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General transcription factor IIB

putative subunit of RNA polymerase II [RGD, Feb 2006] (from NCBI)
Top mentioned proteins: POLYMERASE, HAD, SBF, xVDR, EWS
Papers on GTF2B
Yin yang 1 and adipogenic gene network expression in longissimus muscle of beef cattle in response to nutritional management.
Loor et al., Urbana, United States. In Gene Regul Syst Bio, 2012
The objective of this study was to examine the effect of nutritional management on mRNA expression of YY1 along with its targets genes PPARG, GTF2B, KAT2B, IGFBP5 and STAT5B.
Replication study of 10 genes showing evidence for association with multiple sclerosis: validation of TMEM39A, IL12B and CBLB [correction of CLBL] genes.
Urcelay et al., Spain. In Mult Scler, 2012
RESULTS: Replication extended to the following polymorphisms: PKN2 (rs305217), GTF2B (rs7538427), EPHA4 (rs1517440), YTHDF3 (rs12115114), ANKFN1 (rs17758761) and PTPRM (rs4798571), which did not reach the threshold of significance in a follow-up of the first genome-wide association study (GWAS) conducted in multiple sclerosis; TMEM39A (rs1132200), which appeared as a newly identified susceptibility gene in the same study; a gene previously reaching GWAS significance in Italy, CBLB (rs9657904); IL12B (rs6887695, rs10045431), a susceptibility gene shared by diverse autoimmune diseases and, finally, another gene showing inconclusive association with multiple sclerosis, CNR1 (rs1049353).
Genomic study of RNA polymerase II and III SNAPc-bound promoters reveals a gene transcribed by both enzymes and a broad use of common activators.
Hernandez et al., Lausanne, Switzerland. In Plos Genet, 2011
To define the set of active SNAP(c)-dependent promoters in human cells, we have localized genome-wide four SNAP(c) subunits, GTF2B (TFIIB), BRF2, pol II, and pol III.
Gene expression of AGS cells stimulated with released proteins by Helicobacter pylori.
Song et al., South Korea. In J Gastroenterol Hepatol, 2008
Out of the selected 40 up- and five downregulated genes, 29 upregulated genes classified as general RNA polymerase II transcription factor activity (GTF2B, PPARGC1A), SH3/SH2 adaptor activity (CRKL), transferase activity (ACLY, CRKL, PIGC, PLK4), and oxidoreductase activity (IDH1) were confirmed to be upregulated by released proteins and not by H. pylori adhesion by real-time reverse transcription-polymerase chain reaction.
Involvement of GTA protein NC2beta in neuroblastoma pathogenesis suggests that it physiologically participates in the regulation of cell proliferation.
Purrello et al., Catania, Italy. In Mol Cancer, 2007
respectively) were also reduced, whereas we didn't detect any quantitative alteration of the mRNAs from GTF2B and NC2alpha (localized at 1p22-p21 and 11q13.3,
Localization of TFIIB binding regions using serial analysis of chromatin occupancy.
McWeeney et al., Portland, United States. In Bmc Mol Biol, 2006
Findings show that TFIIB binding is not restricted to the 5' upstream region indicates that the propensity for pre-initiation complex to contribute to transcript diversity is far greater than previously appreciated.
Deletion mapping suggests that the 1p22 melanoma susceptibility gene is a tumor suppressor localized to a 9-Mb interval.
Hayward et al., Brisbane, Australia. In Genes Chromosomes Cancer, 2004
The candidate genes PRKCL2 and GTF2B, within SRO2, and TGFBR3, CDC7, and EVI5, in a broad region encompassing SRO3, were screened in 1p22-linked melanoma kindreds, but no coding mutations were detected.
Genetic characterization of general transcription factors TFIIF and TFIIB of Homo sapiens sapiens.
Grzeschik et al., Catania, Italy. In Cytogenet Cell Genet, 1994
Analysis of loci GTF2F1 and GTF2B, encoding Rap 74 (a subunit of TFIIF) and TFIIB, respectively, showed that they are present in a single copy in the human genome and are localized at 19p13.3 and 1p22, respectively.
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