpha/beta IFN subtypes against influenza virus in vivo and enhancement of specific immune responses in DNA vaccinated mice expressing haemagglutinin and nucleoprotein.
Bartlett et al., Perth, Australia. In Vaccine, 2007
Differences in antiviral efficacy were found among the subtypes with IFNA5 and IFNA6 being most effective, while IFNA1 was the least effective in reducing lung virus replication.
A pilot trial of combination cisplatin, 5-fluorouracil and interferon-alpha in the treatment of advanced esophageal carcinoma.
Al Fadda et al., Riyadh, Saudi Arabia. In Chemotherapy, 2002
Patients received cisplatin 80 mg/m(2) on day 1, 5-FU 750 mg/m(2)/day by continuous intravenous infusion for 5 days, and interferon-alpha 5 x 10(6) units/m(2)/day by subcutaneous injection on days 1-5 of each cycle.
Type I interferon gene therapy protects against cytomegalovirus-induced myocarditis.
James et al., Perth, Australia. In Immunology, 2002
IFNA6 treatment reduced MCMV replication whilst IFNA5 and A2 enhanced virus replication.
Expression of interferon-alpha subtypes in peripheral mononuclear cells from patients with chronic hepatitis C: a role for interferon-alpha5.
Prieto et al., Pamplona, Spain. In J Viral Hepat, 2001
Interferon (IFN)-alpha is a family of antiviral proteins encoded by different genes.
Human CD34+ hematopoietic cells transduced by retrovirus-mediated interferon alpha gene maintains regeneration capacity and engraftment in NOD/SCID mice.
Ahmed et al., Valhalla, United States. In Exp Hematol, 1999
To achieve long-term expression of human interferon alpha-5 (IFNalpha) gene in the bone marrow (BM) hematopoietic microenvironment, replication-deficient retroviral vector LSN-IFNalpha was used to deliver the IFNalpha gene into human BM CD34+ cells.
The interferon gene cluster: a candidate region for MS predisposition? Multiple Sclerosis Study Group.
Hardt et al., Bochum, Germany. In Genes Immun, 1999
In addition, LD analysis between IFNA10 [Cys20Stop], IFNA17 [58Stop] and the IFNA microsatellite alleles provides evidence for IFNA14, IFNA16 or IFNA5 as additional, most likely candidate genes.
Alpha interferon suppresses the cyclin D3 and cdc25A genes, leading to a reversible G0-like arrest.
Kimchi et al., Israel. In Mol Cell Biol, 1996
Alpha interferon suppresses the cyclin D3 and cdc25A genes, leading to a reversible G0-like arrest.
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