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GoPubMed Proteins lists recent and important papers and reviews for proteins. Page last changed on 19 Dec 2016.

Zinc finger protein 85

Top mentioned proteins: HAD, CAN, IgM, IgA, platelet factor 4
Papers on HPF4
Catastrophic APS in the context of other thrombotic microangiopathies.
Cervera et al., Barcelona, Spain. In Curr Rheumatol Rep, 2015
Thus, the differential diagnosis of an aPL-positive patient presenting with a microthrombotic storm is broad; the workup should include a special attention to signs of infection and disseminated malignant disease, assessing the funduscopic signs of malignant hypertension, testing ADAMTS13 activity and anti-heparin-platelet factor 4 (HPF4) antibodies, and searching previous exposure to certain drugs.
[Expressions of hypoxia inducible factor 1 alpha and P53 in the epididymis of varicocele rats].
Li et al., In Zhonghua Nan Ke Xue, 2014
23+/- 2. 23] % ) (both P <0.05) , while the expressions of HTF-1 a and p53 were remarkably higher in A (1.74 +/- 0. 16 and 1.71 +/- 0. 11) than in B (0.32 +/- 0. 08 and 0.56 +/- 0.13) and C (0.12 +/- 0. 03 and 0.25 +/-0.06)
Heparin: induced thrombocytopenia: incidence and laboratory approach to diagnosis in Indians.
Luthra et al., Delhi, India. In Indian J Pathol Microbiol, 2014
ELISA tests were positive in 21 cases (17%) which demonstrated the presence of anti-HPF4 antibodies in non-HIT cases as well.
Ex vivo effects of low-dose rivaroxaban on specific coagulation assays and coagulation factor activities in patients under real life conditions.
Lindhoff-Last et al., Frankfurt am Main, Germany. In Thromb Haemost, 2013
Plasma samples from 40 patients receiving rivaroxaban 10 mg daily were investigated to measure activities of clotting factor II, V, VII, VIII, IX, XI, XII and XIII; protein C- and protein S-levels; lupus anticoagulants; anticardiolipin IgG and IgM; D-dimer, heparin-platelet factor 4 (HPF4) antibodies and screening tests for von Willebrand disease (VWD).
Differential ability of tissue factor antibody clones on detection of tissue factor in blood cells and microparticles.
Hansen et al., Tromsø, Norway. In Thromb Res, 2012
MATERIALS AND METHODS: We studied TF expression on platelets, monocytes, lymphocytes and microparticles (MPs) by flow cytometry (FCM) with five commercially available mouse anti-human TF antibodies (HTF-1, TF9-10H10, CLB/TF-5, VIC7 and VD8).
A conserved homeobox transcription factor Htf1 is required for phialide development and conidiogenesis in Fusarium species.
Wang et al., Fuzhou, China. In Plos One, 2011
We previously showed that Htf1, a homeobox transcription factor, is required for conidiogenesis in the rice pathogen Magnaporthe oryzae.
A homeobox gene is essential for conidiogenesis of the rice blast fungus Magnaporthe oryzae.
Wang et al., Fuzhou, China. In Mol Plant Microbe Interact, 2010
Through targeted gene replacement, we find that deletion of HTF1, one of seven homeobox genes in the fungal genome, does not affect mycelial growth but causes total defect of conidial production.
Plasmapheresis and heparin reexposure as a management strategy for cardiac surgical patients with heparin-induced thrombocytopenia.
Arepally et al., Durham, United States. In Anesth Analg, 2010
METHODS: A retrospective chart review between November 2004 and March 2008 retrieved perioperative clinical and laboratory data for 11 adult cardiac surgical patients with a preoperative history of HIT and a current positive antiheparin/platelet factor 4 (anti-HPF4) antibody titer, who were managed with plasmapheresis and heparin anticoagulation.
Mining Clinical Data using Minimal Predictive Rules.
Hauskrecht et al., Pittsburgh, United States. In Amia Annu Symp Proc, 2009
We develop an algorithm to efficiently mine these MPRs and apply it to predict Heparin Platelet Factor 4 antibody (HPF4) test orders from electronic health records.
Compliance with platelet count monitoring recommendations and management of possible heparin-induced thrombocytopenia in hospitalized patients receiving low-molecular-weight heparin.
van Solinge et al., Utrecht, Netherlands. In Ann Pharmacother, 2009
Testing for the presence of heparin-platelet factor 4 antibodies (HPF4-Ab) and starting alternative anticoagulation is recommended when HIT is suspected.
A temporal abstraction framework for classifying clinical temporal data.
Hauskrecht et al., Pittsburgh, United States. In Amia Annu Symp Proc, 2008
We apply our approach to predict HPF4 test orders from electronic patient health records.
Conditional anomaly detection methods for patient-management alert systems.
Hauskrecht et al., In Proc Int Conf Mach Learn, 2008
We show the benefits of the instance-based methods on two real-world detection problems: detection of unusual admission decisions for patients with the community-acquired pneumonia and detection of unusual orders of an HPF4 test that is used to confirm Heparin induced thrombocytopenia - a life-threatening condition caused by the Heparin therapy.
Heparin-induced thrombocytopenia: impact of bovine versus porcine heparin in HIT pathogenesis.
Ahmad, Orlando, United States. In Front Biosci, 2006
HPF4 antibodies may cause platelet/endothelial cell activation to promote HIT pathogenesis.
Rapid detection of heparin-induced platelet antibodies with particle gel immunoassay (ID-HPF4)
Schwind et al., In Lancet, 1999
Early and acute diagnosis is needed to prevent thromboembolic complications in heparin-induced thrombocytopenia.
Generation and pathogenicity of anti-platelet factor 4 antibodies: diagnostic implications.
Vissac et al., In Clin Appl Thromb Hemost, 1999
Generation and expression of the autoantigen, HPF4 complexes, is a key feature for triggering antibody binding and pathogenicity.
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