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GoPubMed Proteins lists recent and important papers and reviews for proteins. Page last changed on 19 Dec 2016.

CDKN1A interacting zinc finger protein 1

Ciz1, Cip1-interacting zinc finger protein 1
Top mentioned proteins: OLF, TMEM16C, p21, DYT6, RDP
Papers on Ciz1
CIZ1 is upregulated in hepatocellular carcinoma and promotes the growth and migration of the cancer cells.
Wang et al., Nanjing, China. In Tumour Biol, Nov 2015
Although dys-regulation of CIZ1 (Cip1 interacting zinc finger protein 1) has been observed in various cancer types, its expression and functions in HCC remain unknown.
Cyclin-A-CDK2-mediated phosphorylation of CIZ1 blocks replisome formation and initiation of mammalian DNA replication.
Coverley et al., York, United Kingdom. In J Cell Sci, May 2015
CIZ1 is a nuclear matrix protein that cooperates with cyclin A2 (encoded by CCNA2) and CDK2 to promote mammalian DNA replication.
Isolated and combined dystonia syndromes - an update on new genes and their phenotypes.
Bhatia et al., London, United Kingdom. In Eur J Neurol, Apr 2015
Some genes, such as ANO3, GNAL and CIZ1, have been discovered for isolated dystonia, but they are probably not a common cause of classic cervical dystonia.
CIZ1 promoted the growth and migration of gallbladder cancer cells.
Liu et al., Shanghai, China. In Tumour Biol, Apr 2015
CIZ1 (Cip1 interacting zinc finger protein 1), a binding partner of p21(Cip1/Waf1), has been found to be involved in the tumorigenesis recently.
Ciz1 promotes tumorigenicity of prostate carcinoma cells.
Kong et al., Shenyang, China. In Front Biosci, 2014
Recent work has revealed the significance of CIP-interacting zinc finger protein 1 (CIZ1) in cancer cell biology, but its roles in prostatic carcinoma are unknown.
Dystonia: an update on phenomenology, classification, pathogenesis and treatment.
Bhatia et al., Heidelberg, Germany. In Curr Opin Neurol, 2014
The recently discovered genes ANO3, GNAL and CIZ1 appear not to be a common cause of adult-onset cervical dystonia.
Recent advances in the genetics of dystonia.
LeDoux et al., Memphis, United States. In Curr Neurol Neurosci Rep, 2014
These genes include PRRT2 (DYT10), CIZ1 (DYT23), ANO3 (DYT24), GNAL (DYT25), and TUBB4A (DYT4).
Genetics in dystonia.
Klein, Lübeck, Germany. In Parkinsonism Relat Disord, 2014
However, three of these putative new genes still await independent confirmation (TUBB4/DYT4; CIZ1/DYT23; ANO3/DYT24) and only 11 'DYT' genes have been unequivocally demonstrated to cause different forms of dystonia.
Genetics in dystonia: an update.
Ozelius et al., New York City, United States. In Curr Neurol Neurosci Rep, 2013
The past year has been extremely successful with regard to the genetics of dystonia, with the identification of four new dystonia genes (CIZ1, ANO3, GNAL, and TUBB4A).
CIZ1 regulates the proliferation, cycle distribution and colony formation of RKO human colorectal cancer cells.
Qu et al., Jinan, China. In Mol Med Report, 2013
Cip1-interacting zinc finger protein 1 (CIZ1) is a nuclear protein that was observed to bind to p21Cip1/Waf1.
Klein et al., In Continuum (minneap Minn), 2013
Isolated dystonia (with dystonic tremor) can be caused by mutations in TOR1A (DYT1), TUBB4 (DYT4), THAP1 (DYT6), PRKRA (DYT16), CIZ1 (DYT23), ANO3 (DYT24), and GNAL (DYT25).
The genetics of dystonia: new twists in an old tale.
Wood et al., London, United Kingdom. In Brain, 2013
In just over a year, four new genes have been shown to cause primary dystonia (CIZ1, ANO3, TUBB4A and GNAL), PRRT2 has been identified as the cause of paroxysmal kinesigenic dystonia and other genes, such as SLC30A10 and ATP1A3, have been linked to more complicated forms of dystonia or new phenotypes.
Genetics of dystonia: what's known? What's new? What's next?
Klein et al., Lübeck, Germany. In Mov Disord, 2013
Isolated dystonia can be caused by mutations in TOR1A (DYT1), TUBB4 (DYT4), THAP1 (DYT6), CIZ1 (DYT23), ANO3 (DYT24), and GNAL (DYT25).
Mutations in GNAL cause primary torsion dystonia.
Ozelius et al., New York City, United States. In Nat Genet, 2013
Only three genes for primary torsion dystonia (PTD), TOR1A (DYT1), THAP1 (DYT6) and CIZ1 (ref.
Dystonia, facial dysmorphism, intellectual disability and breast cancer associated with a chromosome 13q34 duplication and overexpression of TFDP1: case report.
Okun et al., In Bmc Med Genet, 2012
Some cases of primary and neurodegenerative dystonia have been associated with mutations in individual genes critical to the G1-S checkpoint pathway (THAP1, ATM, CIZ1 and TAF1).
Mutations in CIZ1 cause adult onset primary cervical dystonia.
LeDoux et al., Memphis, United States. In Ann Neurol, 2012
Mutations in CIZ1 may cause adult onset, primary cervical dystonia
Differential detection of alternatively spliced variants of Ciz1 in normal and cancer cells using a custom exon-junction microarray.
Coverley et al., York, United Kingdom. In Bmc Cancer, 2009
alternatively spliced variant of Ciz1 is associated with cancer.
Altered splicing in exon 8 of the DNA replication factor CIZ1 affects subnuclear distribution and is associated with Alzheimer's disease.
Mitchelmore et al., Roskilde, Denmark. In Mol Cell Neurosci, 2008
An isoform of CIZ1 which lacks a glutamine-rich region, due to alternative splicing in exon 8, is upregulated in Alzheimer's brains relative to the full-length CIZ1 protein.
Ciz1, a p21 cip1/Waf1-interacting zinc finger protein and DNA replication factor, is a novel molecular partner for human enhancer of rudimentary homolog.
Kozlowski et al., Warsaw, Poland. In Febs J, 2008
Ciz1 is a nuclear zinc finger protein interacting with p21, and is a DNA replication factor. The region of Ciz1 necessary for the interaction with ERH spans residues 531-644, encompassing its first zinc finger motif
Cancer-associated missplicing of exon 4 influences the subnuclear distribution of the DNA replication factor CIZ1.
Coverley et al., York, United Kingdom. In Hum Mutat, 2007
conditional exclusion of exon 4 influences the spatial distribution of the Ciz1 protein within the nucleus, showing that CIZ1 alternative splicing could influence organized patterns of DNA replication
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