gopubmed logo
find other proteinsAll proteins
GoPubMed Proteins lists recent and important papers and reviews for proteins. Page last changed on 19 Dec 2016.

G1 to S phase transition 1

G1 to S phase transition, GSPT1, eRF3a
Top mentioned proteins: PCNA, CAN, p21, p27, CDK2
Papers on G1 to S phase transition
CD147 Overexpression Promotes Tumorigenicity in Chinese Hamster Ovary Cells.
Bian et al., Xi'an, China. In Cell Biol Int, Jan 2016
CD147 accelerated the G1 to S phase transition and enhanced the CHO cell proliferation.
miR-382 targeting PTEN-Akt axis promotes liver regeneration.
Yang et al., Shanghai, China. In Oncotarget, Jan 2016
miR-382 overexpression accelerated the proliferation and the G1 to S phase transition of the cell cycle both in mouse NCTC1469 and human HL7702 normal liver cells, while miR-382 downregulation had inverse effects.
MicroRNA-21 Contributes to Liver Regeneration by Targeting PTEN.
Yang et al., Shanghai, China. In Med Sci Monit, Dec 2015
Overexpression of miR-21 was associated with increased proliferation and a rapid G1-to-S phase transition of the cell cycle in BNL CL.2 normal liver cells in vitro.
Overexpressed CISD2 has prognostic value in human gastric cancer and promotes gastric cancer cell proliferation and tumorigenesis via AKT signaling pathway.
Zhang et al., Guangzhou, China. In Oncotarget, Dec 2015
Furthermore, we found that CISD2 affected cell proliferation and tumorigenicity of gastric cancer cells through mediating the G1-to-S phase transition.
Regulation of the retinoblastoma-E2F pathway by the ubiquitin-proteasome system.
Henry et al., East Lansing, United States. In Biochim Biophys Acta, Oct 2015
The retinoblastoma tumor suppressor (RB) and its related family members p107 and p130 regulate cell proliferation through the transcriptional repression of genes involved in cellular G1 to S phase transition.
gga-miR-101-3p Plays a Key Role in Mycoplasma gallisepticum (HS Strain) Infection of Chicken.
Peng et al., Wuhan, China. In Int J Mol Sci, 2014
Over-expression of gga-miR-101-3p significantly inhibited EZH2 expression and hence inhibited proliferation of chicken embryonic fibroblast (DF-1 cells) by blocking the G1-to-S phase transition.
Insulin-like growth factor binding protein-3 is a new predictor of radiosensitivity on esophageal squamous cell carcinoma.
Liu et al., Guangzhou, China. In Sci Rep, 2014
Downregulation of P21 and P27, and upregulation of p-P27 (phospho-Thr187), cyclin-dependent kinase 2 (CDK2), and cyclin E1 might contribute to the G0/G1 to S phase transition promoted by IGFBP-3.
Glucose and glutamine metabolism control by APC and SCF during the G1-to-S phase transition of the cell cycle.
Abarca-Rojano et al., Mexico. In J Physiol Biochem, 2014
Recent studies have given us a clue as to how modulations of both metabolic pathways and cyclins by the ubiquitin system influence cell cycle progression.
Control of cell cycle transcription during G1 and S phases.
de Bruin et al., London, United Kingdom. In Nat Rev Mol Cell Biol, 2013
During G1 phase, growth-dependent cyclin-dependent kinase (CDK) activity promotes DNA replication and initiates G1-to-S phase transition.
The patterns and dynamics of genomic instability in metastatic pancreatic cancer.
Futreal et al., Sanger, United States. In Nature, 2010
We find that pancreatic cancer acquires rearrangements indicative of telomere dysfunction and abnormal cell-cycle control, namely dysregulated G1-to-S-phase transition with intact G2-M checkpoint.
Cell cycle proteins in epithelial cell differentiation: implications for breast cancer.
Musgrove et al., Sydney, Australia. In Cell Cycle, 2010
A subset of the G1 to S phase transition proteins, D-type cyclins, Rb family proteins and the CDK inhibitors, are particularly involved in the commitment to differentiation.
Differential expression of GSPT1 GGCn alleles in cancer.
Brito et al., Lisbon, Portugal. In Cancer Genet Cytogenet, 2009
GSPT1 GGC(12) allele was present in 2.2% of colorectal cancer patients but was absent in Crohn disease patients and in the control group
Molecular basis of eRF3 recognition by the MLLE domain of poly(A)-binding protein.
Gehring et al., Montréal, Canada. In Plos One, 2009
crystal structures of the MLLE domain from PABPC1 in complex with the two PAM2 regions of eRF3
eRF3a/GSPT1 12-GGC allele increases the susceptibility for breast cancer development.
Brito et al., Lisbon, Portugal. In Oncol Rep, 2009
eRF3a/GSPT1 12-GGC allele increases the susceptibility for breast cancer development.
Prefrontal cortex shotgun proteome analysis reveals altered calcium homeostasis and immune system imbalance in schizophrenia.
Turck et al., São Paulo, Brazil. In Eur Arch Psychiatry Clin Neurosci, 2009
Using shotgun mass spectrometry, we found this protein differentially expressed in the dorsolateral prefrontal cortex from patients with schizophrenia.
G1 to S phase cell cycle transition in somatic and embryonic stem cells.
Lako et al., Newcastle upon Tyne, United Kingdom. In J Anat, 2008
It is well known that G1 to S phase transition is tightly regulated by the expression and phosphorylation of a number of well-characterized cyclins, cyclin-dependent kinases and members of the retinoblastoma gene family.
Proteasomal degradation of human release factor eRF3a regulates translation termination complex formation.
Jean-Jean et al., Paris, France. In Rna, 2008
eRF3a is degraded by the proteasome when not associated with eRF1; proteasomal degradation of eRF3a controls translation termination complex formation by adjusting the eRF3a level to that of eRF1.
tRNA traffic meets a cell-cycle checkpoint.
Hopper et al., Tucson, United States. In Cell, 2007
Findings by Ghavidel et al. (2007) reported in this issue suggest that tRNA trafficking and the transcription factor Gcn4 are key intermediates in the process by which yeast cells detect DNA damage and delay cell-cycle progression at the G1 to S phase transition.
Rb function in cell-cycle regulation and apoptosis.
Dean et al., Saint Louis, United States. In Nat Cell Biol, 2000
One regulator of the critical G1 to S-phase transition in the cell cycle is the retinoblastoma tumour suppressor protein Rb, which interacts with the E2F family of cell-cycle transcription factors to repress gene transcription required for this transition.
CUL-2 is required for the G1-to-S-phase transition and mitotic chromosome condensation in Caenorhabditis elegans.
Kipreos et al., Athens, United States. In Nat Cell Biol, 1999
Here we show that, in Caenorhabditis elegans, cul-2 is expressed in proliferating cells and is required at two distinct points in the cell cycle, the G1-to-S-phase transition and mitosis.
share on facebooktweetadd +1mail to friends