gopubmed logo
find other proteinsAll proteins
GoPubMed Proteins lists recent and important papers and reviews for proteins. Page last changed on 19 Dec 2016.

Ecotropic viral integration site 2B

EVI2B, D17S376
Top mentioned proteins: NF1, SUZ12, OMgp, CD45, centaurin-alpha2
Papers on EVI2B
EVI2B, ATP2A2, S100B, TM4SF3, and OLFM4 as potential prognostic markers for postoperative Taiwanese colorectal cancer patients.
Lin et al., Kao-hsiung, Taiwan. In Dna Cell Biol, 2012
Postoperative relapse was significantly correlated with gene overexpression, including EVI2B (p=0.001,
Integrated Analysis Reveals hsa-miR-142 as a Representative of a Lymphocyte-Specific Gene Expression and Methylation Signature.
Anastassiou et al., New York City, United States. In Cancer Inform, 2011
The set R consistently contains many genes, such as TRAF3IP3, NCKAP1L, CD53, LAPTM5, PTPRC, EVI2B, DOCK2, LCP2, CYBB and FYB.
New B-cell CD molecules.
Engel et al., Barcelona, Spain. In Immunol Lett, 2011
During the Ninth HLDA Workshop (HLDA9) eighteen new CDs were allocated to cell-surface molecules expressed on B cells: CD210a (IL10RA), CD215 (IL15RA), CD270 (TNFRSF14), CD307a (FCRL1), CD307b (FCRL2), CD307c (FCRL3), CD307d (FCRL4), CD351 (FCAMR), CD352 (SLAMF6), CD353 (SLAMF8), CD354 (TREM1), CD355 (CRTAM), CD357 (TNFRSF18), CD358 (TNFRSF21), CD360 (IL21RA), CD361 (EVI2B), CD362 (SDC2), and CD363 (S1PR1).
Complete physical map and gene content of the human NF1 tumor suppressor region in human and mouse.
Kehrer-Sawatzki et al., Martinsried, Germany. In Genes Chromosomes Cancer, 2003
Moreover, we completed the genomic organization and cDNA structure of all functional genes, CYTOR4, FLJ12735, FLJ22729, CENTA2, MGC13061, NF1, OMG, EVI2B, EVI2A, KIAA1821, MGC11316, HCA66, KIAA0160, and WI-12393, from this region.
Agouti signal protein regulation in human melanoma cells.
Van Daal et al., Brisbane, Australia. In Pigment Cell Res, 2003
Results identify five genes, namely PPARbeta, eIF-4B, RRM2, MINOR and EVI2B that are down-regulated by ASIP, indicating a likely role for ASIP in human melanogenesis.
Distinct gene expression profiling in chronic lymphocytic leukemia with 11q23 deletion.
Knuutila et al., Helsinki, Finland. In Leukemia, 2001
A cluster of genes, LCP1, PARP, BLR1, DEK, NPM, MCL1, SLP76, STAM, HIVEP1, EVI2B, CD25, HTLF, HIVEP2, BCL2, MNDA, PBX3, EB12, TCF1, CGRP, CD14, ILB, GZMK, GPR17 and CD79B, was associated (P < 0.05) with the unfavorable 11q deletion and also with the unfavorable Binet stages B and C. We present here gene expression profiling that is associated with CLL patients with the 11q23 deletion.
Molecular characterization and gene content of breakpoint boundaries in patients with neurofibromatosis type 1 with 17q11.2 microdeletions.
Kehrer-Sawatzki et al., Martinsried, Germany. In Am J Hum Genet, 2001
Since patients with the NF1 large-deletion syndrome have a significantly increased risk of neurofibroma development and mental retardation, hemizygosity for genes from the deleted region around the neurofibromin locus (CYTOR4, FLJ12735, FLJ22729, HSA272195 (centaurin-alpha2), NF1, OMGP, EVI2A, EVI2B, WI-9521, HSA272196, HCA66, KIAA0160, and WI-12393) may contribute to the severe phenotype of these patients.
A common set of at least 11 functional genes is lost in the majority of NF1 patients with gross deletions.
Thiel et al., Martinsried, Germany. In Genomics, 2000
Since D17S117 and D17S57 were previously reported as the outer limits for the great majority of NF1 deletions, we suggest that most NF1 patients with deletion of the entire NF1 gene are hemizygous for the same set of at least 10 additional genes, including SHGC-37343, SHGC-2390, SHGC-34232, OMG, EVI2B, EVI2A, WI-9521, WI-6742, SHGC-34334, and KIAA0160, and thus present with a relatively uniform clinical phenotype.
EVI2B, a gene lying in an intron of the neurofibromatosis type 1 (NF1) gene, is as the NF1 gene involved in differentiation of melanocytes and keratinocytes and is overexpressed in cells derived from NF1 neurofibromas.
Bartelt et al., Ulm, Germany. In Dna Cell Biol, 1999
The EVI2B gene is one of three genes embedded in intron 27b of the neurofibromatosis type 1 (NF1; M. Recklinghausen) gene, which are transcribed in the direction opposite that of the NF1 gene.
Genomic characterization of the Neurofibromatosis Type 1 gene of Fugu rubripes.
Krone et al., Ulm, Germany. In Gene, 1998
However, a single exon gene with similarity to the human EVI2B gene has been found on the reverse strand of Fugu intron 27b.
Deletions spanning the neurofibromatosis 1 gene: identification and phenotype of five patients.
Stephens et al., Seattle, United States. In Am J Hum Genet, 1994
Minimally, each of the deletions involved the entire 350-kb NF1 gene; the three genes--EVI2A, EVI2B, and OMG--that are contained within an NF1 intron; and considerable flanking DNA.
cDNA sequence and genomic structure of EV12B, a gene lying within an intron of the neurofibromatosis type 1 gene.
Culver et al., Salt Lake City, United States. In Genomics, 1991
Here we describe a second locus, EVI2B, also located between the two NF1 translocation breakpoints.
The gene encoding the oligodendrocyte-myelin glycoprotein is embedded within the neurofibromatosis type 1 gene.
White et al., Salt Lake City, United States. In Mol Cell Biol, 1991
OMGP is similar in genomic structure to two other expressed genes, EVI2A and EVI2B, which lie approximately 20 and 5 kb telomeric of the OMGP locus, respectively.
share on facebooktweetadd +1mail to friends