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DIP2 disco-interacting protein 2 homolog A

Dip2, disco-interacting protein 2, KIAA0184, C21orf106, Disco-interacting protein 2 homolog A, DIP2A
The protein encoded by this gene may be involved in axon patterning in the central nervous system. This gene is not highly expressed. Several transcript variants encoding different isoforms have been found for this gene. [provided by RefSeq, Mar 2009] (from NCBI)
Top mentioned proteins: DYX1C1, S-100, Robo1, SET, Ru2
Papers on Dip2
Genetic variant in DIP2A gene is associated with developmental dyslexia in Chinese population.
Song et al., Wuhan, China. In Am J Med Genet B Neuropsychiatr Genet, Nov 2015
DIP2A has been identified to be partially deleted on chromosome region 21q22.3,
Polymorphisms in DCDC2 and S100B associate with developmental dyslexia.
Kere et al., Huddinge, Sweden. In J Hum Genet, Jul 2015
We aimed at discovering single nucleotide variation present in known and new candidate genes for developmental dyslexia: CYP19A1, DCDC2, DIP2A, DYX1C1, GCFC2 (also known as C2orf3), KIAA0319, MRPL19, PCNT, PRMT2, ROBO1 and S100B.
Expression Patterns and Potential Biological Roles of Dip2a.
Zheng et al., Changchun, China. In Plos One, 2014
Disconnected (disco)-interacting protein 2 homolog A is a member of the DIP2 protein family encoded by Dip2a gene.
Follistatin-like 1 attenuates apoptosis via disco-interacting protein 2 homolog A/Akt pathway after middle cerebral artery occlusion in rats.
Zhang et al., Loma Linda, United States. In Stroke, 2014
We hypothesized that recombinant FSTL1 attenuates neuronal apoptosis through its receptor disco-interacting protein 2 homolog A (DIP2A) and the Akt pathway after middle cerebral artery occlusion (MCAO) in rats.
Alzheimer's disease: early alterations in brain DNA methylation at ANK1, BIN1, RHBDF2 and other loci.
Bennett et al., Boston, United States. In Nat Neurosci, 2014
Furthermore, we functionally validated these CpG associations and identified the nearby genes whose RNA expression was altered in AD: ANK1, CDH23, DIP2A, RHBDF2, RPL13, SERPINF1 and SERPINF2.
Identification of risk genes for autism spectrum disorder through copy number variation analysis in Austrian families.
Vincent et al., Toronto, Canada. In Neurogenetics, 2014
Twenty-two CNVs were validated from this set (five of which are apparently de novo), many of which appear likely to disrupt genes that may be considered as good candidates for neuropsychiatric disorders, including DLG2, S100B, ARX, DIP2A, HPCAL1, and GPHN.
A plant small polypeptide is a novel component of DNA-binding protein phosphatase 1-mediated resistance to plum pox virus in Arabidopsis.
Vera et al., Valencia, Spain. In Plant Physiol, 2011
Here, we report on the identification of a novel family of highly conserved small polypeptides that interact with DBP1 proteins both in tobacco and Arabidopsis, which we have designated DBP-interacting protein 2 (DIP2).
A theoretical molecular network for dyslexia: integrating available genetic findings.
Franke et al., Nijmegen, Netherlands. In Mol Psychiatry, 2011
We found that 10 of the 14 dyslexia candidate genes (ROBO1, KIAA0319, KIAA0319L, S100B, DOCK4, FMR1, DIP2A, GTF2I, DYX1C1 and DCDC2) fit into a theoretical molecular network involved in neuronal migration and neurite outgrowth.
DNA methylation patterns associate with genetic and gene expression variation in HapMap cell lines.
Pritchard et al., Chicago, United States. In Genome Biol, 2010
The most intriguing trans signal was obtained for SNP rs10876043 in the disco-interacting protein 2 homolog B gene (DIP2B, previously postulated to play a role in DNA methylation), that had a genome-wide significant association with the first principal component of patterns of methylation; however, we found only modest signal of trans-acting associations overall.
DIP2 disco-interacting protein 2 homolog A (Drosophila) is a candidate receptor for follistatin-related protein/follistatin-like 1--analysis of their binding with TGF-β superfamily proteins.
Mimori et al., Kanazawa, Japan. In Febs J, 2010
DIP2A could be a cell-surface receptor protein and mediate a FOS down-regulation signal of FRP. FRP bound to DIP2A and CD14, and also with proteins of the TGF-beta superfamily
DIP2A functions as a FSTL1 receptor.
Walsh et al., Boston, United States. In J Biol Chem, 2010
These data indicate that DIP2A functions as a novel receptor that mediates the cardiovascular protective effects of FSTL1.
Identification of novel dyslexia candidate genes through the analysis of a chromosomal deletion.
Schrander-Stumpel et al., Maastricht, Netherlands. In Am J Med Genet B Neuropsychiatr Genet, 2009
In this article, we report the identification of four new dyslexia candidate genes (PCNT, DIP2A, S100B, and PRMT2) on chromosome region 21q22.3
Exploring the nuclear proteome of Medicago truncatula at the switch towards seed filling.
Gallardo et al., Dijon, France. In Plant J, 2008
Some had already been shown to play a role during the early stages of seed formation whereas for others the findings are novel (e.g. the DIP2 and ES43 transcriptional regulators or the RNA silencing-related ARGONAUTE proteins).
Genetic complexity of EHV-1 defective interfering particles and identification of novel IR4/UL5 hybrid proteins produced during persistent infection.
O'Callaghan et al., Shreveport, United States. In Virus Genes, 2006
DIP2 and DIP3 contain a previously described unique IR4/UL5 (EICP22/EICP27) hybrid gene (Hyb1.0).
[Cell-cycle negative regulatory gene ANA is over-expressed in the brain tissues of patients with Down syndrome].
Guo et al., Beijing, China. In Beijing Da Xue Xue Bao, 2006
RESULTS: The expression levels of 6 genes in cortex and cerebellum, including DYRK1A, SYNJ1, PCP4, C21orf5, C21orf2 and C21orf106, were comparable between DS and the control.
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