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GoPubMed Proteins lists recent and important papers and reviews for proteins. Page last changed on 19 Dec 2016.

Yip1 domain family, member 1

Top mentioned proteins: Ubiquitin, IRF, fibrillin-1, V1a, MURF3
Papers on FINGER1
Mahogunin ring finger 1 confers cytoprotection against mutant SOD1 aggresomes and is defective in an ALS mouse model.
Mishra et al., Jodhpur, India. In Neurobiol Dis, Feb 2016
Here, we found that the Mahogunin ring finger-1 (MGRN1) E3 ubiquitin ligase, which catalyzes mono-ubiquitination to the substrate, was dysregulated in the cellular and mouse models of ALS and that it preferentially interacted with various mutant forms of SOD1.
Leucine improves growth performance of intrauterine growth retardation piglets by modifying gene and protein expression related to protein synthesis.
Wang et al., Nanjing, China. In Nutrition, Jan 2016
The messenger RNA expression of muscle mammalian target of rapamycin (mTOR), muscle atrophy F-box (MAFbx), and muscle-specific ring finger-1 were investigated.
The role of E3 ubiquitin-ligases MuRF-1 and MAFbx in loss of skeletal muscle mass.
Reznick et al., Haifa, Israel. In Free Radic Biol Med, Jan 2016
Since their identification in 2001, the muscle specific E3s, muscle RING finger-1 (MuRF-1) and muscle atrophy F-box (MAFbx), have been shown to be implicated in the regulation of skeletal muscle atrophy in various pathological and physiological conditions.
DJ-1 protects against undernutrition-induced atrophy through inhibition of the MAPK-ubiquitin ligase pathway in myoblasts.
Kim et al., South Korea. In Life Sci, Jan 2016
The undernourished cells revealed an elevation in the expression of muscle-specific RING finger-1 (MuRF-1) and atrogin-1, and in the phosphorylations of p38 mitogen-activated protein kinase (MAPK) and stress-activated protein kinase/c-Jun N-terminal kinase compared with control groups.
Fenofibrate unexpectedly induces cardiac hypertrophy in mice lacking MuRF1.
Willis et al., Chapel Hill, United States. In Cardiovasc Pathol, Nov 2015
UNASSIGNED: The muscle-specific ubiquitin ligase muscle ring finger-1 (MuRF1) is critical in regulating both pathological and physiological cardiac hypertrophy in vivo.
The ubiquitin ligase MuRF1 regulates PPARα activity in the heart by enhancing nuclear export via monoubiquitination.
Willis et al., Chapel Hill, United States. In Mol Cell Endocrinol, Oct 2015
We report here for the first time an ubiquitin ligase (muscle ring finger-1/MuRF1) that inhibits fatty acid oxidation by inhibiting PPARα, but not PPARβ/δ or PPARγ in cardiomyocytes in vitro.
Molecular Changes in Sub-lesional Muscle Following Acute Phase of Spinal Cord Injury.
Banik et al., Charleston, United States. In Neurochem Res, Sep 2015
Western blot analyses performed on the visibly red, central portion of the gastrocnemius muscle showed significantly higher expression of muscle specific E3 ubiquitin ligases (muscle ring finger-1 and muscle atrophy f-box) and significantly lower expression of phosphorylated Akt-1/2/3 in the injury group compared to the sham group.
Angiotensin II Induces Skeletal Muscle Atrophy by Activating TFEB-Mediated MuRF1 Expression.
Fielitz et al., Berlin, Germany. In Circ Res, Sep 2015
Angiotensin (Ang) II induces skeletal muscle atrophy in part by increased muscle-enriched E3 ubiquitin ligase muscle RING-finger-1 (MuRF1) expression, which may involve protein kinase D1 (PKD1).
Muscle ring finger-3 protects against diabetic cardiomyopathy induced by a high fat diet.
Willis et al., Chapel Hill, United States. In Bmc Endocr Disord, 2014
We recently identified that muscle ring finger-1 (MuRF1) and MuRF2 differentially inhibit PPAR activities by mono-ubiquitination, leading to the hypothesis that MuRF3 may regulate PPAR activity in vivo to regulate DCM.
[Mechanism, diagnosis, and treatment of steroid myopathy].
Kohsaka, Tokyo, Japan. In Brain Nerve, 2013
Recent studies have shown that steroid-mediated induction of ubiquitin ligases (atrogin-1, muscle RING finger-1) and suppression of mammalian/mechanistic target of rapamycin cause an imbalance between anabolism and catabolism of muscle proteins, resulting in muscle atrophy.
Loss of the tumor suppressor Snf5 leads to aberrant activation of the Hedgehog-Gli pathway.
Dorsch et al., Cambridge, United States. In Nat Med, 2010
To investigate the mechanism by which glioma-associated oncogene family zinc finger-1 (GLI1), a crucial effector of Hh signaling, regulates Hh pathway activation, we searched for GLI1-interacting proteins.
Molecular regulation of skeletal muscle mass.
Russell, Australia. In Clin Exp Pharmacol Physiol, 2010
The present review focuses on the role and regulation of pathways involving Akt, atrogin-1 and muscle ring finger-1 (MuRF1; atrogenes), peroxisome proliferator-activated receptor-gamma coactivator-1alpha (PGC-1alpha) and striated activator of Rho signalling (STARS), with exercise and disease.
Cardiac muscle ring finger-1--friend or foe?
Patterson et al., Chapel Hill, United States. In Trends Cardiovasc Med, 2010
The ubiquitin ligase muscle ring finger-1 (MuRF1) binds creatine kinase, leading to its ubiquitination and possible degradation.
The ubiquitin-proteasome system in spongiform degenerative disorders.
Chin et al., Atlanta, United States. In Biochim Biophys Acta, 2008
The link between aberrant ubiquitination and spongiform neurodegeneration has been strengthened by the discovery that a null mutation in the E3 ubiquitin-protein ligase mahogunin ring finger-1 (Mgrn1) causes an autosomal recessively inherited form of spongiform neurodegeneration in animals.
Angiotensin II as candidate of cardiac cachexia.
Akao et al., New Orleans, United States. In Curr Opin Clin Nutr Metab Care, 2006
RECENT FINDINGS: In animals, angiotensin II produces weight loss through a pressor-independent mechanism, accompanied by decreased levels of circulating and skeletal muscle insulin-like growth factor-1 and increased mRNA levels of the ubiquitin ligases atrogin-1 and Muscle RING finger-1 in skeletal muscle.
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