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Zinc finger, MYM-type 3

DXS6673E, MYM, ZNF261, ZNF258, KIAA0385, DXHXS6673E
This gene is located on the X chromosome and is subject to X inactivation. It is highly conserved in vertebrates and most abundantly expressed in the brain. The encoded protein is a component of histone deacetylase-containing multiprotein complexes that function through modifying chromatin structure to keep genes silent. A chromosomal translocation (X;13) involving this gene is associated with X-linked mental retardation. Several alternatively spliced transcript variants have been found for this gene. [provided by RefSeq, Jan 2010] (from NCBI)
Top mentioned proteins: ZNF198, FGFR1, HAD, ACID, BCR
Papers on DXS6673E
X-chromosomal inactivation directly influences the phenotypic manifestation of X-linked protoporphyria.
Di Pierro et al., Milano, Italy. In Clin Genet, Jan 2016
Methylation assays of the androgen receptor and zinc-finger MYM type 3 short tandem repeat polymorphisms estimated each heterozygotes X-chromosomal inactivation pattern.
X-chromosome inactivation in female newborns conceived by assisted reproductive technologies.
Ma et al., Vancouver, Canada. In Fertil Steril, 2014
MAIN OUTCOME MEASURE(S): XCI skewing values determined by assaying allelic ratio of methylated alleles at the androgen receptor (AR), fragile X mental retardation 1 (FMR1), and DXS6673E loci.
Characterization of the SUMO-binding activity of the myeloproliferative and mental retardation (MYM)-type zinc fingers in ZNF261 and ZNF198.
Matunis et al., Baltimore, United States. In Plos One, 2013
Four human proteins including ZNF261, ZNF198, ZNF262, and ZNF258 contain a stretch of tandem zinc fingers called myeloproliferative and mental retardation (MYM)-type zinc fingers.
XLID-causing mutations and associated genes challenged in light of data from large-scale human exome sequencing.
Mandel et al., Illkirch-Graffenstaden, France. In Am J Hum Genet, 2013
We also highlight 15 other genes (CCDC22, CLIC2, CNKSR2, FRMPD4, HCFC1, IGBP1, KIAA2022, KLF8, MAOA, NAA10, NLGN3, RPL10, SHROOM4, ZDHHC15, and ZNF261) for which replication studies are warranted.
Proteins ZNF198 and SUZ12 are down-regulated in hepatitis B virus (HBV) X protein-mediated hepatocyte transformation and in HBV replication.
Andrisani et al., West Lafayette, United States. In Hepatology, 2011
We identified by a genome-wide short hairpin RNA (shRNA) library screen the genes zinc finger, MYM-type 2 (ZNF198) and suppressor of zeste 12 homolog (Drosophila) (SUZ12) whose protein depletion rescues pX-expressing cells from DNA damage-induced apoptosis.
Myeloproliferative disorders with t(8;9)(p12;q33): a case report and review of the literature.
He et al., Suzhou, China. In Pediatr Hematol Oncol, 2011
The most frequent partner genes are in decreasing order of frequency: ZNF198 (or ZMYM2, zinc finger MYM type 2), CEP110 (centrosomal protein 110 kDa), FOP (or FGFR1OP, FGFR1 [fibroblast growth factor receptor 1] oncogene partner), and BCR (breakpoint cluster region) located on 13q12, 9q33, 6q27, and 22q11, respectively.
Crypton transposons: identification of new diverse families and ancient domestication events.
Jurka et al., Mountain View, United States. In Mob Dna, 2010
Genes containing the DUF3504 domain are potassium channel tetramerization domain containing 1 (KCTD1), KIAA1958, zinc finger MYM type 2 (ZMYM2), ZMYM3, ZMYM4, glutamine-rich protein 1 (QRICH1) and "without children" (WOC).
Association of extremely skewed X-chromosome inactivation with Taiwanese women presenting with recurrent pregnancy loss.
Teng et al., Tainan City, Taiwan. In J Formos Med Assoc, 2008
Human androgen receptor or DXS6673E and DX15-134 loci were used in the XCI assay.
ZNF198 stabilizes the LSD1-CoREST-HDAC1 complex on chromatin through its MYM-type zinc fingers.
Yu et al., Dallas, United States. In Plos One, 2007
ZNF198 is a member of a family of MYM-type zinc finger proteins that associate with LCH.
Analysis of transcriptional modulation of the presenilin 1 gene promoter by ZNF237, a candidate binding partner of the Ets transcription factor ERM.
Das et al., Fort Worth, United States. In Brain Res, 2007
One of the interacting proteins was ZNF237, a member of the MYM gene family.
The putative Drosophila transcription factor woc is required to prevent telomeric fusions.
Gatti et al., Roma, Italy. In Mol Cell, 2006
Woc is a Drosophila zinc finger protein that shares homology with the human polypeptides ZNF261 and ZNF198 implicated in mental retardation and leukemia syndromes.
First case report of X linked dystonia parkinsonism (XDP) or 'lubag' in Australia.
O'sullivan et al., Melbourne, Australia. In J Clin Neurosci, 2005
RESULTS: Linkage was confirmed using markers ZNF261, DXS10017, and DXS10018.
Glut 1 expression in transitional cell carcinoma of the urinary bladder is associated with poor patient survival.
Lerner et al., Houston, United States. In Anticancer Res, 2001
Using the polyclonal anti-Glut1 antibody MYM, microwave-aided antigen retrieval, and standard immunoperoxidase ABC technique, we immunostained sections of formalin-fixed and paraffin-embedded tissue from cystectomy specimens from 40 patients with TCCB, who received no adjuvant therapy.
Clinical significance of human erythrocyte glucose transporter 1 expression at the deepest invasive site of advanced colorectal carcinoma.
Shimamoto et al., Hiroshima, Japan. In Oncology, 2000
Glut1 expression was examined immunohistochemically with a labeled streptavidin-biotin kit using anti-Glut1 polyclonal antibody MYM.
Cloning of ZNF237, a novel member of the MYM gene family that maps to human chromosome 13q11-->q12.
Cross et al., London, United Kingdom. In Cytogenet Cell Genet, 1999
Two alternatively spliced regions were identified by RT-PCR; the major splice variant is predicted to encode a 383 amino acid protein that contains a single diverged MYM domain.
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