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GoPubMed Proteins lists recent and important papers and reviews for proteins. Page last changed on 19 Dec 2016.

Zinc finger protein, multitype 1

FOG, FOG-1, Friend of GATA-1, Friend of GATA
Top mentioned proteins: GATA-1, CAN, GATA4, HAD, FasT
Papers on FOG
Genomic Analyses of Sperm Fate Regulator Targets Reveal a Common Set of Oogenic mRNAs in Caenorhabditis elegans.
Kimble et al., Madison, United States. In Genetics, Jan 2016
In Caenorhabditis elegans, the FOG-1 and FOG-3 proteins behave genetically as terminal regulators of sperm fate specification.
The V81M variant of tyrosine hydroxylase is associated with more severe freezing of gait in Parkinson's disease.
Vrana et al., United States. In Parkinsonism Relat Disord, Jan 2016
RESULTS: PD subjects homozygous for the common V81M polymorphism, have higher overall freezing of gait scores after controlling for disease duration, although this polymorphism does not associate with the occurrence of PD or FOG.
Erythropoietin and IGF-1 signaling synchronize cell proliferation and maturation during erythropoiesis.
Chretien et al., Fontenay-aux-Roses, France. In Genes Dev, Jan 2016
Erythropoietin controls the proliferation and survival of red blood cell precursors, while variations in GATA-1/FOG-1 complex composition and concentrations drive their maturation.
Small molecules enhance functional O-mannosylation of Alpha-dystroglycan.
Wu et al., Shanghai, China. In Bioorg Med Chem, Jan 2016
Enhancing functional O-mannosyl glycans (FOG) of α-DG on the cell surfaces is a potential approach to address this unmet challenge.
Freezing of Gait in Parkinsonism and its Potential Drug Treatment.
Wang et al., Nanjing, China. In Curr Neuropharmacol, Jan 2016
Studies of treatment for FOG are scarce.
Erythropoiesis in vertebrates: from ontogeny to clinical relevance.
Fock et al., São Paulo, Brazil. In Front Biosci (elite Ed), Dec 2015
Several transcription factors and cytokines, such as GATA-1, GATA-2, FOG-1 and erythropoietin (EPO), constitute an elaborated molecular network that regulates erythropoiesis as they are involved in the differentiation and maturation of RBCs.
Genome-wide association analyses based on whole-genome sequencing in Sardinia provide insights into regulation of hemoglobin levels.
Cucca et al., Cagliari, Italy. In Nat Genet, Nov 2015
Five signals are due to variants at previously undetected loci: MPHOSPH9, PLTP-PCIF1, ZFPM1 (FOG1), NFIX and CCND3.
Fibromyalgia: the prototypical central sensitivity syndrome.
Boomershine, Brentwood, United States. In Curr Rheumatol Rev, 2014
The core FM symptom domains can be recalled using the FIBRO mnemonic and include Fatigue and Fog (cognitive dysfunction), Insomnia (difficulties with all aspects of sleep including initiation, maintenance and restorative), Blues (depression and anxiety), Rigidity (stiffness in muscles and joints) and Ow! (widespread pain and tenderness).
Signal transduction pathways, intrinsic regulators, and the control of cell fate choice.
Fossett, Baltimore, United States. In Biochim Biophys Acta, 2013
Specifically, the interface between the GATA:FOG regulatory complex and the JAK/STAT, BMP, and Hedgehog pathways is examined.
Combinatorial regulation of tissue specification by GATA and FOG factors.
Crispino et al., Chicago, United States. In Development, 2012
GATA family transcriptional regulators and their dedicated co-factors, termed Friend of GATA (FOG) proteins, control cell fate and differentiation in multiple tissue types from Drosophila to man.
Tissue-specific mitotic bookmarking by hematopoietic transcription factor GATA1.
Blobel et al., Philadelphia, United States. In Cell, 2012
The GATA1 coregulators FOG1 and TAL1 dissociate from mitotic chromatin, suggesting that GATA1 functions as platform for their postmitotic recruitment.
FOG-1 and GATA-1 act sequentially to specify definitive megakaryocytic and erythroid progenitors.
Nerlov et al., Monterotondo, Italy. In Embo J, 2012
FOG-1 is required for the formation of all committed Mk- and E-lineage progenitors. It mediates transcriptional megakaryocyte/erythrocyte programming of HSCs as well as their subsequent Mk/E-lineage commitment.
Pleiotropic platelet defects in mice with disrupted FOG1-NuRD interaction.
Poncz et al., Philadelphia, United States. In Blood, 2012
Disruption of the GATA1/FOG1/NuRD transcriptional system results in a complex, pleiotropic platelet defect beyond gray platelet syndrome-like macrothrombocytopenia.
Friend of GATA (FOG) interacts with the nucleosome remodeling and deacetylase complex (NuRD) to support primitive erythropoiesis in Xenopus laevis.
Christian et al., Portland, United States. In Plos One, 2011
a requirement for FOG and its interaction with NuRD during primitive erythropoiesis
Inherited thrombocytopenia due to GATA-1 mutations.
Kacena et al., Indianapolis, United States. In Semin Thromb Hemost, 2011
A wide variety of mutations in GATA-1 affect its function, as well as its interaction with its cofactors (especially Friend of GATA) and the genes upon which GATA-1 acts.
Insights into association of the NuRD complex with FOG-1 from the crystal structure of an RbAp48·FOG-1 complex.
Mackay et al., Cambridge, United Kingdom. In J Biol Chem, 2011
The FOG-1 peptide contacts a negatively charged binding pocket on top of the RbAp48 beta-propeller that is distinct from the binding surface used by RpAp48 to contact histone H4
Zinc finger protein, multitype 1, suppresses human Th2 development via downregulation of IL-4.
Kaminuma et al., Sagamihara, Japan. In Int Arch Allergy Immunol, 2010
ZFPM1 that facilitates human Th1 differentiation via the downregulation of IL-4 is a potential target for the treatment of allergic diseases.
GATA-1 converts lymphoid and myelomonocytic progenitors into the megakaryocyte/erythrocyte lineages.
Akashi et al., Boston, United States. In Immunity, 2003
GATA-1 upregulated critical MegE-related transcription factors such as FOG-1 and GATA-2 in lymphoid and GM progenitors, and their MegE development did not require "permissive" erythropoietin signals.
Acquired mutations in GATA1 in the megakaryoblastic leukemia of Down syndrome.
Crispino et al., Chicago, United States. In Nat Genet, 2002
We show that the shorter GATA1 protein, which lacks the N-terminal activation domain, binds DNA and interacts with its essential cofactor Friend of GATA1 (FOG1; encoded by ZFPM1) to the same extent as does full-length GATA1, but has a reduced transactivation potential.
FOG-2, a cofactor for GATA transcription factors, is essential for heart morphogenesis and development of coronary vessels from epicardium.
Orkin et al., Boston, United States. In Cell, 2000
We disrupted the FOG-2 gene in mice to define its requirement in vivo.
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