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Dystroglycan 1

laminin-binding glycoprotein involved in linking the extracellular matrix to the actin cytoskeleton [RGD, Feb 2006] (from NCBI)
Top mentioned proteins: IFN-gamma, EGFR, Plasminogen, Akt, uPA
Papers on DAG I
Dystroglycan promotes filopodial formation and process branching in differentiating oligodendroglia.
Colognato et al., Stony Brook, United States. In J Neurochem, 2012
These results suggest that dystroglycan-Lm interactions influence oligodendroglial process dynamics and therefore may regulate the myelination capacity of individual oligodendroglia.
Dynamics of dystroglycan complex proteins and laminin changes due to angiogenesis in rat cerebral hypoperfusion.
Nagy et al., Budapest, Hungary. In Microvasc Res, 2011
Alterations in the immunoreactivities and expressions of beta-dystroglycan, utrophin, and laminin seem to correlate with angiogenesis rather than BBB disruption during cerebral hypoperfusion.
Dystroglycan modulates the ability of insulin-like growth factor-1 to promote oligodendrocyte differentiation.
Colognato et al., Stony Brook, United States. In J Neurosci Res, 2010
we identified and examined a connection between dystroglycan and the ability of insulin-like growth factor-1 (IGF-1) to promote oligodendrocyte differentiation
Interdependence of laminin-mediated clustering of lipid rafts and the dystrophin complex in astrocytes.
Moukhles et al., Vancouver, Canada. In J Biol Chem, 2009
Findings demonstrate the interdependence between laminin binding to dystroglycan and GM1-containing lipid raft reorganization and provide novel insight into the dystrophin complex regulation of AQP4 polarization in astrocytes.
Stretch-induced activation of AMP kinase in the lung requires dystroglycan.
Jones et al., Chicago, United States. In Am J Respir Cell Mol Biol, 2008
These results suggest that exposure to cyclic stretch activates the metabolic sensing pathway AMPK in the lung epithelium and supports a novel role for dystroglycan in this mechanotransduction.
2-Arylidenedihydroindole-3-ones: design, synthesis, and biological activity on bladder carcinoma cell lines.
Boutonnat et al., France. In Bioorg Med Chem Lett, 2007
The most active compound (3c) inhibited the proliferation, induced apoptosis, and decreased the expression of p-Stat5 and p-Pyk2 in DAG-1 and RT112 lines in which the FGFR3 is either mutated or overexpressed.
hAG-2 and hAG-3, human homologues of genes involved in differentiation, are associated with oestrogen receptor-positive breast tumours and interact with metastasis gene C4.4a and dystroglycan.
Terrett et al., Abingdon, United Kingdom. In Br J Cancer, 2003
Yeast two-hybrid cloning identified metastasis-associated GPI-anchored C4.4a protein and extracellular alpha-dystroglycan (DAG-1) as binding partners for both hAG-2 and hAG-3, which if replicated in clinical oncology would demonstrate a potential role in tumour metastasis through the regulation of receptor adhesion and functioning.
Dag-1 carcinoma cell in studying the mechanisms of progression and therapeutic resistance in bladder cancer.
Seigneurin et al., Grenoble, France. In Eur Urol, 2001
OBJECTIVE: We describe a new human bladder carcinoma cell line (DAG-1) established from a resected bladder cancer fragment and maintained in culture for more than 5 years and over 300 passages.
Effect of diacylglycerols on osteoclastic bone resorption.
Dempster et al., New York City, United States. In Calcif Tissue Int, 1996
1,2-dioctanoyl-sn-glycerol (DAG I) and 1,2-dihexanoyl-sn-glycerol (DAG III) caused a marked inhibition of resorption by rat osteoclasts at 6 hours, but there was recovery of bone-resorptive ability over a 24-hour incubation period.
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