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GoPubMed Proteins lists recent and important papers and reviews for proteins. Page last changed on 19 Dec 2016.

Fibrinogen beta chain

Fibrinopeptide B
Top mentioned proteins: Plasminogen, HAD, fibrillin-1, CAN, ACID
Papers on Fibrinopeptide B
Computer simulations of sample preconcentration in carrier-free systems and isoelectric focusing in microchannels using simple ampholytes.
Emmer et al., Stockholm, Sweden. In Electrophoresis, Oct 2015
Neurotensin and [Glu1]-Fibrinopeptide B were separated by IEF in microchannels featuring a liquid lid for simple sample handling and placement of the driving electrodes.
Simple setup for gas-phase H/D exchange mass spectrometry coupled to electron transfer dissociation and ion mobility for analysis of polypeptide structure on a liquid chromatographic time scale.
Rand et al., Copenhagen, Denmark. In Anal Chem, 2015
Comparison of the solution-phase- and gas-phase deuterium uptake of Leu-Enkephalin and Glu-Fibrinopeptide B, confirmed that this gas-phase HDX-MS approach allows for labeling of sites (heteroatom-bound non-amide hydrogens located on side-chains, N-terminus and C-terminus) not accessed by classical solution-phase HDX-MS.
Evidence for ion-ion interactions between peptides and anions (HSO₄⁻ or ClO₄⁻) derived from high-acidity acids.
Cole et al., New Orleans, United States. In J Mass Spectrom, 2014
The existence of gas-phase electrostatic ion-ion interactions between protonated sites on peptides ([Glu] Fibrinopeptide B, Angiotensin I and [Asn(1), Val(5)]-Angiotensin II) and attaching anions (ClO4(-) and HSO4(-)) derived from strong inorganic acids has been confirmed by CID MS/MS.
A new model for multiply charged adduct formation between peptides and anions in electrospray mass spectrometry.
Cole et al., New Orleans, United States. In J Am Soc Mass Spectrom, 2011
Evidence supporting the model is derived from the fact that for [Glu] Fibrinopeptide B, higher GB anions dominated in adducts observed at higher negative charge states, whereas lower GB anions appeared predominately in lower charge state adducts.
Alzheimer's disease peptide beta-amyloid interacts with fibrinogen and induces its oligomerization.
Strickland et al., New York City, United States. In Proc Natl Acad Sci U S A, 2011
Data suggest that the interaction between Abeta and fibrinogen may be an important contributor to the vascular abnormalities found in Alzheimer's disease.
The effects of the fibrin-derived peptide Bbeta(15-42) in acute and chronic rodent models of myocardial ischemia-reperfusion.
Petzelbauer et al., Düsseldorf, Germany. In Shock, 2007
fibrin-derived peptide Bbeta(15-42) has a role in models of myocardial ischemia-reperfusion
Fibrinogen and von Willebrand factor-independent platelet aggregation in vitro and in vivo.
Ni et al., Ottawa, Canada. In J Thromb Haemost, 2006
beta(3) integrin, thrombin, and Ca(2+) play critical roles in platelet aggregation without fibrinogen or VWF
Electron capture dissociation in a digital ion trap mass spectrometer.
Brancia et al., Manchester, United Kingdom. In Anal Chem, 2006
[Glu(1)]-Fibrinopeptide B and substance P were used to evaluate the performance of the current design.
Fibrin monomer and fibrinopeptide B act additively to increase DNA synthesis in smooth muscle cells cultured from human saphenous vein.
Powell et al., London, United Kingdom. In J Vasc Surg, 2001
DNA synthesis was measured by bromodeoxyuridine incorporation into DNA, cell proliferation by cell counting, cyclic adenosine monophosphate by enzyme-linked immunosorbent assay, and fibrinopeptide B labeled with iodine 125 used for binding studies.
Negative ion matrix-assisted laser desorption/ionization time-of-flight post-source decay calibration by using fibrinopeptide B.
Cassady et al., Oxford, United States. In J Am Soc Mass Spectrom, 1998
Using fibrinopeptide B as the calibrant, the negative ion PSD results for angiotensin II (M(r) 1046.2),
Altered coagulation in cerebral ischemia. Platelet, thrombin, and plasmin activity.
Francis et al., Los Angeles, United States. In Arch Neurol, 1990
Fibrinolytic activity was measured using assays for fibrinopeptide B-beta 1-42 and plasminogen activator inhibitor 1. Fibrinopeptide B-beta 1-42 was significantly reduced acutely (6.3 +/- 2.2 pmol/mL; n = 35) and at follow-up (4.8 +/- 1.5 pmol/mL; n = 21).
Role of fibrinopeptide B in early atherosclerotic lesion formation.
Zarins et al., Chicago, United States. In Am J Surg, 1990
Fibrinopeptide B (FPB) has been shown to be a potent chemotactic agent for macrophages, which are abundant as intimal foam cells in atherosclerotic lesions, especially in cholesterol-fed rabbits.
Increased fibrin/fibrinogen degradation products without increase of plasmin-alpha 2-plasmin inhibitor complex after hepatectomy for hepatocellular carcinoma.
Ohsato et al., Kitakyūshū, Japan. In Thromb Res, 1990
Fibrinopeptide B beta 15-42 (B beta 15-42) in the plasma also increased immediately after the peak of FPA, followed by a gradual decline in B beta 15-42 levels.
[Effect of remission induction chemotherapy on blood coagulability in patients with gynecological malignancies].
Oguri et al., In Nihon Gan Chiryo Gakkai Shi, 1989
In 12 patients (38 courses) with gynecological malignancies who had been treated with remission induction chemotherapy, we measured the levels of ESR, Platelet, PT, APTT, Hepa T, AT III, alpha 2 PI, FDP and D-dimer, and we also measured such molecular markers as Fibrinopeptide A (FPA), Fibrinopeptide B beta 1-42 (FPB beta 1-42), and Fibrinopeptide B beta 15-42 (FPB beta 15-42) before and after chemotherapy.
Conversion of fibrinogen to fibrin induced by preferential release of fibrinopeptide B.
Kornalík et al., Praha, Czech Republic. In Biochim Biophys Acta, 1989
Fibrin clot-promoting enzyme preferentially releasing fibrinopeptide B from fibrinogen was isolated from the crude venom of Agkistrodon contortrix and its mode of action was studied in detail.
Fibrinopeptide B in fibrin assembly and metabolism: physiologic significance in delayed release of the peptide.
Dardik et al., In Ann N Y Acad Sci, 1983
The delayed release of peptide B that accelerates towards the end of fibrin formation unmasks accessory (b-) epitopes for monomer interaction.
Measurement of desarginine fibrinopeptide B in human blood.
Auerbach et al., In J Clin Invest, 1981
First fibrinopeptide A and fibrin I are formed and then fibrinopeptide B (B beta 1-14) and fibrin II.
Fibrinopeptide B and aggregation of fibrinogen.
Dardik et al., In Science, 1979
Removal of fibrinopeptide B from human fibrinogen by reaction with the procoagulant enzyme from copperhead snake venom below 25 degrees C resulted in tight aggregation of the fibrinogen, which, in turn, progressively blocked a concomitant but sluggish release of fibrinopeptide A by the enzyme.
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