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Olfactory receptor 49

Olfactory receptors interact with odorant molecules in the nose, to initiate a neuronal response that triggers the perception of a smell. The olfactory receptor proteins are members of a large family of G-protein-coupled receptors (GPCR) arising from single coding-exon genes. Olfactory receptors share a 7-transmembrane domain structure with many neurotransmitter and hormone receptors and are responsible for the recognition and G protein-mediated transduction of odorant signals. The olfactory receptor gene family is the largest in the genome. The nomenclature assigned to the olfactory receptor genes and proteins for this organism is independent of other organisms. [provided by RefSeq, Jul 2008] (from NCBI)
Top mentioned proteins: HAD, CAN, SET, caspase-3, CD55
Papers on IC6
Collagen fragment biomarkers as serological biomarkers of lean body mass - a biomarker pilot study from the DAHANCA25B cohort and matched controls.
Lønbro et al., Denmark. In J Cachexia Sarcopenia Muscle, Dec 2015
Serum from blood was analyzed for the ProC3, IC6, and C6M peptide biomarkers and LBM were derived from the dual X-ray absorptiometry scans.
Type VI collagen turnover-related peptides-novel serological biomarkers of muscle mass and anabolic response to loading in young men.
Suetta et al., Copenhagen, Denmark. In J Cachexia Sarcopenia Muscle, 2013
METHODS: In order to test this hypothesis, we set out to develop an ELISA assay against an type VI collagen N-terminal globular domain epitope (IC6) and measured the levels of IC6 and an MMP-generated degradation fragment of collagen 6, (C6M) in a human immobilization-remobilization study setup with young (n = 11) and old (n = 9) men.
Computed tomography evaluation of different chest tube sites for residual pleural volumes after coronary artery bypass surgery.
Yekeler et al., İstanbul, Turkey. In Ann Saudi Med, 2011
The patients were separated into three groups: In one group (IC6, n=20), pleural tubes were inserted through the sixth intercostal space at the midaxillary line; in the second group (SX-r, n=20), rigid straight pleural tubes were inserted from the mediastinum through the subxiphoid area; and in the third group (SX-s, n=20), soft curved drainage tubes were inserted from the mediastinum through the subxiphoid area.
MYC activation associated with the integration of HPV DNA at the MYC locus in genital tumors.
Sastre-Garau et al., Paris, France. In Oncogene, 2006
Using in situ hybridization, HPV16 or 18 sequences were found at chromosome band 8q24, the localization of MYC, in IC1, IC2, IC3, IC6 and CAC-1 cells and at other sites in IC4, IC5, IC7 and IC8 cells.
[Analysis of antigenic determinant profiles of the Ebola virus VP35 protein N-terminal region using its short recombinant fragments].
Netesov et al., In Mol Gen Mikrobiol Virusol, 2002
Five short affinity-purified fragments of the EV VP35 protein were analyzed, by using the methods of IEA and immunoblotting, with polyclonal antiviral sera (PAS) against EV and with hybrid monoclonal antibodies (Mabs) IC6 and 6F7 specific to EV VP35 protein.
Presence of human chromosome 1 with expression of human decay-accelerating factor (DAF) prevents lysis of mouse/human hybrid cells by human complement.
White et al., Cambridge, United Kingdom. In Scand J Immunol, 1991
This effect could be abrogated by addition of anti-DAF monoclonal antibody (IC6).
Heterogeneity of expression and secretion of native and mutant [AspB10]insulin in AtT20 cells.
Halban et al., Boston, United States. In Mol Endocrinol, 1991
A second line (IC6) expressing the same mutant gene at much higher levels appeared to direct all mutant proinsulin to the regulated pathway, suggesting that for this particular mutant proinsulin, the secretory pathway employed by the transfected cells can be affected by the amount of proinsulin synthesized.
Decay-accelerating factor regulates complement-mediated damage in the human atherosclerotic wall.
Vlaicu et al., Cluj-Napoca / Kolozsvár, Romania. In Immunol Lett, 1990
Using two monoclonal antibodies, IC6 and IA10, DAF was localized by immunohistochemistry using streptavidin-biotin-peroxidase complex or silver-intensified immunogold techniques in aortic, iliac and femoral samples obtained at surgery and autopsy from 32 patients.
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