Papers using
I-kappa B Kinase
antibodies
Papers on
I-kappa B Kinase
A Novel Synthetic Compound, YH-1118, Inhibited LPS-Induced Inflammatory Response by Suppressing IkappaB Kinase/NF-kappaB Pathway in Raw 264.7 Cells.Lee et al., Inch'ŏn, South Korea. In J Microbiol Biotechnol, Jul 2015
For the search of a potent first-in-class compound to inactivate macrophages responsible for inflammatory responses, in the present study, we investigated the anti-inflammatory effects of YH-1118, a novel synthetic compound, in a lipopolysaccharide (LPS)-stimulated mouse macrophage cell line, Raw 264.7.
[Battle with herpes for 37 years].Shimomura, In Nihon Ganka Gakkai Zasshi, Mar 2015
IkappaB kinase-β (IKK2) inhibitors, which inhibit the activity of NF-κB, were used to examine gene expression during HSV reactivation in a mouse model.
Platelet-activating factor: a role in preterm delivery and an essential interaction with Toll-like receptor signaling in mice.Hirsch et al., Chicago, United States. In Biol Reprod, 2014
In peritoneal macrophages, the PTAFR agonist carbamyl PAF induces production of inflammatory markers previously demonstrated to be upregulated during bacterially induced labor, including: inducible nitric oxide synthase (Nos2), the chemokine Ccl5 (RANTES), tumor necrosis factor (Tnf), and level of their end-products (NO, CCL5, TNF) in a process dependent upon both IkappaB kinase and calcium/calmodulin-dependent protein kinase II.
Oncogenic activation of NF-kappaB.Staudt, Bethesda, United States. In Cold Spring Harb Perspect Biol, 2010
Many genetic aberrations in lymphomas alter CARD11, MALT1, or BCL10, which constitute a signaling complex that is intermediate between the B-cell receptor and IkappaB kinase.
NF-kappaB signaling: a tale of two pathways in skeletal myogenesis.Guttridge et al., Columbus, United States. In Physiol Rev, 2010
Activation of NF-kappaB is controlled by an IkappaB kinase (IKK) complex that can direct either canonical (classical) NF-kappaB signaling by degrading the IkappaB inhibitor and releasing p65/p50 dimers to the nucleus, or causes p100 processing and nuclear translocation of RelB/p52 via a noncanonical (alternative) pathway.