gopubmed logo
find other proteinsAll proteins
GoPubMed Proteins lists recent and important papers and reviews for proteins. Page last changed on 19 Dec 2016.

Erythrocyte membrane protein band 4.1 like 5

EPB41L5, YMO1, Epb4.1l5, Erythrocyte Protein Band 4.1-Like 5, NBL5, BE37
Top mentioned proteins: EHM2, Ezrin, Actin, NODAL, V1a
Papers on EPB41L5
A patient with a mild holoprosencephaly spectrum phenotype and heterotaxy and a 1.3 Mb deletion encompassing GLI2.
Lichtenbelt et al., Utrecht, Netherlands. In Am J Med Genet A, 2012
The deletion contains five genes, in addition to GLI2, including the EPB4.1l5
Genome-wide association scan identifies a risk locus for preeclampsia on 2q14, near the inhibin, beta B gene.
Moses et al., San Antonio, United States. In Plos One, 2011
We also explored the mRNA expression in decidua of genes ±500 kb of INHBB and found a nominally significant correlation between a transcript encoded by the EPB41L5 gene, ∼250 kb centromeric to INHBB, and preeclampsia (p = 0.03).
Dual roles of Notch in regulation of apically restricted mitosis and apicobasal polarity of neuroepithelial cells.
Okamoto et al., Wako, Japan. In Neuron, 2011
Data suggest that the Crb/Moe complex and Notch play roles in a positive feedback loop to maintain the apicobasal polarity in neuroepithelial cells.
Left-right patterning in the mouse requires Epb4.1l5-dependent morphogenesis of the node and midline.
Anderson et al., New York City, United States. In Dev Biol, 2010
Left-right patterning in the mouse requires Epb4.1l5-dependent morphogenesis of the node and midline
Epithelial cell shape is regulated by Lulu proteins via myosin-II.
Tanoue et al., Kōbe, Japan. In J Cell Sci, 2010
Lulu (Epb41l5) is a major regulator of morphogenesis, although the downstream molecular and cellular mechanisms remain obscure in mammals.
Changes in cortical cytoskeletal and extracellular matrix gene expression in prostate cancer are related to oncogenic ERG deregulation.
Engers et al., Düsseldorf, Germany. In Bmc Cancer, 2009
METHODS: Expression of EPB41L1, EPB41L2, EPB41L3 (protein: 4.1B), EPB41L4B (EHM2), EPB41L5, EPB49 (dematin), VIL2 (ezrin), and DLG1 (summarized as "cortical cytoskeleton" genes) as well as ERG was measured by quantitative RT-PCR in a well-characterized set of 45 M0 prostate adenocarcinoma and 13 benign tissues.
FERM proteins in animal morphogenesis.
Tepass, Toronto, Canada. In Curr Opin Genet Dev, 2009
This review will provide a brief overview of the FERM domain structure and the FERM protein superfamily and then discuss recent advances in our understanding of the mechanism of function and developmental requirement of several FERM proteins including Moesin, Myosin-VIIA, Myosin-XV, Coracle/Band4.1 as well as Yurt and its vertebrate homologs Mosaic Eyes and EPB41L5/YMO1/Limulus.
Yurt, Coracle, Neurexin IV and the Na(+),K(+)-ATPase form a novel group of epithelial polarity proteins.
Tepass et al., Toronto, Canada. In Nature, 2009
We also find that the mammalian Yrt orthologue EPB41L5 (also known as YMO1 and Limulus) is required for lateral membrane formation, indicating a conserved function of Yrt proteins in epithelial polarity.
EPB41L5 functions to post-transcriptionally regulate cadherin and integrin during epithelial-mesenchymal transition.
Aizawa et al., Kōbe, Japan. In J Cell Biol, 2008
Results describe the involvement of EPB41L5 in epithelial-mesenchymal transition during mouse gastrulation.
Tissue-specific requirements for specific domains in the FERM protein Moe/Epb4.1l5 during early zebrafish development.
Jensen et al., Amherst Center, United States. In Bmc Dev Biol, 2007
our data provide further evidence that Moe is a negative regulator of rod outer segment size
FERM protein EPB41L5 is a novel member of the mammalian CRB-MPP5 polarity complex.
Roepman et al., Nijmegen, Netherlands. In Exp Cell Res, 2007
the importance of a conserved Crumbs-MPP5-EPB41L5 polarity complex in mammals for separation of the apical and basolateral domains through specialized cell-cell junctions
The FERM protein Epb4.1l5 is required for organization of the neural plate and for the epithelial-mesenchymal transition at the primitive streak of the mouse embryo.
Anderson et al., New York City, United States. In Development, 2007
Lulu mutation in Epb4.1l5 helps anchor the actin-myosin contractile machinery to the membrane to allow the dynamic rearrangements of epithelia that mediate embryonic morphogenesis.
The FERM protein Yurt is a negative regulatory component of the Crumbs complex that controls epithelial polarity and apical membrane size.
Tepass et al., Toronto, Canada. In Dev Cell, 2006
We also show that the mammalian Yurt orthologs YMO1 and EHM2 bind to mammalian Crb proteins.
Isolation and screening of brittlestar-associated bacteria for antibacterial activity.
Lundie et al., United States. In Curr Microbiol, 2002
Forty-one of the isolates were capable of 20-100% inhibition of one or more of 19 subject bacteria at 10% salinity at 37 degrees C. Three isolates, BE37, BE52, and BE53, exhibited the greatest range of antibacterial activity at both 10% and 35% salinity.
Structural diversity of band 4.1 superfamily members.
Tsukita et al., Japan. In J Cell Sci, 1994
Sequence analyses of these clones revealed that the band 4.1 superfamily can be subdivided into 5 gene families; band 4.1 protein, ERM (ezrin/radixin/moesin/merlin/NBL6/NBL7+ ++), talin, PTPH1 (PTPH1/PTPMEG/NBL1-3), and NBL4 (NBL4/NBL5) families.
share on facebooktweetadd +1mail to friends