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GoPubMed Proteins lists recent and important papers and reviews for proteins. Page last changed on 19 Dec 2016.

Corticotropin releasing hormone

Top mentioned proteins: V1a, CRF1, CAN, HAD, Neuropeptide
Papers using CRH antibodies
Role of pituitary hormones on 17alpha-ethinylestradiol-induced cholestasis in rat
Baud Olivier, In PLoS ONE, 2006
... The polyclonal antibodies against rat 11β-HSD-2, 11β-HSD-1, GR, and CRH were purchased from Santa Cruz Biotechnology Inc ...
Effects of estrogen antagonists and agonists on the ACTH response to restraint stress in female rats
Christian Helen C et al., In The Journal of Endocrinology, 2000
... Dexamethasone (Faulding Pharmaceutical plc, Leamington Spa, Warks, UK) and corticotrophin-releasing hormone (CRH; Bachem, Heidelberg, Germany) used in ...
Papers on CRH
Environmental enrichment delays limbic epileptogenesis and restricts pathologic synaptic plasticity.
Jones et al., Melbourne, Australia. In Epilepsia, Feb 2016
Timm's staining revealed significant reductions in aberrant mossy fiber sprouting in EE rats (p < 0.05), and these effects of EE were accompanied by reduced expression of TrkB and CRH genes.
MCG101-induced cancer anorexia-cachexia features altered expression of hypothalamic Nucb2 and Cartpt and increased plasma levels of cocaine- and amphetamine-regulated transcript peptides.
Smedh et al., Göteborg, Sweden. In Oncol Rep, Feb 2016
Expression of mRNA for corticotropin-releasing hormone (CRH), cocaine- and amphetamine-regulated transcript (CART), nesfatin-1, thyrotropin (TSH) and the TSH receptor were explored.
The nutrient and energy sensor Sirt1 regulates the hypothalamic-pituitary-adrenal (HPA) axis by altering the production of the prohormone convertase 2 (PC2) essential in the maturation of corticotropin releasing hormone (CRH) from its prohormone in male rats.
Nillni et al., United States. In J Biol Chem, Feb 2016
The axis initiates with the production and release of corticotropin-releasing hormone (CRH) from the paraventricular nucleus (PVN) of the hypothalamus.
Electroacupuncture regulate hypothalamic-pituitary-adrenal axis and enhance hippocampal serotonin system in a rat model of depression.
Dong et al., Shanghai, China. In Neurosci Lett, Feb 2016
Hypothalamic corticotropin-releasing hormone (CRH) mRNA, plasma adrenocorticotropic hormone (ACTH) and corticosterone (CORT) were estimated as indices of HPA axis activity.
Adolescent chronic stress causes hypothalamo-pituitary-adrenocortical hypo-responsiveness and depression-like behavior in adult female rats.
Herman et al., Cincinnati, United States. In Psychoneuroendocrinology, Jan 2016
Adolescent females tested immediately after CVS did not exhibit differences in stress reactivity or immobility in the forced swim test, despite evidence for enhanced central HPA axis drive (increased CRH mRNA expression in PVN).
GABAergic regulation of the HPA and HPG axes and the impact of stress on reproductive function.
Maguire et al., Boston, United States. In J Steroid Biochem Mol Biol, Jan 2016
UNASSIGNED: The hypothalamic-pituitary-adrenal (HPA) and hypothalamic-pituitary-gonadal (HPG) axes are regulated by GABAergic signaling at the level of corticotropin-releasing hormone (CRH) and gonadotropin-releasing hormone (GnRH) neurons, respectively.
Neuropeptidergic regulation of pair-bonding and stress buffering: Lessons from voles.
Wang et al., Medford, United States. In Horm Behav, Nov 2015
In the present review, we highlight the work from this burgeoning field and focus on the role(s) of the neuropeptides oxytocin (OT), vasopressin (AVP), and corticotrophin releasing hormone (CRH) mediating stress buffering.
Molecular, cellular, morphological, physiological and behavioral aspects of gonadotropin-inhibitory hormone.
Tsutsui et al., Tokyo, Japan. In Gen Comp Endocrinol, Oct 2015
GnIH neurons regulate not only gonadotropin synthesis and release in the pituitary, but also regulate various neurons in the brain, such as GnRH1, GnRH2, dopamine, POMC, NPY, orexin, MCH, CRH, oxytocin, and kisspeptin neurons.
Hypothalamic-Pituitary--Adrenal Axis-Feedback Control.
Keller-Wood, United States. In Compr Physiol, Aug 2015
Negative feedback control by corticosteroids limits pituitary secretion of corticotropin, ACTH, and hypothalamic secretion of corticotropin-releasing hormone, CRH, and vasopressin, AVP, resulting in regulation of both basal and stress-induced ACTH secretion.
Glucocorticoid signaling drives epigenetic and transcription factors to induce key regulators of human parturition.
Chrousos et al., München, Germany. In Sci Signal, 2014
Two recent articles by Di Stefano et al. in the Archives and Wang et al. in this issue of Science Signaling reveal novel mechanisms by which glucocorticoid signaling can drive the epigenetic and transcriptional machinery to induce molecules involved in parturition, including the neuropeptide corticotropin-releasing hormone (CRH), the enzyme cyclooxygenase-2 (COX-2), and the autacoid hormone prostaglandin E2.
Key role of CRF in the skin stress response system.
Rivier et al., Memphis, United States. In Endocr Rev, 2013
The discovery of corticotropin-releasing factor (CRF) or CRH defining the upper regulatory arm of the hypothalamic-pituitary-adrenal (HPA) axis, along with the identification of the corresponding receptors (CRFRs 1 and 2), represents a milestone in our understanding of central mechanisms regulating body and local homeostasis.
The role of CRH in behavioral responses to acute restraint stress in zebrafish.
Bonan et al., Porto Alegre, Brazil. In Prog Neuropsychopharmacol Biol Psychiatry, 2012
Rigorous acute stressor stimuli induce behavioral changes, accompanied by an increase of cortisol levels with delayed control of CRH mRNA expression.
Development and function of the human fetal adrenal cortex: a key component in the feto-placental unit.
Jaffe et al., San Francisco, United States. In Endocr Rev, 2011
Recent studies employing modern technologies such as gene expression profiling and laser capture microdissection have revealed that development and/or function of the fetal adrenal cortex may be regulated by a panoply of molecules, including transcription factors, extracellular matrix components, locally produced growth factors, and placenta-derived CRH, in addition to the primary regulator, fetal pituitary ACTH.
Lifespan extension induced by AMPK and calcineurin is mediated by CRTC-1 and CREB.
Dillin et al., Los Angeles, United States. In Nature, 2011
We demonstrate that CRTC-1 is a direct AMPK target, and interacts with the CREB homologue-1 (CRH-1) transcription factor in vivo.
Neuroendocrine mechanisms in pregnancy and parturition.
Challis et al., Siena, Italy. In Endocr Rev, 2010
The CRH/urocortin (Ucn) family is another important neuroendocrine pathway involved in term and preterm birth.
Stress hormone regulation: biological role and translation into therapy.
Ising et al., München, Germany. In Annu Rev Psychol, 2009
We focus in this review on the biological roles of corticosteroids, corticotrophin-releasing hormone (CRH), and arginine vasopressin (AVP), and we evaluate the effects of treatments directed against the actions of these hormones.
Localization of corticotropin-releasing factor, urotensin I, and CRF-binding protein gene expression in the brain of the zebrafish, Danio rerio.
Bernier et al., Guelph, Canada. In J Comp Neurol, 2007
CRF localized in the preoptic area, periventricular hypothalamic and tectal regions, and dorsal part of the trigeminal motor nucleus.
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